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257 related items for PubMed ID: 25243423

  • 1. Exogenous heat shock cognate protein 70 pretreatment attenuates cardiac and hepatic dysfunction with associated anti-inflammatory responses in experimental septic shock.
    Hsu JH, Yang RC, Lin SJ, Liou SF, Dai ZK, Yeh JL, Wu JR.
    Shock; 2014 Dec; 42(6):540-7. PubMed ID: 25243423
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  • 2. 17-DMAG, an HSP90 Inhibitor, Ameliorates Multiple Organ Dysfunction Syndrome via Induction of HSP70 in Endotoxemic Rats.
    Wang YL, Shen HH, Cheng PY, Chu YJ, Hwang HR, Lam KK, Lee YM.
    PLoS One; 2016 Dec; 11(5):e0155583. PubMed ID: 27224288
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  • 3. Extracellular heat shock protein HSC70 protects against lipopolysaccharide-induced hypertrophic responses in rat cardiomyocytes.
    Jan RL, Yang SC, Liu YC, Yang RC, Tsai SP, Huang SE, Yeh JL, Hsu JH.
    Biomed Pharmacother; 2020 Aug; 128():110370. PubMed ID: 32521457
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  • 4. Heat shock protein 70 confers cardiovascular protection during endotoxemia via inhibition of nuclear factor-kappaB activation and inducible nitric oxide synthase expression in the rostral ventrolateral medulla.
    Chan JY, Ou CC, Wang LL, Chan SH.
    Circulation; 2004 Dec 07; 110(23):3560-6. PubMed ID: 15557375
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  • 5. Nodakenin suppresses lipopolysaccharide-induced inflammatory responses in macrophage cells by inhibiting tumor necrosis factor receptor-associated factor 6 and nuclear factor-κB pathways and protects mice from lethal endotoxin shock.
    Rim HK, Cho W, Sung SH, Lee KT.
    J Pharmacol Exp Ther; 2012 Sep 07; 342(3):654-64. PubMed ID: 22637723
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  • 6. Effects of naltrexone on lipopolysaccharide-induced sepsis in rats.
    Lin SL, Lee YM, Chang HY, Cheng YW, Yen MH.
    J Biomed Sci; 2005 Sep 07; 12(2):431-40. PubMed ID: 15917999
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  • 7. Extracellular heat shock cognate protein 70 induces cardiac functional tolerance to endotoxin: differential effect on TNF-alpha and ICAM-1 levels in heart tissue.
    Su X, Sykes JB, Ao L, Raeburn CD, Fullerton DA, Meng X.
    Cytokine; 2010 Jul 07; 51(1):60-6. PubMed ID: 20478717
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  • 8. Alpha-lipoic acid prevents endotoxic shock and multiple organ dysfunction syndrome induced by endotoxemia in rats.
    Shen HH, Lam KK, Cheng PY, Kung CW, Chen SY, Lin PC, Chung MT, Lee YM.
    Shock; 2015 Apr 07; 43(4):405-11. PubMed ID: 25514429
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  • 9. Deferoxamine attenuates lipopolysaccharide-induced inflammatory responses and protects against endotoxic shock in mice.
    Wang S, Liu C, Pan S, Miao Q, Xue J, Xun J, Zhang Y, Gao Y, Duan X, Fan Y.
    Biochem Biophys Res Commun; 2015 Sep 18; 465(2):305-11. PubMed ID: 26277391
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  • 10. Synergistic therapeutic potential of dexamethasone and L-arginine in lipopolysaccharide-induced septic shock.
    Chatterjee S, Premachandran S, Shukla J, Poduval TB.
    J Surg Res; 2007 Jun 01; 140(1):99-108. PubMed ID: 17292408
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  • 12. Caffeic acid phenethyl ester protects mice from lethal endotoxin shock and inhibits lipopolysaccharide-induced cyclooxygenase-2 and inducible nitric oxide synthase expression in RAW 264.7 macrophages via the p38/ERK and NF-kappaB pathways.
    Jung WK, Choi I, Lee DY, Yea SS, Choi YH, Kim MM, Park SG, Seo SK, Lee SW, Lee CM, Park YM, Choi IW.
    Int J Biochem Cell Biol; 2008 Jun 01; 40(11):2572-82. PubMed ID: 18571461
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  • 13. NF-κB activation mediates LPS-or zymosan-induced hypotension and inflammation reversed by BAY61-3606, a selective Syk inhibitor, in rat models of septic and non-septic shock.
    Sahan-Firat S, Temiz-Resitoglu M, Guden DS, Senol SP, Sari AN, Cil M, Unsal D, Korkmaz B, Tunctan B, Malik KU, Buharalioglu CK.
    Clin Exp Pharmacol Physiol; 2019 Feb 01; 46(2):173-182. PubMed ID: 30347478
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  • 14. Effects of 5,14-HEDGE, a 20-HETE mimetic, on lipopolysaccharide-induced changes in MyD88/TAK1/IKKβ/IκB-α/NF-κB pathway and circulating miR-150, miR-223, and miR-297 levels in a rat model of septic shock.
    Sari AN, Korkmaz B, Serin MS, Kacan M, Unsal D, Buharalioglu CK, Sahan Firat S, Manthati VL, Falck JR, Malik KU, Tunctan B.
    Inflamm Res; 2014 Sep 01; 63(9):741-56. PubMed ID: 24915805
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  • 17. Effect of calpain inhibitor I, an inhibitor of the proteolysis of I kappa B, on the circulatory failure and multiple organ dysfunction caused by endotoxin in the rat.
    Ruetten H, Thiemermann C.
    Br J Pharmacol; 1997 Jun 01; 121(4):695-704. PubMed ID: 9208136
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  • 18. Gene deletion of protein tyrosine phosphatase 1B protects against sepsis-induced cardiovascular dysfunction and mortality.
    Coquerel D, Neviere R, Delile E, Mulder P, Marechal X, Montaigne D, Renet S, Remy-Jouet I, Gomez E, Henry JP, do Rego JC, Richard V, Tamion F.
    Arterioscler Thromb Vasc Biol; 2014 May 01; 34(5):1032-44. PubMed ID: 24578383
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  • 20. Cardiovascular and inflammatory response to cholecystokinin during endotoxemic shock.
    Saia RS, Bertozi G, Mestriner FL, Antunes-Rodrigues J, Queiróz Cunha F, Cárnio EC.
    Shock; 2013 Jan 01; 39(1):104-13. PubMed ID: 23247127
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