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Journal Abstract Search
312 related items for PubMed ID: 25209720
1. Essential role of the cancer stem/progenitor cell marker nucleostemin for indole-3-carbinol anti-proliferative responsiveness in human breast cancer cells. Tin AS, Park AH, Sundar SN, Firestone GL. BMC Biol; 2014 Sep 12; 12():72. PubMed ID: 25209720 [Abstract] [Full Text] [Related]
2. 1-Benzyl-indole-3-carbinol is a novel indole-3-carbinol derivative with significantly enhanced potency of anti-proliferative and anti-estrogenic properties in human breast cancer cells. Nguyen HH, Lavrenov SN, Sundar SN, Nguyen DH, Tseng M, Marconett CN, Kung J, Staub RE, Preobrazhenskaya MN, Bjeldanes LF, Firestone GL. Chem Biol Interact; 2010 Aug 05; 186(3):255-66. PubMed ID: 20570586 [Abstract] [Full Text] [Related]
3. Indole-3-carbinol activates the ATM signaling pathway independent of DNA damage to stabilize p53 and induce G1 arrest of human mammary epithelial cells. Brew CT, Aronchik I, Hsu JC, Sheen JH, Dickson RB, Bjeldanes LF, Firestone GL. Int J Cancer; 2006 Feb 15; 118(4):857-68. PubMed ID: 16152627 [Abstract] [Full Text] [Related]
4. Target protein interactions of indole-3-carbinol and the highly potent derivative 1-benzyl-I3C with the C-terminal domain of human elastase uncouples cell cycle arrest from apoptotic signaling. Aronchik I, Chen T, Durkin KA, Horwitz MS, Preobrazhenskaya MN, Bjeldanes LF, Firestone GL. Mol Carcinog; 2012 Nov 15; 51(11):881-94. PubMed ID: 22012859 [Abstract] [Full Text] [Related]
5. Indole-3-carbinol and its N-alkoxy derivatives preferentially target ERα-positive breast cancer cells. Caruso JA, Campana R, Wei C, Su CH, Hanks AM, Bornmann WG, Keyomarsi K. Cell Cycle; 2014 Nov 15; 13(16):2587-99. PubMed ID: 25486199 [Abstract] [Full Text] [Related]
6. Translocation of Bax to mitochondria induces apoptotic cell death in indole-3-carbinol (I3C) treated breast cancer cells. Rahman KM, Aranha O, Glazyrin A, Chinni SR, Sarkar FH. Oncogene; 2000 Nov 23; 19(50):5764-71. PubMed ID: 11126363 [Abstract] [Full Text] [Related]
7. The indole-3-carbinol cyclic tetrameric derivative CTet inhibits cell proliferation via overexpression of p21/CDKN1A in both estrogen receptor-positive and triple-negative breast cancer cell lines. De Santi M, Galluzzi L, Lucarini S, Paoletti MF, Fraternale A, Duranti A, De Marco C, Fanelli M, Zaffaroni N, Brandi G, Magnani M. Breast Cancer Res; 2011 Mar 24; 13(2):R33. PubMed ID: 21435243 [Abstract] [Full Text] [Related]
8. The antiproliferative response of indole-3-carbinol in human melanoma cells is triggered by an interaction with NEDD4-1 and disruption of wild-type PTEN degradation. Aronchik I, Kundu A, Quirit JG, Firestone GL. Mol Cancer Res; 2014 Nov 24; 12(11):1621-1634. PubMed ID: 25009292 [Abstract] [Full Text] [Related]
9. Indole-3-carbinol downregulation of telomerase gene expression requires the inhibition of estrogen receptor-alpha and Sp1 transcription factor interactions within the hTERT promoter and mediates the G1 cell cycle arrest of human breast cancer cells. Marconett CN, Sundar SN, Tseng M, Tin AS, Tran KQ, Mahuron KM, Bjeldanes LF, Firestone GL. Carcinogenesis; 2011 Sep 24; 32(9):1315-23. PubMed ID: 21693539 [Abstract] [Full Text] [Related]
12. Indole-3-carbinol stimulates transcription of the interferon gamma receptor 1 gene and augments interferon responsiveness in human breast cancer cells. Chatterji U, Riby JE, Taniguchi T, Bjeldanes EL, Bjeldanes LF, Firestone GL. Carcinogenesis; 2004 Jul 24; 25(7):1119-28. PubMed ID: 14988219 [Abstract] [Full Text] [Related]