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349 related items for PubMed ID: 2509554

  • 1. Activation of murine T cells by toxic shock syndrome toxin-1. The toxin-binding structures expressed on murine accessory cells are MHC class II molecules.
    Uchiyama T, Tadakuma T, Imanishi K, Araake M, Saito S, Yan XJ, Fujikawa H, Igarashi H, Yamaura N.
    J Immunol; 1989 Nov 15; 143(10):3175-82. PubMed ID: 2509554
    [Abstract] [Full Text] [Related]

  • 2. Activation of human T cells by toxic shock syndrome toxin-1: the toxin-binding structures expressed on human lymphoid cells acting as accessory cells are HLA class II molecules.
    Uchiyama T, Imanishi K, Saito S, Araake M, Yan XJ, Fujikawa H, Igarashi H, Kato H, Obata F, Kashiwagi N.
    Eur J Immunol; 1989 Oct 15; 19(10):1803-9. PubMed ID: 2583222
    [Abstract] [Full Text] [Related]

  • 3. Binding of toxic shock syndrome toxin-1 to murine major histocompatibility complex class II molecules.
    Scholl PR, Sekaly RP, Diez A, Glimcher LH, Geha RS.
    Eur J Immunol; 1990 Sep 15; 20(9):1911-6. PubMed ID: 2209697
    [Abstract] [Full Text] [Related]

  • 4. Activation of murine T cells by streptococcal pyrogenic exotoxin type A. Requirement for MHC class II molecules on accessory cells and identification of V beta elements in T cell receptor of toxin-reactive T cells.
    Imanishi K, Igarashi H, Uchiyama T.
    J Immunol; 1990 Nov 15; 145(10):3170-6. PubMed ID: 2230113
    [Abstract] [Full Text] [Related]

  • 5. The alpha 1 domain of the HLA-DR molecule is essential for high-affinity binding of the toxic shock syndrome toxin-1.
    Karp DR, Teletski CL, Scholl P, Geha R, Long EO.
    Nature; 1990 Aug 02; 346(6283):474-6. PubMed ID: 2377209
    [Abstract] [Full Text] [Related]

  • 6. Dissection of the function of HLA class II and costimulation in B cell-mediated and toxic shock syndrome toxin-1-induced T cell proliferation.
    Dennig D, O'Reilly RJ.
    J Immunol; 1993 Jun 15; 150(12):5231-40. PubMed ID: 7685791
    [Abstract] [Full Text] [Related]

  • 7. Carboxy-terminal residues of major histocompatibility complex class II-associated peptides control the presentation of the bacterial superantigen toxic shock syndrome toxin-1 to T cells.
    Wen R, Broussard DR, Surman S, Hogg TL, Blackman MA, Woodland DL.
    Eur J Immunol; 1997 Mar 15; 27(3):772-81. PubMed ID: 9079821
    [Abstract] [Full Text] [Related]

  • 8. Staphylococcal enterotoxin B and toxic shock syndrome toxin-1 bind to distinct sites on HLA-DR and HLA-DQ molecules.
    Scholl PR, Diez A, Geha RS.
    J Immunol; 1989 Oct 15; 143(8):2583-8. PubMed ID: 2551962
    [Abstract] [Full Text] [Related]

  • 9. Molecular requirements for T cell activation by the staphylococcal toxic shock syndrome toxin-1.
    Norton SD, Schlievert PM, Novick RP, Jenkins MK.
    J Immunol; 1990 Mar 15; 144(6):2089-95. PubMed ID: 2313089
    [Abstract] [Full Text] [Related]

  • 10. In vivo treatment with monoclonal anti-I-A antibodies: disappearance of splenic antigen-presenting cell function concomitant with modulation of splenic cell surface I-A and I-E antigens.
    Kruisbeek AM, Titus JA, Stephany DA, Gause BL, Longo DL.
    J Immunol; 1985 Jun 15; 134(6):3605-14. PubMed ID: 2580891
    [Abstract] [Full Text] [Related]

  • 11. Study of the biological activities of toxic shock syndrome toxin-1: II. Induction of the proliferative response and the interleukin 2 production by T cells from human peripheral blood mononuclear cells stimulated with the toxin.
    Uchiyama T, Kamagata Y, Yan XJ, Kohno M, Yoshioka M, Fujikawa H, Igarashi H, Okubo M, Awano F, Saito-Taki T.
    Clin Exp Immunol; 1987 Jun 15; 68(3):638-47. PubMed ID: 3498581
    [Abstract] [Full Text] [Related]

  • 12. Identification of binding domains on the superantigen, toxic shock syndrome-1, for class II MHC molecules.
    Soos JM, Russell JK, Jarpe MA, Pontzer CH, Johnson HM.
    Biochem Biophys Res Commun; 1993 Mar 31; 191(3):1211-7. PubMed ID: 8466498
    [Abstract] [Full Text] [Related]

  • 13. Relative activities of distinct isotypes of murine and human major histocompatibility complex class II molecules in binding toxic shock syndrome toxin 1 and determination of CD antigens expressed on T cells generated upon stimulation by the toxin.
    Uchiyama T, Saito S, Inoko H, Yan XJ, Imanishi K, Araake M, Igarashi H.
    Infect Immun; 1990 Dec 31; 58(12):3877-82. PubMed ID: 2123824
    [Abstract] [Full Text] [Related]

  • 14. Production of human and murine interleukin-2 by toxic shock syndrome toxin-1.
    Micusan VV, Mercier G, Bhatti AR, Reiser RF, Bergdoll MS, Oth D.
    Immunology; 1986 Jun 31; 58(2):203-8. PubMed ID: 3486824
    [Abstract] [Full Text] [Related]

  • 15. Pathogenesis of the toxic shock syndrome: T cell mediated lethal shock caused by the superantigen TSST-1.
    Miethke T, Duschek K, Wahl C, Heeg K, Wagner H.
    Eur J Immunol; 1993 Jul 31; 23(7):1494-500. PubMed ID: 8325325
    [Abstract] [Full Text] [Related]

  • 16. Effect of isotypes and allelic polymorphism on the binding of staphylococcal exotoxins to MHC class II molecules.
    Scholl PR, Diez A, Karr R, Sekaly RP, Trowsdale J, Geha RS.
    J Immunol; 1990 Jan 01; 144(1):226-30. PubMed ID: 2404064
    [Abstract] [Full Text] [Related]

  • 17. An evolutionary conserved mechanism of T cell activation by microbial toxins. Evidence for different affinities of T cell receptor-toxin interaction.
    Fleischer B, Gerardy-Schahn R, Metzroth B, Carrel S, Gerlach D, Köhler W.
    J Immunol; 1991 Jan 01; 146(1):11-7. PubMed ID: 1670601
    [Abstract] [Full Text] [Related]

  • 18. Intercellular adhesion molecule-1 and leukocyte function-associated antigen-3 provide costimulation for superantigen-induced T lymphocyte proliferation in the absence of a specific presenting molecule.
    Lamphear JG, Stevens KR, Rich RR.
    J Immunol; 1998 Jan 15; 160(2):615-23. PubMed ID: 9551895
    [Abstract] [Full Text] [Related]

  • 19. Engagement of MHC-class II molecules by staphylococcal exotoxins delivers a comitogenic signal to human B cells.
    Fuleihan R, Mourad W, Geha RS, Chatila T.
    J Immunol; 1991 Mar 01; 146(5):1661-6. PubMed ID: 1847171
    [Abstract] [Full Text] [Related]

  • 20. Engagement of major histocompatibility complex class II molecules leads to nitrite production in bone marrow-derived macrophages.
    Hauschildt S, Bessler WG, Scheipers P.
    Eur J Immunol; 1993 Nov 01; 23(11):2988-92. PubMed ID: 7693484
    [Abstract] [Full Text] [Related]


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