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609 related items for PubMed ID: 24793433

  • 1. Dioxin-induced retardation of development through a reduction in the expression of pituitary hormones and possible involvement of an aryl hydrocarbon receptor in this defect: a comparative study using two strains of mice with different sensitivities to dioxin.
    Takeda T, Taura J, Hattori Y, Ishii Y, Yamada H.
    Toxicol Appl Pharmacol; 2014 Aug 01; 278(3):220-9. PubMed ID: 24793433
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  • 2. The aryl hydrocarbon receptor is indispensable for dioxin-induced defects in sexually-dimorphic behaviors due to the reduction in fetal steroidogenesis of the pituitary-gonadal axis in rats.
    Hattori Y, Takeda T, Nakamura A, Nishida K, Shioji Y, Fukumitsu H, Yamada H, Ishii Y.
    Biochem Pharmacol; 2018 Aug 01; 154():213-221. PubMed ID: 29753751
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  • 3. Maternal exposure to dioxin imprints sexual immaturity of the pups through fixing the status of the reduced expression of hypothalamic gonadotropin-releasing hormone.
    Takeda T, Fujii M, Hattori Y, Yamamoto M, Shimazoe T, Ishii Y, Himeno M, Yamada H.
    Mol Pharmacol; 2014 Jan 01; 85(1):74-82. PubMed ID: 24132183
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  • 4. Fetal pituitary gonadotropin as an initial target of dioxin in its impairment of cholesterol transportation and steroidogenesis in rats.
    Mutoh J, Taketoh J, Okamura K, Kagawa T, Ishida T, Ishii Y, Yamada H.
    Endocrinology; 2006 Feb 01; 147(2):927-36. PubMed ID: 16254025
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  • 8. Comparative contribution of the aryl hydrocarbon receptor gene to perinatal stage development and dioxin-induced toxicity between the urogenital complex and testis in the mouse.
    Ohsako S, Fukuzawa N, Ishimura R, Kawakami T, Wu Q, Nagano R, Zaha H, Sone H, Yonemoto J, Tohyama C.
    Biol Reprod; 2010 Mar 01; 82(3):636-43. PubMed ID: 20007409
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  • 10. Effects of aryl hydrocarbon receptor null mutation and in utero and lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure on prostate and seminal vesicle development in C57BL/6 mice.
    Lin TM, Ko K, Moore RW, Simanainen U, Oberley TD, Peterson RE.
    Toxicol Sci; 2002 Aug 01; 68(2):479-87. PubMed ID: 12151645
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  • 11. Suppression of fetal testicular cytochrome P450 17 by maternal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin: a mechanism involving an initial effect on gonadotropin synthesis in the pituitary.
    Taketoh J, Mutoh J, Takeda T, Ogishima T, Takeda S, Ishii Y, Ishida T, Yamada H.
    Life Sci; 2007 Mar 13; 80(14):1259-67. PubMed ID: 17291543
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  • 15. TCDD-induced expression of Ah receptor responsive genes in the pituitary and brain of cellular retinol-binding protein (CRBP-I) knockout mice.
    Huang P, Ceccatelli S, Hoegberg P, Sten Shi TJ, Håkansson H, Rannug A.
    Toxicol Appl Pharmacol; 2003 Nov 01; 192(3):262-74. PubMed ID: 14575644
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  • 17. A mouse strain less responsive to dioxin-induced prostaglandin E2 synthesis is resistant to the onset of neonatal hydronephrosis.
    Aida-Yasuoka K, Yoshioka W, Kawaguchi T, Ohsako S, Tohyama C.
    Toxicol Sci; 2014 Oct 01; 141(2):465-74. PubMed ID: 25015655
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  • 18. Role of the aryl hydrocarbon receptor in the development of control and 2,3,7,8-tetrachlorodibenzo-p-dioxin-exposed male mice.
    Lin TM, Ko K, Moore RW, Buchanan DL, Cooke PS, Peterson RE.
    J Toxicol Environ Health A; 2001 Oct 26; 64(4):327-42. PubMed ID: 11693491
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  • 19. Differential susceptibilities of Holtzman and Sprague-Dawley rats to fetal death and placental dysfunction induced by 2,3,7,8-teterachlorodibenzo-p-dioxin (TCDD) despite the identical primary structure of the aryl hydrocarbon receptor.
    Kawakami T, Ishimura R, Nohara K, Takeda K, Tohyama C, Ohsako S.
    Toxicol Appl Pharmacol; 2006 May 01; 212(3):224-36. PubMed ID: 16202437
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  • 20. Can resveratrol attenuate testicular damage in neonatal and adult rats exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin during gestation?
    Erthal RP, Siervo GEML, Silveira LTR, Scarano WR, Fernandes GSA.
    Reprod Fertil Dev; 2018 Mar 01; 30(3):442-450. PubMed ID: 28784202
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