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150 related items for PubMed ID: 2498910

  • 1. Mesolimbic dopamine and early post-6-OHDA lesion enhanced responses to d-amphetamine.
    Lynch MR, Carey RJ.
    Pharmacol Biochem Behav; 1989 Feb; 32(2):577-80. PubMed ID: 2498910
    [Abstract] [Full Text] [Related]

  • 2. Amphetamine-induced rotation reveals post 6-OHDA lesion neurochemical reorganization.
    Lynch MR, Carey RJ.
    Behav Brain Res; 1989 Feb 01; 32(1):69-74. PubMed ID: 2495013
    [Abstract] [Full Text] [Related]

  • 3. Relationship of limbic dopamine levels to amphetamine- and anticholinergic-induced hyperactivity in the rat.
    Carey RJ.
    Biol Psychiatry; 1986 Mar 01; 21(3):317-21. PubMed ID: 3081055
    [No Abstract] [Full Text] [Related]

  • 4. The role of mesolimbic dopamine in conditioned locomotion produced by amphetamine.
    Gold LH, Swerdlow NR, Koob GF.
    Behav Neurosci; 1988 Aug 01; 102(4):544-52. PubMed ID: 3139012
    [Abstract] [Full Text] [Related]

  • 5. Stereotyped behaviour patterns and hyperactivity induced by amphetamine and apomorphine after discrete 6-hydroxydopamine lesions of extrapyramidal and mesolimbic nuclei.
    Castall B, Marsden CD, Naylor RJ, Pycock CJ.
    Brain Res; 1977 Mar 04; 123(1):89-111. PubMed ID: 300267
    [Abstract] [Full Text] [Related]

  • 6. The relationship between striatal and mesolimbic dopamine dysfunction and the nature of circling responses following 6-hydroxydopamine and electrolytic lesions of the ascending dopamine systems of rat brain.
    Costall B, Marsden CD, Naylor RJ, Pycock CJ.
    Brain Res; 1976 Dec 10; 118(1):87-113. PubMed ID: 990957
    [Abstract] [Full Text] [Related]

  • 7. Neuroleptic-like disruption of the conditioned avoidance response requires destruction of both the mesolimbic and nigrostriatal dopamine systems.
    Koob GF, Simon H, Herman JP, Le Moal M.
    Brain Res; 1984 Jun 15; 303(2):319-29. PubMed ID: 6430466
    [Abstract] [Full Text] [Related]

  • 8. Increased sensitivity to amphetamine and facilitation of amphetamine self-administration after 6-hydroxydopamine lesions of the amygdala.
    Deminière JM, Taghzouti K, Tassin JP, Le Moal M, Simon H.
    Psychopharmacology (Berl); 1988 Jun 15; 94(2):232-6. PubMed ID: 3127850
    [Abstract] [Full Text] [Related]

  • 9. Effects of d-amphetamine and apomorphine upon operant behavior and schedule-induced licking in rats with 6-hydroxydopamine-induced lesions of the nucleus accumbens.
    Robbins TW, Roberts DC, Koob GF.
    J Pharmacol Exp Ther; 1983 Mar 15; 224(3):662-73. PubMed ID: 6402587
    [Abstract] [Full Text] [Related]

  • 10. Differential effects of mesocortical, mesolimbic, and mesostriatal dopamine depletion on spontaneous, conditioned, and drug-induced locomotor activity.
    Jones GH, Robbins TW.
    Pharmacol Biochem Behav; 1992 Nov 15; 43(3):887-95. PubMed ID: 1448483
    [Abstract] [Full Text] [Related]

  • 11. Anatomical analysis of the involvement of mesolimbocortical dopamine in the locomotor stimulant actions of d-amphetamine and apomorphine.
    Clarke PB, Jakubovic A, Fibiger HC.
    Psychopharmacology (Berl); 1988 Nov 15; 96(4):511-20. PubMed ID: 3149775
    [Abstract] [Full Text] [Related]

  • 12. Differential effects of limbic versus striatal dopamine loss on motoric function.
    Carey RJ.
    Behav Brain Res; 1983 Mar 15; 7(3):283-96. PubMed ID: 6404287
    [Abstract] [Full Text] [Related]

  • 13. Amphetamine disruption of prepulse inhibition of acoustic startle is reversed by depletion of mesolimbic dopamine.
    Swerdlow NR, Mansbach RS, Geyer MA, Pulvirenti L, Koob GF, Braff DL.
    Psychopharmacology (Berl); 1990 Mar 15; 100(3):413-6. PubMed ID: 2315438
    [Abstract] [Full Text] [Related]

  • 14. Functional recovery following transplantation of ventral mesencephalic cells in rat subjected to 6-OHDA lesions of the mesolimbic dopaminergic neurons.
    Nadaud D, Herman JP, Simon H, Le Moal M.
    Brain Res; 1984 Jun 18; 304(1):137-41. PubMed ID: 6331577
    [Abstract] [Full Text] [Related]

  • 15. Regulatory behaviour, exploration and locomotion following NMDA or 6-OHDA lesions in the rat nucleus accumbens.
    Weissenborn R, Winn P.
    Behav Brain Res; 1992 Nov 15; 51(2):127-37. PubMed ID: 1466779
    [Abstract] [Full Text] [Related]

  • 16. Do forebrain structures compete for behavioral expression? Evidence from amphetamine-induced behavior, microdialysis, and caudate-accumbens lesions in medial frontal cortex damaged rats.
    Whishaw IQ, Fiorino D, Mittleman G, Castañeda E.
    Brain Res; 1992 Mar 27; 576(1):1-11. PubMed ID: 1381257
    [Abstract] [Full Text] [Related]

  • 17. Hyperactivity and hypoactivity produced by lesions to the mesolimbic dopamine system.
    Koob GF, Stinus L, Le Moal M.
    Behav Brain Res; 1981 Nov 27; 3(3):341-59. PubMed ID: 6796098
    [Abstract] [Full Text] [Related]

  • 18. Changes in locomotion and dopamine neurotransmission following amphetamine, haloperidol, and exposure to novel environmental stimuli.
    Bardo MT, Bowling SL, Pierce RC.
    Psychopharmacology (Berl); 1990 Nov 27; 101(3):338-43. PubMed ID: 2163539
    [Abstract] [Full Text] [Related]

  • 19. Hyperactivity following intradentate injection of colchicine: a role for dopamine systems in the nucleus accumbens.
    Emerich DF, Walsh TJ.
    Pharmacol Biochem Behav; 1990 Sep 27; 37(1):149-54. PubMed ID: 2124711
    [Abstract] [Full Text] [Related]

  • 20. 6-Hydroxydopamine lesion of the dopamine mesocorticolimbic cell bodies increases (+)-amphetamine self-administration.
    Deminière JM, Simon H, Herman JP, Le Moal M.
    Psychopharmacology (Berl); 1984 Sep 27; 83(3):281-4. PubMed ID: 6433392
    [Abstract] [Full Text] [Related]

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