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Journal Abstract Search

591 related items for PubMed ID: 25047849

  • 1.
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  • 2. Porphyromonas gulae Activates Unprimed and Gamma Interferon-Primed Macrophages via the Pattern Recognition Receptors Toll-Like Receptor 2 (TLR2), TLR4, and NOD2.
    Holden JA, O'Brien-Simpson NM, Lenzo JC, Orth RKH, Mansell A, Reynolds EC.
    Infect Immun; 2017 Sep; 85(9):. PubMed ID: 28630066
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  • 4. Porphyromonas gingivalis-stimulated macrophage subsets exhibit differential induction and responsiveness to interleukin-10.
    Foey AD, Habil N, Al-Shaghdali K, Crean S.
    Arch Oral Biol; 2017 Jan; 73():282-288. PubMed ID: 27816791
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  • 7. [Adipose-derived stem cells promote the polarization from M1 macrophages to M2 macrophages].
    Yin X, Pang C, Bai L, Zhang Y, Geng L.
    Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi; 2016 Mar; 32(3):332-8. PubMed ID: 26927552
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  • 8. In vivo exposure to Porphyromonas gingivalis up-regulates nitric oxide but suppresses tumour necrosis factor-alpha production by cultured macrophages.
    Frolov I, Houri-Hadad Y, Soskolne A, Shapira L.
    Immunology; 1998 Mar; 93(3):323-8. PubMed ID: 9640241
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  • 10. TLR2 stimulation impairs anti-inflammatory activity of M2-like macrophages, generating a chimeric M1/M2 phenotype.
    Quero L, Hanser E, Manigold T, Tiaden AN, Kyburz D.
    Arthritis Res Ther; 2017 Nov 02; 19(1):245. PubMed ID: 29096690
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  • 11. [Porphyromonas gingivalis lipopolysaccharide regulates macrophage polarization via triggering receptors expressed on myeloid cells-1].
    Liu L, Yang Y, Zhou JY, Wu YF, Zhao L.
    Zhonghua Kou Qiang Yi Xue Za Zhi; 2021 Feb 09; 56(2):175-181. PubMed ID: 33557502
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  • 12. The adenosine-dependent angiogenic switch of macrophages to an M2-like phenotype is independent of interleukin-4 receptor alpha (IL-4Rα) signaling.
    Ferrante CJ, Pinhal-Enfield G, Elson G, Cronstein BN, Hasko G, Outram S, Leibovich SJ.
    Inflammation; 2013 Aug 09; 36(4):921-31. PubMed ID: 23504259
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  • 13. Escherichia coli maltose-binding protein activates mouse peritoneal macrophages and induces M1 polarization via TLR2/4 in vivo and in vitro.
    Ni W, Zhang Q, Liu G, Wang F, Yuan H, Guo Y, Zhang X, Xie F, Li Q, Tai G.
    Int Immunopharmacol; 2014 Jul 09; 21(1):171-80. PubMed ID: 24825603
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  • 15. Experimental stress suppresses recruitment of macrophages but enhanced their P. gingivalis LPS-stimulated secretion of nitric oxide.
    Shapira L, Frolov I, Halabi A, Ben-Nathan D.
    J Periodontol; 2000 Mar 09; 71(3):476-81. PubMed ID: 10776937
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  • 16. Effects of aging on endotoxin tolerance induced by lipopolysaccharides derived from Porphyromonas gingivalis and Escherichia coli.
    Sun Y, Li H, Yang MF, Shu W, Sun MJ, Xu Y.
    PLoS One; 2012 Mar 09; 7(6):e39224. PubMed ID: 22723968
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  • 17. Gingipain of Porphyromonas gingivalis manipulates M1 macrophage polarization through C5a pathway.
    Hou Y, Yu H, Liu X, Li G, Pan J, Zheng C, Yu W.
    In Vitro Cell Dev Biol Anim; 2017 Aug 09; 53(7):593-603. PubMed ID: 28634882
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  • 18. Strain-dependent activation of monocytes and inflammatory macrophages by lipopolysaccharide of Porphyromonas gingivalis.
    Shapira L, Champagne C, Van Dyke TE, Amar S.
    Infect Immun; 1998 Jun 09; 66(6):2736-42. PubMed ID: 9596741
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  • 19. TET1 Knockdown Inhibits Porphyromonas gingivalis LPS/IFN-γ-Induced M1 Macrophage Polarization through the NF-κB Pathway in THP-1 Cells.
    Huang Y, Tian C, Li Q, Xu Q.
    Int J Mol Sci; 2019 Apr 24; 20(8):. PubMed ID: 31022963
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  • 20. Enoxaparin sodium bone cement plays an anti-inflammatory immunomodulatory role by inducing the polarization of M2 macrophages.
    Fan W, Fu D, Zhang L, Xiao Z, Shen X, Chen J, Qi X.
    J Orthop Surg Res; 2023 May 23; 18(1):380. PubMed ID: 37221568
    [Abstract] [Full Text] [Related]

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