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Journal Abstract Search

206 related items for PubMed ID: 25377090

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  • 2. Altered excitability and exocytosis in chromaffin cells from the R6/1 mouse model of Huntington's disease is linked to over-expression of mutated huntingtin.
    Martínez-Ramírez C, Baraibar AM, Nanclares C, Méndez-López I, Gómez A, Muñoz MP, de Diego AMG, Gandía L, Casarejos MJ, García AG.
    J Neurochem; 2018 Nov; 147(4):454-476. PubMed ID: 30182387
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  • 4. Voltage-gated calcium channels are abnormal in cultured spinal motoneurons in the G93A-SOD1 transgenic mouse model of ALS.
    Chang Q, Martin LJ.
    Neurobiol Dis; 2016 Sep; 93():78-95. PubMed ID: 27151771
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  • 5. Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.
    Beqollari D, Romberg CF, Dobrowolny G, Martini M, Voss AA, Musarò A, Bannister RA.
    Skelet Muscle; 2016 Sep; 6():24. PubMed ID: 27340545
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  • 8. Modulatory mechanism of the endogenous peptide catestatin on neuronal nicotinic acetylcholine receptors and exocytosis.
    Herrero CJ, Alés E, Pintado AJ, López MG, García-Palomero E, Mahata SK, O'Connor DT, García AG, Montiel C.
    J Neurosci; 2002 Jan 15; 22(2):377-88. PubMed ID: 11784782
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  • 9. Early excitability changes in lumbar motoneurons of transgenic SOD1G85R and SOD1G(93A-Low) mice.
    Pambo-Pambo A, Durand J, Gueritaud JP.
    J Neurophysiol; 2009 Dec 15; 102(6):3627-42. PubMed ID: 19828728
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  • 12. C-Boutons and Their Influence on Amyotrophic Lateral Sclerosis Disease Progression.
    Wells TL, Myles JR, Akay T.
    J Neurosci; 2021 Sep 22; 41(38):8088-8101. PubMed ID: 34380764
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  • 13. Progressive changes in synaptic inputs to motoneurons in adult sacral spinal cord of a mouse model of amyotrophic lateral sclerosis.
    Jiang M, Schuster JE, Fu R, Siddique T, Heckman CJ.
    J Neurosci; 2009 Dec 02; 29(48):15031-8. PubMed ID: 19955354
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  • 14. The human G93A-SOD1 mutation in a pre-symptomatic rat model of amyotrophic lateral sclerosis increases the vulnerability to a mild spinal cord compression.
    Jokic N, Yip PK, Michael-Titus A, Priestley JV, Malaspina A.
    BMC Genomics; 2010 Nov 15; 11():633. PubMed ID: 21078175
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  • 15. Increased anxiety-like behavior and selective learning impairments are concomitant to loss of hippocampal interneurons in the presymptomatic SOD1(G93A) ALS mouse model.
    Quarta E, Bravi R, Scambi I, Mariotti R, Minciacchi D.
    J Comp Neurol; 2015 Aug 01; 523(11):1622-38. PubMed ID: 25684566
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  • 16. Key role of the nicotinic receptor in neurotransmitter exocytosis in human chromaffin cells.
    Pérez-Alvarez A, Albillos A.
    J Neurochem; 2007 Dec 01; 103(6):2281-90. PubMed ID: 17883397
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  • 17. Preferential potentiation of AMPA-mediated currents in brainstem hypoglossal motoneurons by subchronic exposure of mice expressing the human superoxide dismutase 1 G93A gene mutation to neurotoxicant methylmercury in vivo.
    Yuan Y, Bailey JM, Rivera-Lopez GM, Atchison WD.
    Neurotoxicology; 2024 Jan 01; 100():72-84. PubMed ID: 38065418
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  • 19. Early Hypoexcitability in a Subgroup of Spinal Motoneurons in Superoxide Dismutase 1 Transgenic Mice, a Model of Amyotrophic Lateral Sclerosis.
    Filipchuk A, Pambo-Pambo A, Gaudel F, Liabeuf S, Brocard C, Gueritaud JP, Durand J.
    Neuroscience; 2021 May 21; 463():337-353. PubMed ID: 33556455
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