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190 related items for PubMed ID: 24896098

  • 1. Early treatment with anti-VLA-4 mAb can prevent the infiltration and/or development of pathogenic CD11b+CD4+ T cells in the CNS during progressive EAE.
    Mindur JE, Ito N, Dhib-Jalbut S, Ito K.
    PLoS One; 2014; 9(6):e99068. PubMed ID: 24896098
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  • 2. Tellurium compound AS101 ameliorates experimental autoimmune encephalomyelitis by VLA-4 inhibition and suppression of monocyte and T cell infiltration into the CNS.
    Lee JH, Halperin-Sheinfeld M, Baatar D, Mughal MR, Tae HJ, Kim JW, Carter A, Lustig A, Snir O, Lavie G, Okun E, Mattson MP, Sredni B, Taub DD.
    Neuromolecular Med; 2014 Jun; 16(2):292-307. PubMed ID: 24272426
    [Abstract] [Full Text] [Related]

  • 3. Macrophage brain infiltration in experimental autoimmune encephalomyelitis is not completely compromised by suppressed T-cell invasion: in vivo magnetic resonance imaging illustration in effective anti-VLA-4 antibody treatment.
    Deloire MS, Touil T, Brochet B, Dousset V, Caillé JM, Petry KG.
    Mult Scler; 2004 Oct; 10(5):540-8. PubMed ID: 15471371
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  • 8. Anti-CD48 Monoclonal Antibody Attenuates Experimental Autoimmune Encephalomyelitis by Limiting the Number of Pathogenic CD4+ T Cells.
    McArdel SL, Brown DR, Sobel RA, Sharpe AH.
    J Immunol; 2016 Oct 15; 197(8):3038-3048. PubMed ID: 27581174
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  • 9. Discordant effects of anti-VLA-4 treatment before and after onset of relapsing experimental autoimmune encephalomyelitis.
    Theien BE, Vanderlugt CL, Eagar TN, Nickerson-Nutter C, Nazareno R, Kuchroo VK, Miller SD.
    J Clin Invest; 2001 Apr 15; 107(8):995-1006. PubMed ID: 11306603
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  • 10. Modulation of experimental autoimmune encephalomyelitis by VLA-2 blockade.
    Tsunoda I, Terry EJ, Marble BJ, Lazarides E, Woods C, Fujinami RS.
    Brain Pathol; 2007 Jan 15; 17(1):45-55. PubMed ID: 17493037
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  • 12. Defects in CD4+ T cell LFA-1 integrin-dependent adhesion and proliferation protect Cd47-/- mice from EAE.
    Azcutia V, Bassil R, Herter JM, Engelbertsen D, Newton G, Autio A, Mayadas T, Lichtman AH, Khoury SJ, Parkos CA, Elyaman W, Luscinskas FW.
    J Leukoc Biol; 2017 Feb 15; 101(2):493-505. PubMed ID: 27965383
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  • 13. DAB389IL-2 suppresses autoimmune inflammation in the CNS and inhibits T cell-mediated lysis of glial target cells.
    Bhopale MK, Hilliard B, Constantinescu CS, Fujioka T, Ventura E, Phillips SM, Rostami A.
    Exp Mol Pathol; 2014 Feb 15; 96(1):108-17. PubMed ID: 23872438
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  • 14. Blockade of MCAM/CD146 impedes CNS infiltration of T cells over the choroid plexus.
    Breuer J, Korpos E, Hannocks MJ, Schneider-Hohendorf T, Song J, Zondler L, Herich S, Flanagan K, Korn T, Zarbock A, Kuhlmann T, Sorokin L, Wiendl H, Schwab N.
    J Neuroinflammation; 2018 Aug 22; 15(1):236. PubMed ID: 30134924
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  • 15. Opioid growth factor and low-dose naltrexone impair central nervous system infiltration by CD4 + T lymphocytes in established experimental autoimmune encephalomyelitis, a model of multiple sclerosis.
    Hammer LA, Waldner H, Zagon IS, McLaughlin PJ.
    Exp Biol Med (Maywood); 2016 Jan 22; 241(1):71-8. PubMed ID: 26202376
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  • 16. Innate IFN-γ promotes development of experimental autoimmune encephalomyelitis: a role for NK cells and M1 macrophages.
    Dungan LS, McGuinness NC, Boon L, Lynch MA, Mills KH.
    Eur J Immunol; 2014 Oct 22; 44(10):2903-17. PubMed ID: 25056715
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  • 17. Lack of junctional adhesion molecule (JAM)-B ameliorates experimental autoimmune encephalomyelitis.
    Tietz S, Périnat T, Greene G, Enzmann G, Deutsch U, Adams R, Imhof B, Aurrand-Lions M, Engelhardt B.
    Brain Behav Immun; 2018 Oct 22; 73():3-20. PubMed ID: 29920328
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  • 18. Role of Th17 cells in the pathogenesis of CNS inflammatory demyelination.
    Rostami A, Ciric B.
    J Neurol Sci; 2013 Oct 15; 333(1-2):76-87. PubMed ID: 23578791
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  • 19. Infiltration of Th1 and Th17 cells and activation of microglia in the CNS during the course of experimental autoimmune encephalomyelitis.
    Murphy AC, Lalor SJ, Lynch MA, Mills KH.
    Brain Behav Immun; 2010 May 15; 24(4):641-51. PubMed ID: 20138983
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  • 20. Antibodies to CD44 and integrin alpha4, but not L-selectin, prevent central nervous system inflammation and experimental encephalomyelitis by blocking secondary leukocyte recruitment.
    Brocke S, Piercy C, Steinman L, Weissman IL, Veromaa T.
    Proc Natl Acad Sci U S A; 1999 Jun 08; 96(12):6896-901. PubMed ID: 10359810
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