These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
Pubmed for Handhelds
PUBMED FOR HANDHELDS
Journal Abstract Search
68 related items for PubMed ID: 24966188
1. A static pressure sensitive receptor APJ promote H9c2 cardiomyocyte hypertrophy via PI3K-autophagy pathway. Xie F, Liu W, Feng F, Li X, Yang L, Lv D, Qin X, Li L, Chen L. Acta Biochim Biophys Sin (Shanghai); 2014 Aug; 46(8):699-708. PubMed ID: 24966188 [Abstract] [Full Text] [Related]
2. Caveolin-1-Autophagy Pathway Mediated Cardiomyocyte Hypertrophy Induced by Apelin-13. Wu D, Xie F, Xiao L, Feng F, Huang S, He L, Liu M, Zhou Q, Li L, Chen L. DNA Cell Biol; 2017 Aug; 36(8):611-618. PubMed ID: 28537760 [Abstract] [Full Text] [Related]
3. Apelin-13 promotes cardiomyocyte hypertrophy via PI3K-Akt-ERK1/2-p70S6K and PI3K-induced autophagy. Xie F, Liu W, Feng F, Li X, He L, Lv D, Qin X, Li L, Li L, Chen L. Acta Biochim Biophys Sin (Shanghai); 2015 Dec; 47(12):969-80. PubMed ID: 26607438 [Abstract] [Full Text] [Related]
4. High-density lipoprotein inhibits mechanical stress-induced cardiomyocyte autophagy and cardiac hypertrophy through angiotensin II type 1 receptor-mediated PI3K/Akt pathway. Lin L, Liu X, Xu J, Weng L, Ren J, Ge J, Zou Y. J Cell Mol Med; 2015 Aug; 19(8):1929-38. PubMed ID: 25946687 [Abstract] [Full Text] [Related]
5. Panax notoginseng saponins inhibit ischemia-induced apoptosis by activating PI3K/Akt pathway in cardiomyocytes. Chen S, Liu J, Liu X, Fu Y, Zhang M, Lin Q, Zhu J, Mai L, Shan Z, Yu X, Yang M, Lin S. J Ethnopharmacol; 2011 Sep 01; 137(1):263-70. PubMed ID: 21619920 [Abstract] [Full Text] [Related]
6. The endoplasmic reticulum stress-autophagy pathway is involved in apelin-13-induced cardiomyocyte hypertrophy in vitro. Xie F, Wu D, Huang SF, Cao JG, Li HN, He L, Liu MQ, Li LF, Chen LX. Acta Pharmacol Sin; 2017 Dec 01; 38(12):1589-1600. PubMed ID: 28748915 [Abstract] [Full Text] [Related]
7. [Erythropoietin inhibits angiotensin II induced cardiomyocyte hypertrophy in vitro via activating PI3K/Akt-eNOS pathway]. Wen Y, Ma YX, Zhang XJ, Hong LF, Feng DY, Lu ZH. Zhonghua Xin Xue Guan Bing Za Zhi; 2009 May 01; 37(5):436-40. PubMed ID: 19781221 [Abstract] [Full Text] [Related]
8. Oxidative stress impairs myocyte autophagy, resulting in myocyte hypertrophy. Wang JP, Chi RF, Wang K, Ma T, Guo XF, Zhang XL, Li B, Qin FZ, Han XB, Fan BA. Exp Physiol; 2018 Apr 01; 103(4):461-472. PubMed ID: 29327381 [Abstract] [Full Text] [Related]
9. G protein-coupled APJ receptor signaling induces focal adhesion formation and cell motility. Hashimoto Y, Ishida J, Yamamoto R, Fujiwara K, Asada S, Kasuya Y, Mochizuki N, Fukamizu A. Int J Mol Med; 2005 Nov 01; 16(5):787-92. PubMed ID: 16211245 [Abstract] [Full Text] [Related]
15. 17beta-estradiol reduces cardiac hypertrophy mediated through the up-regulation of PI3K/Akt and the suppression of calcineurin/NF-AT3 signaling pathways in rats. Wu CH, Liu JY, Wu JP, Hsieh YH, Liu CJ, Hwang JM, Lee SD, Chen LM, Chang MH, Kuo WW, Shyu JC, Tsai JH, Huang CY. Life Sci; 2005 Dec 12; 78(4):347-56. PubMed ID: 16183079 [Abstract] [Full Text] [Related]
18. The protective effect of Apelin-13 against cardiac hypertrophy through activating the PI3K-AKT-mTOR signaling pathway. Peng Y, Jingming R, Shaowen C, Feng H, Pengli Z. Iran J Basic Med Sci; 2023 Feb 12; 26(2):183-189. PubMed ID: 36742144 [Abstract] [Full Text] [Related]
19. AKT is involved in granulosa cell autophagy regulation via mTOR signaling during rat follicular development and atresia. Choi J, Jo M, Lee E, Choi D. Reproduction; 2014 Jan 12; 147(1):73-80. PubMed ID: 24131573 [Abstract] [Full Text] [Related]
20. Non-activated APJ suppresses the angiotensin II type 1 receptor, whereas apelin-activated APJ acts conversely. Sun X, Iida S, Yoshikawa A, Senbonmatsu R, Imanaka K, Maruyama K, Nishimura S, Inagami T, Senbonmatsu T. Hypertens Res; 2011 Jun 12; 34(6):701-6. PubMed ID: 21412239 [Abstract] [Full Text] [Related] Page: [Next] [New Search]