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Journal Abstract Search

228 related items for PubMed ID: 24987096

  • 1. IQGAP1 is important for activation of caspase-1 in macrophages and is targeted by Yersinia pestis type III effector YopM.
    Chung LK, Philip NH, Schmidt VA, Koller A, Strowig T, Flavell RA, Brodsky IE, Bliska JB.
    mBio; 2014 Jul 01; 5(4):e01402-14. PubMed ID: 24987096
    [Abstract] [Full Text] [Related]

  • 2. A Yersinia effector with enhanced inhibitory activity on the NF-κB pathway activates the NLRP3/ASC/caspase-1 inflammasome in macrophages.
    Zheng Y, Lilo S, Brodsky IE, Zhang Y, Medzhitov R, Marcu KB, Bliska JB.
    PLoS Pathog; 2011 Apr 01; 7(4):e1002026. PubMed ID: 21533069
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  • 3. YopJ-induced caspase-1 activation in Yersinia-infected macrophages: independent of apoptosis, linked to necrosis, dispensable for innate host defense.
    Zheng Y, Lilo S, Mena P, Bliska JB.
    PLoS One; 2012 Apr 01; 7(4):e36019. PubMed ID: 22563435
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  • 4. Uncovering an Important Role for YopJ in the Inhibition of Caspase-1 in Activated Macrophages and Promoting Yersinia pseudotuberculosis Virulence.
    Schoberle TJ, Chung LK, McPhee JB, Bogin B, Bliska JB.
    Infect Immun; 2016 Apr 01; 84(4):1062-1072. PubMed ID: 26810037
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  • 5. Caspase-1 activation in macrophages infected with Yersinia pestis KIM requires the type III secretion system effector YopJ.
    Lilo S, Zheng Y, Bliska JB.
    Infect Immun; 2008 Sep 01; 76(9):3911-23. PubMed ID: 18559430
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  • 6. Application of a Saccharomyces cerevisiae model to study requirements for trafficking of Yersinia pestis YopM in eucaryotic cells.
    Skrzypek E, Myers-Morales T, Whiteheart SW, Straley SC.
    Infect Immun; 2003 Feb 01; 71(2):937-47. PubMed ID: 12540576
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  • 9. The Yersinia Virulence Factor YopM Hijacks Host Kinases to Inhibit Type III Effector-Triggered Activation of the Pyrin Inflammasome.
    Chung LK, Park YH, Zheng Y, Brodsky IE, Hearing P, Kastner DL, Chae JJ, Bliska JB.
    Cell Host Microbe; 2016 Sep 14; 20(3):296-306. PubMed ID: 27569559
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  • 12. The plague virulence protein YopM targets the innate immune response by causing a global depletion of NK cells.
    Kerschen EJ, Cohen DA, Kaplan AM, Straley SC.
    Infect Immun; 2004 Aug 14; 72(8):4589-602. PubMed ID: 15271919
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  • 13. Interleukin-10 induction is an important virulence function of the Yersinia pseudotuberculosis type III effector YopM.
    McPhee JB, Mena P, Zhang Y, Bliska JB.
    Infect Immun; 2012 Jul 14; 80(7):2519-27. PubMed ID: 22547545
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  • 14. Caspase-3 mediates the pathogenic effect of Yersinia pestis YopM in liver of C57BL/6 mice and contributes to YopM's function in spleen.
    Ye Z, Gorman AA, Uittenbogaard AM, Myers-Morales T, Kaplan AM, Cohen DA, Straley SC.
    PLoS One; 2014 Jul 14; 9(11):e110956. PubMed ID: 25372388
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  • 15. The C-terminal tail of Yersinia pseudotuberculosis YopM is critical for interacting with RSK1 and for virulence.
    McCoy MW, Marré ML, Lesser CF, Mecsas J.
    Infect Immun; 2010 Jun 14; 78(6):2584-98. PubMed ID: 20368345
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  • 17. YopM puts caspase-1 on ice.
    Jørgensen I, Miao EA.
    Cell Host Microbe; 2012 Dec 13; 12(6):737-8. PubMed ID: 23245318
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  • 19. YopM inhibits platelet aggregation and is necessary for virulence of Yersinia pestis in mice.
    Leung KY, Reisner BS, Straley SC.
    Infect Immun; 1990 Oct 13; 58(10):3262-71. PubMed ID: 2401564
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  • 20. Structure-function analysis of Yersinia pestis YopM's interaction with alpha-thrombin to rule on its significance in systemic plague and to model YopM's mechanism of binding host proteins.
    Hines J, Skrzypek E, Kajava AV, Straley SC.
    Microb Pathog; 2001 Apr 13; 30(4):193-209. PubMed ID: 11312613
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