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Journal Abstract Search


275 related items for PubMed ID: 25232125

  • 1. Loss of glial neurofascin155 delays developmental synapse elimination at the neuromuscular junction.
    Roche SL, Sherman DL, Dissanayake K, Soucy G, Desmazieres A, Lamont DJ, Peles E, Julien JP, Wishart TM, Ribchester RR, Brophy PJ, Gillingwater TH.
    J Neurosci; 2014 Sep 17; 34(38):12904-18. PubMed ID: 25232125
    [Abstract] [Full Text] [Related]

  • 2. Neuregulin1 displayed on motor axons regulates terminal Schwann cell-mediated synapse elimination at developing neuromuscular junctions.
    Lee YI, Li Y, Mikesh M, Smith I, Nave KA, Schwab MH, Thompson WJ.
    Proc Natl Acad Sci U S A; 2016 Jan 26; 113(4):E479-87. PubMed ID: 26755586
    [Abstract] [Full Text] [Related]

  • 3. Neurofascins are required to establish axonal domains for saltatory conduction.
    Sherman DL, Tait S, Melrose S, Johnson R, Zonta B, Court FA, Macklin WB, Meek S, Smith AJ, Cottrell DF, Brophy PJ.
    Neuron; 2005 Dec 08; 48(5):737-42. PubMed ID: 16337912
    [Abstract] [Full Text] [Related]

  • 4. Spatiotemporal ablation of myelinating glia-specific neurofascin (Nfasc NF155) in mice reveals gradual loss of paranodal axoglial junctions and concomitant disorganization of axonal domains.
    Pillai AM, Thaxton C, Pribisko AL, Cheng JG, Dupree JL, Bhat MA.
    J Neurosci Res; 2009 Jun 08; 87(8):1773-93. PubMed ID: 19185024
    [Abstract] [Full Text] [Related]

  • 5. Glial and neuronal isoforms of Neurofascin have distinct roles in the assembly of nodes of Ranvier in the central nervous system.
    Zonta B, Tait S, Melrose S, Anderson H, Harroch S, Higginson J, Sherman DL, Brophy PJ.
    J Cell Biol; 2008 Jun 30; 181(7):1169-77. PubMed ID: 18573915
    [Abstract] [Full Text] [Related]

  • 6. Homozygous mutation in the Neurofascin gene affecting the glial isoform of Neurofascin causes severe neurodevelopment disorder with hypotonia, amimia and areflexia.
    Smigiel R, Sherman DL, Rydzanicz M, Walczak A, Mikolajkow D, Krolak-Olejnik B, Kosinska J, Gasperowicz P, Biernacka A, Stawinski P, Marciniak M, Andrzejewski W, Boczar M, Krajewski P, Sasiadek MM, Brophy PJ, Ploski R.
    Hum Mol Genet; 2018 Nov 01; 27(21):3669-3674. PubMed ID: 30124836
    [Abstract] [Full Text] [Related]

  • 7. Biallelic mutations in neurofascin cause neurodevelopmental impairment and peripheral demyelination.
    Efthymiou S, Salpietro V, Malintan N, Poncelet M, Kriouile Y, Fortuna S, De Zorzi R, Payne K, Henderson LB, Cortese A, Maddirevula S, Alhashmi N, Wiethoff S, Ryten M, Botia JA, Provitera V, Schuelke M, Vandrovcova J, SYNAPS Study Group, Walsh L, Torti E, Iodice V, Najafi M, Karimiani EG, Maroofian R, Siquier-Pernet K, Boddaert N, De Lonlay P, Cantagrel V, Aguennouz M, El Khorassani M, Schmidts M, Alkuraya FS, Edvardson S, Nolano M, Devaux J, Houlden H.
    Brain; 2019 Oct 01; 142(10):2948-2964. PubMed ID: 31501903
    [Abstract] [Full Text] [Related]

  • 8. In vivo deletion of immunoglobulin domains 5 and 6 in neurofascin (Nfasc) reveals domain-specific requirements in myelinated axons.
    Thaxton C, Pillai AM, Pribisko AL, Labasque M, Dupree JL, Faivre-Sarrailh C, Bhat MA.
    J Neurosci; 2010 Apr 07; 30(14):4868-76. PubMed ID: 20371806
    [Abstract] [Full Text] [Related]

  • 9. Reelin promotes peripheral synapse elimination and maturation.
    Quattrocchi CC, Huang C, Niu S, Sheldon M, Benhayon D, Cartwright J, Mosier DR, Keller F, D'Arcangelo G.
    Science; 2003 Aug 01; 301(5633):649-53. PubMed ID: 12893944
    [Abstract] [Full Text] [Related]

  • 10. Blocking skeletal muscle DHPRs/Ryr1 prevents neuromuscular synapse loss in mutant mice deficient in type III Neuregulin 1 (CRD-Nrg1).
    Liu Y, Sugiura Y, Chen F, Lee KF, Ye Q, Lin W.
    PLoS Genet; 2019 Mar 01; 15(3):e1007857. PubMed ID: 30870432
    [Abstract] [Full Text] [Related]

  • 11. Axonal domain disorganization in Caspr1 and Caspr2 mutant myelinated axons affects neuromuscular junction integrity, leading to muscle atrophy.
    Saifetiarova J, Liu X, Taylor AM, Li J, Bhat MA.
    J Neurosci Res; 2017 Jul 01; 95(7):1373-1390. PubMed ID: 28370195
    [Abstract] [Full Text] [Related]

  • 12. Focal loss of the paranodal domain protein Neurofascin155 in the internal capsule impairs cortically induced muscle activity in vivo.
    Kunisawa K, Hatanaka N, Shimizu T, Kobayashi K, Osanai Y, Mouri A, Shi Q, Bhat MA, Nambu A, Ikenaka K.
    Mol Brain; 2020 Nov 23; 13(1):159. PubMed ID: 33228720
    [Abstract] [Full Text] [Related]

  • 13. Synapse elimination from the mouse neuromuscular junction in vitro: a non-Hebbian activity-dependent process.
    Nelson PG, Fields RD, Yu C, Liu Y.
    J Neurobiol; 1993 Nov 23; 24(11):1517-30. PubMed ID: 8283186
    [Abstract] [Full Text] [Related]

  • 14. Developmental neuromuscular synapse elimination: Activity-dependence and potential downstream effector mechanisms.
    Lee YI.
    Neurosci Lett; 2020 Jan 23; 718():134724. PubMed ID: 31877335
    [Abstract] [Full Text] [Related]

  • 15. Anti-Neurofascin-155 IgG4 antibodies prevent paranodal complex formation in vivo.
    Manso C, Querol L, Lleixà C, Poncelet M, Mekaouche M, Vallat JM, Illa I, Devaux JJ.
    J Clin Invest; 2019 Mar 14; 129(6):2222-2236. PubMed ID: 30869655
    [Abstract] [Full Text] [Related]

  • 16. Perisynaptic Schwann Cells at the Neuromuscular Synapse: Adaptable, Multitasking Glial Cells.
    Ko CP, Robitaille R.
    Cold Spring Harb Perspect Biol; 2015 Aug 20; 7(10):a020503. PubMed ID: 26430218
    [Abstract] [Full Text] [Related]

  • 17. Properties of Glial Cell at the Neuromuscular Junction Are Incompatible with Synaptic Repair in the SOD1G37R ALS Mouse Model.
    Martineau É, Arbour D, Vallée J, Robitaille R.
    J Neurosci; 2020 Sep 30; 40(40):7759-7777. PubMed ID: 32859714
    [Abstract] [Full Text] [Related]

  • 18. Differential expression and functions of neuronal and glial neurofascin isoforms and splice variants during PNS development.
    Basak S, Raju K, Babiarz J, Kane-Goldsmith N, Koticha D, Grumet M.
    Dev Biol; 2007 Nov 15; 311(2):408-22. PubMed ID: 17936266
    [Abstract] [Full Text] [Related]

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  • 20. Differences in the constituent fiber types contribute to the intermuscular variation in the timing of the developmental synapse elimination.
    Lee YI.
    Sci Rep; 2019 Jun 18; 9(1):8694. PubMed ID: 31213646
    [Abstract] [Full Text] [Related]


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