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Journal Abstract Search

218 related items for PubMed ID: 24506938

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  • 3. Deletion of Sox9 in the liver leads to hepatic cystogenesis in mice by transcriptionally downregulating Sec63.
    Xu WP, Cui YL, Chen LL, Ding K, Ding CH, Chen F, Zhang X, Xie WF.
    J Pathol; 2021 May; 254(1):57-69. PubMed ID: 33512716
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  • 7. Polycystic liver disease is a disorder of cotranslational protein processing.
    Drenth JP, Martina JA, van de Kerkhof R, Bonifacino JS, Jansen JB.
    Trends Mol Med; 2005 Jan; 11(1):37-42. PubMed ID: 15649821
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  • 11. Cysts of PRKCSH mutated polycystic liver disease patients lack hepatocystin but express Sec63p.
    Waanders E, Croes HJ, Maass CN, te Morsche RH, van Geffen HJ, van Krieken JH, Fransen JA, Drenth JP.
    Histochem Cell Biol; 2008 Mar; 129(3):301-10. PubMed ID: 18224332
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  • 12. Polycystic liver disease: an overview of pathogenesis, clinical manifestations and management.
    Cnossen WR, Drenth JP.
    Orphanet J Rare Dis; 2014 May 01; 9():69. PubMed ID: 24886261
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  • 13. PRKCSH/80K-H, the protein mutated in polycystic liver disease, protects polycystin-2/TRPP2 against HERP-mediated degradation.
    Gao H, Wang Y, Wegierski T, Skouloudaki K, Pütz M, Fu X, Engel C, Boehlke C, Peng H, Kuehn EW, Kim E, Kramer-Zucker A, Walz G.
    Hum Mol Genet; 2010 Jan 01; 19(1):16-24. PubMed ID: 19801576
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  • 14. Inhibition of Cdc25A suppresses hepato-renal cystogenesis in rodent models of polycystic kidney and liver disease.
    Masyuk TV, Radtke BN, Stroope AJ, Banales JM, Masyuk AI, Gradilone SA, Gajdos GB, Chandok N, Bakeberg JL, Ward CJ, Ritman EL, Kiyokawa H, LaRusso NF.
    Gastroenterology; 2012 Mar 01; 142(3):622-633.e4. PubMed ID: 22155366
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  • 15. Isolated polycystic liver disease genes define effectors of polycystin-1 function.
    Besse W, Dong K, Choi J, Punia S, Fedeles SV, Choi M, Gallagher AR, Huang EB, Gulati A, Knight J, Mane S, Tahvanainen E, Tahvanainen P, Sanna-Cherchi S, Lifton RP, Watnick T, Pei YP, Torres VE, Somlo S.
    J Clin Invest; 2017 May 01; 127(5):1772-1785. PubMed ID: 28375157
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  • 16. Abnormal hepatocystin caused by truncating PRKCSH mutations leads to autosomal dominant polycystic liver disease.
    Drenth JP, Tahvanainen E, te Morsche RH, Tahvanainen P, Kääriäinen H, Höckerstedt K, van de Kamp JM, Breuning MH, Jansen JB.
    Hepatology; 2004 Apr 01; 39(4):924-31. PubMed ID: 15057895
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  • 17. IFT-A deficiency in juvenile mice impairs biliary development and exacerbates ADPKD liver disease.
    Wang W, Pottorf TS, Wang HH, Dong R, Kavanaugh MA, Cornelius JT, Dennis KL, Apte U, Pritchard MT, Sharma M, Tran PV.
    J Pathol; 2021 Jul 01; 254(3):289-302. PubMed ID: 33900625
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  • 20. A genetic interaction network of five genes for human polycystic kidney and liver diseases defines polycystin-1 as the central determinant of cyst formation.
    Fedeles SV, Tian X, Gallagher AR, Mitobe M, Nishio S, Lee SH, Cai Y, Geng L, Crews CM, Somlo S.
    Nat Genet; 2011 Jun 19; 43(7):639-47. PubMed ID: 21685914
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