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202 related items for PubMed ID: 2504671

  • 1. Defective protein phosphorylation associated with hypofunctions in stroke-prone spontaneously hypertensive rat platelets.
    Tomita T, Ikeda M, Inoue Y, Mitubori T, Umegaki K.
    Hypertension; 1989 Sep; 14(3):304-15. PubMed ID: 2504671
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  • 2. Phospholipid metabolism in platelets from stroke-prone spontaneously hypertensive rats and Wistar Kyoto rats.
    Ikeda M, Onda T, Mitsubori T, Umegaki K, Tomita I, Tomita T.
    J Pharmacobiodyn; 1992 Feb; 15(2):49-57. PubMed ID: 1328596
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  • 3. Defects of thrombin-induced protein phosphorylation in platelets from stroke-prone spontaneously hypertensive rats.
    Umegaki K, Ikeda M, Nakamura K, Tomita T.
    FEBS Lett; 1986 Feb 03; 196(1):139-44. PubMed ID: 3943626
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  • 4. Reduced functions of intracellular Ca2+ in aggregation, secretion and protein phosphorylation of permeabilized platelets from stroke-prone spontaneously hypertensive rats.
    Ikeda M, Onda T, Tomita I, Tomita T.
    Thromb Res; 1992 Sep 15; 67(6):655-63. PubMed ID: 1440531
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  • 5. Defective calcium transport in platelets from stroke-prone spontaneously hypertensive rats.
    Umegaki K, Ikeda M, Tomita T.
    Thromb Res; 1986 Feb 01; 41(3):415-23. PubMed ID: 3705015
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  • 6. Platelet Ca2+ is not increased in stroke-prone spontaneously hypertensive rats: comparative study with spontaneously hypertensive rats.
    Ono N, Oshima T, Ishida M, Ishida T, Matsuura H, Kambe M, Kajiyama G.
    Hypertension; 1996 Jun 01; 27(6):1312-7. PubMed ID: 8641741
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  • 11. Thrombin-induced abnormal platelet activation in spontaneously hypertensive rats is linked with phosphoinositides turnover and phosphorylation of 47,000 and 20,000 dalton proteins.
    Huzoor-Akbar, Anwer K.
    Thromb Res; 1988 Jan 01; 49(1):5-21. PubMed ID: 2831638
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  • 13. Congenital changes of platelet functions in stroke-prone SHR: aggregability of gel-filtered platelets, PRP and whole blood, and effects of hypotensive treatment.
    Ikeda M, Umegaki K, Takeshita N, Mitsubori M, Tomita T, Tomita I.
    Thromb Res; 1989 Nov 01; 56(3):441-52. PubMed ID: 2617481
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  • 15. Relationship between enhanced phosphoinositide turnover and cellular responses in platelets from spontaneously hypertensive rats.
    Koutouzov S, Remmal A, Meyer P, Marche P.
    J Cardiovasc Pharmacol; 1988 Nov 01; 11 Suppl 1():S6-9. PubMed ID: 2459517
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  • 16. Normal thrombin binding leads to greater fibrinogen binding and increased platelet aggregation in spontaneously hypertensive rats.
    Huzoor-Akbar, Kundu N, Kornhauser R.
    Life Sci; 1993 Nov 01; 53(26):1967-74. PubMed ID: 8255159
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  • 17. Evidence from comparative investigations that impaired platelet activation is not specific for stroke-prone spontaneously hypertensive rats.
    Klee A, Vater S, Schmid-Schönbein GW, Seiffge D.
    Stroke; 1993 Oct 01; 24(10):1528-33. PubMed ID: 8378957
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  • 18. Evidence that the rat is not an appropriate model to study the role of prostaglandins in normal or abnormal platelet aggregation.
    Huzoor-Akbar, Anwer K.
    Thromb Res; 1986 Feb 15; 41(4):555-66. PubMed ID: 3961734
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  • 19. Effect of amlodipine and cilazapril treatment on platelet Ca2+ handling in spontaneously hypertensive rats.
    Oshima T, Ono N, Ozono R, Higashi Y, Ishida M, Ishida T, Miho N, Nakashima H, Yano Y, Kambe M.
    Hypertens Res; 2003 Nov 15; 26(11):901-6. PubMed ID: 14714582
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  • 20. Hypersensitivity of phospholipase C in platelets of spontaneously hypertensive rats.
    Koutouzov S, Remmal A, Marche P, Meyer P.
    Hypertension; 1987 Nov 15; 10(5):497-504. PubMed ID: 2822575
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