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822 related items for PubMed ID: 24803432

  • 1. Inflammasome activation causes dual recruitment of NLRC4 and NLRP3 to the same macromolecular complex.
    Man SM, Hopkins LJ, Nugent E, Cox S, Glück IM, Tourlomousis P, Wright JA, Cicuta P, Monie TP, Bryant CE.
    Proc Natl Acad Sci U S A; 2014 May 20; 111(20):7403-8. PubMed ID: 24803432
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  • 2. Salmonella infection induces recruitment of Caspase-8 to the inflammasome to modulate IL-1β production.
    Man SM, Tourlomousis P, Hopkins L, Monie TP, Fitzgerald KA, Bryant CE.
    J Immunol; 2013 Nov 15; 191(10):5239-46. PubMed ID: 24123685
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  • 3. The CARD plays a critical role in ASC foci formation and inflammasome signalling.
    Proell M, Gerlic M, Mace PD, Reed JC, Riedl SJ.
    Biochem J; 2013 Feb 01; 449(3):613-21. PubMed ID: 23110696
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  • 4. Caspase-1 autoproteolysis is differentially required for NLRP1b and NLRP3 inflammasome function.
    Guey B, Bodnar M, Manié SN, Tardivel A, Petrilli V.
    Proc Natl Acad Sci U S A; 2014 Dec 02; 111(48):17254-9. PubMed ID: 25404286
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  • 5. Bacterial Outer Membrane Vesicle-Mediated Cytosolic Delivery of Flagellin Triggers Host NLRC4 Canonical Inflammasome Signaling.
    Yang J, Hwang I, Lee E, Shin SJ, Lee EJ, Rhee JH, Yu JW.
    Front Immunol; 2020 Dec 02; 11():581165. PubMed ID: 33312172
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  • 6. Involvement of absent in melanoma 2 in inflammasome activation in macrophages infected with Listeria monocytogenes.
    Tsuchiya K, Hara H, Kawamura I, Nomura T, Yamamoto T, Daim S, Dewamitta SR, Shen Y, Fang R, Mitsuyama M.
    J Immunol; 2010 Jul 15; 185(2):1186-95. PubMed ID: 20566831
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  • 7. Differential regulation of caspase-1 activation via NLRP3/NLRC4 inflammasomes mediated by aerolysin and type III secretion system during Aeromonas veronii infection.
    McCoy AJ, Koizumi Y, Higa N, Suzuki T.
    J Immunol; 2010 Dec 01; 185(11):7077-84. PubMed ID: 21037094
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  • 9. Asc modulates the function of NLRC4 in response to infection of macrophages by Legionella pneumophila.
    Case CL, Roy CR.
    mBio; 2011 Dec 01; 2(4):. PubMed ID: 21771913
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  • 10. Redundant roles for inflammasome receptors NLRP3 and NLRC4 in host defense against Salmonella.
    Broz P, Newton K, Lamkanfi M, Mariathasan S, Dixit VM, Monack DM.
    J Exp Med; 2010 Aug 02; 207(8):1745-55. PubMed ID: 20603313
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  • 15. Oxidative metabolism enables Salmonella evasion of the NLRP3 inflammasome.
    Wynosky-Dolfi MA, Snyder AG, Philip NH, Doonan PJ, Poffenberger MC, Avizonis D, Zwack EE, Riblett AM, Hu B, Strowig T, Flavell RA, Jones RG, Freedman BD, Brodsky IE.
    J Exp Med; 2014 Apr 07; 211(4):653-68. PubMed ID: 24638169
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  • 16. A Yersinia effector with enhanced inhibitory activity on the NF-κB pathway activates the NLRP3/ASC/caspase-1 inflammasome in macrophages.
    Zheng Y, Lilo S, Brodsky IE, Zhang Y, Medzhitov R, Marcu KB, Bliska JB.
    PLoS Pathog; 2011 Apr 07; 7(4):e1002026. PubMed ID: 21533069
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  • 19. Actin polymerization as a key innate immune effector mechanism to control Salmonella infection.
    Man SM, Ekpenyong A, Tourlomousis P, Achouri S, Cammarota E, Hughes K, Rizzo A, Ng G, Wright JA, Cicuta P, Guck JR, Bryant CE.
    Proc Natl Acad Sci U S A; 2014 Dec 09; 111(49):17588-93. PubMed ID: 25422455
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  • 20. Murine Borrelia arthritis is highly dependent on ASC and caspase-1, but independent of NLRP3.
    Oosting M, Buffen K, Malireddi SR, Sturm P, Verschueren I, Koenders MI, van de Veerdonk FL, van der Meer JW, Netea MG, Kanneganti TD, Joosten LA.
    Arthritis Res Ther; 2012 Nov 13; 14(6):R247. PubMed ID: 23148704
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