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Journal Abstract Search
817 related items for PubMed ID: 25368181
1. Basal and β-adrenergic regulation of the cardiac calcium channel CaV1.2 requires phosphorylation of serine 1700. Fu Y, Westenbroek RE, Scheuer T, Catterall WA. Proc Natl Acad Sci U S A; 2014 Nov 18; 111(46):16598-603. PubMed ID: 25368181 [Abstract] [Full Text] [Related]
2. Loss of β-adrenergic-stimulated phosphorylation of CaV1.2 channels on Ser1700 leads to heart failure. Yang L, Dai DF, Yuan C, Westenbroek RE, Yu H, West N, de la Iglesia HO, Catterall WA. Proc Natl Acad Sci U S A; 2016 Dec 06; 113(49):E7976-E7985. PubMed ID: 27864509 [Abstract] [Full Text] [Related]
3. Phosphorylation sites required for regulation of cardiac calcium channels in the fight-or-flight response. Fu Y, Westenbroek RE, Scheuer T, Catterall WA. Proc Natl Acad Sci U S A; 2013 Nov 26; 110(48):19621-6. PubMed ID: 24218620 [Abstract] [Full Text] [Related]
4. Regulation of cardiac L-type Ca²⁺ channel CaV1.2 via the β-adrenergic-cAMP-protein kinase A pathway: old dogmas, advances, and new uncertainties. Weiss S, Oz S, Benmocha A, Dascal N. Circ Res; 2013 Aug 16; 113(5):617-31. PubMed ID: 23948586 [Abstract] [Full Text] [Related]
5. Ser-2030, but not Ser-2808, is the major phosphorylation site in cardiac ryanodine receptors responding to protein kinase A activation upon beta-adrenergic stimulation in normal and failing hearts. Xiao B, Zhong G, Obayashi M, Yang D, Chen K, Walsh MP, Shimoni Y, Cheng H, Ter Keurs H, Chen SR. Biochem J; 2006 May 15; 396(1):7-16. PubMed ID: 16483256 [Abstract] [Full Text] [Related]
6. Enhanced basal contractility but reduced excitation-contraction coupling efficiency and beta-adrenergic reserve of hearts with increased Cav1.2 activity. Tang M, Zhang X, Li Y, Guan Y, Ai X, Szeto C, Nakayama H, Zhang H, Ge S, Molkentin JD, Houser SR, Chen X. Am J Physiol Heart Circ Physiol; 2010 Aug 15; 299(2):H519-28. PubMed ID: 20543081 [Abstract] [Full Text] [Related]
10. Cardiomyocyte PKA Ablation Enhances Basal Contractility While Eliminates Cardiac β-Adrenergic Response Without Adverse Effects on the Heart. Zhang Y, Wang WE, Zhang X, Li Y, Chen B, Liu C, Ai X, Zhang X, Tian Y, Zhang C, Tang M, Szeto C, Hua X, Xie M, Zeng C, Wu Y, Zhou L, Zhu W, Yu D, Houser SR, Chen X. Circ Res; 2019 Jun 07; 124(12):1760-1777. PubMed ID: 30982412 [Abstract] [Full Text] [Related]
11. β-adrenergic-mediated dynamic augmentation of sarcolemmal CaV 1.2 clustering and co-operativity in ventricular myocytes. Ito DW, Hannigan KI, Ghosh D, Xu B, Del Villar SG, Xiang YK, Dickson EJ, Navedo MF, Dixon RE. J Physiol; 2019 Apr 07; 597(8):2139-2162. PubMed ID: 30714156 [Abstract] [Full Text] [Related]
12. Phosphorylation of serine 1928 in the distal C-terminal domain of cardiac CaV1.2 channels during beta1-adrenergic regulation. Hulme JT, Westenbroek RE, Scheuer T, Catterall WA. Proc Natl Acad Sci U S A; 2006 Oct 31; 103(44):16574-9. PubMed ID: 17053072 [Abstract] [Full Text] [Related]