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Journal Abstract Search

217 related items for PubMed ID: 2474755

  • 1. Gene regulation by tyrosine kinases: src protein activates various promoters, including c-fos.
    Fujii M, Shalloway D, Verma IM.
    Mol Cell Biol; 1989 Jun; 9(6):2493-9. PubMed ID: 2474755
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  • 2. Inhibition of v-raf-dependent c-fos expression and transformation by a kinase-defective mutant of the mitogen-activated protein kinase Erk2.
    Kortenjann M, Thomae O, Shaw PE.
    Mol Cell Biol; 1994 Jul; 14(7):4815-24. PubMed ID: 8007980
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  • 3. Transformation of NIH 3T3 cells by cotransfection with c-src and nuclear oncogenes.
    Shalloway D, Johnson PJ, Freed EO, Coulter D, Flood WA.
    Mol Cell Biol; 1987 Oct; 7(10):3582-90. PubMed ID: 2446117
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  • 4. pp60c-src variants containing lesions that affect phosphorylation at tyrosines 416 and 527.
    Harvey R, Hehir KM, Smith AE, Cheng SH.
    Mol Cell Biol; 1989 Sep; 9(9):3647-56. PubMed ID: 2476663
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  • 5. Most of the substrates of oncogenic viral tyrosine protein kinases can be phosphorylated by cellular tyrosine protein kinases in normal cells.
    Kamps MP, Sefton BM.
    Oncogene Res; 1988 Sep; 3(2):105-15. PubMed ID: 2465525
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  • 6. Relationship between c-src tyrosine kinase activity and the control of glucose transporter gene expression.
    Matsouka PT, Flier JS.
    Mol Endocrinol; 1989 Nov; 3(11):1845-51. PubMed ID: 2481817
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  • 8. pp60c-src tyrosine kinase, myristylation, and modulatory domains are required for enhanced mitogenic responsiveness to epidermal growth factor seen in cells overexpressing c-src.
    Wilson LK, Luttrell DK, Parsons JT, Parsons SJ.
    Mol Cell Biol; 1989 Apr; 9(4):1536-44. PubMed ID: 2471064
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  • 9. The amino-terminal region of pp60c-src has a modulatory role and contains multiple sites of tyrosine phosphorylation.
    Espino PC, Harvey R, Schweickhardt RL, White GA, Smith AE, Cheng SH.
    Oncogene; 1990 Mar; 5(3):283-93. PubMed ID: 1690377
    [Abstract] [Full Text] [Related]

  • 10. v-src mutations outside the carboxyl-coding region are not sufficient to fully activate transformation by pp60c-src in NIH 3T3 cells.
    Reddy S, Yaciuk P, Kmiecik TE, Coussens PM, Shalloway D.
    Mol Cell Biol; 1988 Feb; 8(2):704-12. PubMed ID: 2451122
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  • 13. A point mutation at tyrosine-809 in the human colony-stimulating factor 1 receptor impairs mitogenesis without abrogating tyrosine kinase activity, association with phosphatidylinositol 3-kinase, or induction of c-fos and junB genes.
    Roussel MF, Shurtleff SA, Downing JR, Sherr CJ.
    Proc Natl Acad Sci U S A; 1990 Sep; 87(17):6738-42. PubMed ID: 2168557
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  • 14. The cellular src gene product regulates junctional cell-to-cell communication.
    Azarnia R, Reddy S, Kmiecik TE, Shalloway D, Loewenstein WR.
    Science; 1988 Jan 22; 239(4838):398-401. PubMed ID: 2447651
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  • 15. From c-src to v-src, or the case of the missing C terminus.
    Sefton BM, Hunter T.
    Cancer Surv; 1986 Jan 22; 5(2):159-72. PubMed ID: 2430701
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  • 16. Restriction of the in vitro and in vivo tyrosine protein kinase activities of pp60c-src relative to pp60v-src.
    Coussens PM, Cooper JA, Hunter T, Shalloway D.
    Mol Cell Biol; 1985 Oct 22; 5(10):2753-63. PubMed ID: 2426575
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  • 19. Interferon-gamma-induced epithelial ICAM-1 expression and monocyte adhesion. Involvement of protein kinase C-dependent c-Src tyrosine kinase activation pathway.
    Chang YJ, Holtzman MJ, Chen CC.
    J Biol Chem; 2002 Mar 01; 277(9):7118-26. PubMed ID: 11751911
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