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Journal Abstract Search

602 related items for PubMed ID: 24570070

  • 1. Tonic inhibition by G protein-coupled receptor kinase 2 of Akt/endothelial nitric-oxide synthase signaling in human vascular endothelial cells under conditions of hyperglycemia with high insulin levels.
    Taguchi K, Sakata K, Ohashi W, Imaizumi T, Imura J, Hattori Y.
    J Pharmacol Exp Ther; 2014 May; 349(2):199-208. PubMed ID: 24570070
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  • 2. Inhibitor of G protein-coupled receptor kinase 2 normalizes vascular endothelial function in type 2 diabetic mice by improving β-arrestin 2 translocation and ameliorating Akt/eNOS signal dysfunction.
    Taguchi K, Matsumoto T, Kamata K, Kobayashi T.
    Endocrinology; 2012 Jul; 153(7):2985-96. PubMed ID: 22581458
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  • 3. [The Role of GRK2 and Its Potential as a New Therapeutic Target in Diabetic Vascular Complications].
    Taguchi K.
    Yakugaku Zasshi; 2015 Jul; 135(8):961-7. PubMed ID: 26234354
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  • 4. G protein-coupled receptor kinase 2, with β-arrestin 2, impairs insulin-induced Akt/endothelial nitric oxide synthase signaling in ob/ob mouse aorta.
    Taguchi K, Matsumoto T, Kamata K, Kobayashi T.
    Diabetes; 2012 Aug; 61(8):1978-85. PubMed ID: 22688330
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  • 5. Dysfunction of endothelium-dependent relaxation to insulin via PKC-mediated GRK2/Akt activation in aortas of ob/ob mice.
    Taguchi K, Kobayashi T, Matsumoto T, Kamata K.
    Am J Physiol Heart Circ Physiol; 2011 Aug; 301(2):H571-83. PubMed ID: 21572010
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  • 6. Angiotensin II causes endothelial dysfunction via the GRK2/Akt/eNOS pathway in aortas from a murine type 2 diabetic model.
    Taguchi K, Kobayashi T, Takenouchi Y, Matsumoto T, Kamata K.
    Pharmacol Res; 2011 Nov; 64(5):535-46. PubMed ID: 21571071
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  • 10. CCN1 acutely increases nitric oxide production via integrin αvβ3-Akt-S6K-phosphorylation of endothelial nitric oxide synthase at the serine 1177 signaling axis.
    Hwang S, Lee HJ, Kim G, Won KJ, Park YS, Jo I.
    Free Radic Biol Med; 2015 Dec; 89():229-40. PubMed ID: 26393424
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  • 12. Roles of ROS and PKC-βII in ionizing radiation-induced eNOS activation in human vascular endothelial cells.
    Sakata K, Kondo T, Mizuno N, Shoji M, Yasui H, Yamamori T, Inanami O, Yokoo H, Yoshimura N, Hattori Y.
    Vascul Pharmacol; 2015 Jul; 70():55-65. PubMed ID: 25869503
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  • 13. Suppression of GRK2 expression reduces endothelial dysfunction by restoring glucose homeostasis.
    Taguchi K, Hida M, Hasegawa M, Narimatsu H, Matsumoto T, Kobayashi T.
    Sci Rep; 2017 Aug 16; 7(1):8436. PubMed ID: 28814745
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  • 17. Protective effects of 6-Gingerol on vascular endothelial cell injury induced by high glucose via activation of PI3K-AKT-eNOS pathway in human umbilical vein endothelial cells.
    Liu D, Wu M, Lu Y, Xian T, Wang Y, Huang B, Zeng G, Huang Q.
    Biomed Pharmacother; 2017 Sep 16; 93():788-795. PubMed ID: 28709132
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  • 18. Leptin-stimulated endothelial nitric-oxide synthase via an adenosine 5'-monophosphate-activated protein kinase/Akt signaling pathway is attenuated by interaction with C-reactive protein.
    Procopio C, Andreozzi F, Laratta E, Cassese A, Beguinot F, Arturi F, Hribal ML, Perticone F, Sesti G.
    Endocrinology; 2009 Aug 16; 150(8):3584-93. PubMed ID: 19359389
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  • 19. G-protein-coupled receptor kinase 2 and endothelial dysfunction: molecular insights and pathophysiological mechanisms.
    Taguchi K, Matsumoto T, Kobayashi T.
    J Smooth Muscle Res; 2015 Aug 16; 51():37-49. PubMed ID: 26447102
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  • 20. Netrin-1 restores cell injury and impaired angiogenesis in vascular endothelial cells upon high glucose by PI3K/AKT-eNOS.
    Xing Y, Lai J, Liu X, Zhang N, Ming J, Liu H, Zhang X.
    J Mol Endocrinol; 2017 May 16; 58(4):167-177. PubMed ID: 28250059
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