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Journal Abstract Search


119 related items for PubMed ID: 2538312

  • 1. Dual mechanism involved in the hydrolysis of polyphosphoinositides in rat pancreatic islets.
    Yamaguchi T, Kanatsuka A, Makino H, Ohsawa H, Yoshida S.
    Endocrinology; 1989 Apr; 124(4):1870-4. PubMed ID: 2538312
    [Abstract] [Full Text] [Related]

  • 2. A role for calcium in the breakdown of inositol phospholipids in intact and digitonin-permeabilized pancreatic islets.
    Best L.
    Biochem J; 1986 Sep 15; 238(3):773-9. PubMed ID: 3541917
    [Abstract] [Full Text] [Related]

  • 3. Studies of the Ca2+ requirements for glucose- and carbachol-induced augmentation of inositol trisphosphate and inositol tetrakisphosphate accumulation in digitonin-permeabilized islets. Evidence for a glucose recognition site in insulin secretion.
    Wolf BA, Florholmen J, Turk J, McDaniel ML.
    J Biol Chem; 1988 Mar 15; 263(8):3565-75. PubMed ID: 2831191
    [Abstract] [Full Text] [Related]

  • 4. Nutrient and hormone-neurotransmitter stimuli induce hydrolysis of polyphosphoinositides in rat pancreatic islets.
    Best L, Malaisse WJ.
    Endocrinology; 1984 Nov 15; 115(5):1814-20. PubMed ID: 6092036
    [Abstract] [Full Text] [Related]

  • 5. Phosphoinositide hydrolysis and insulin secretion in response to glucose stimulation are impaired in isolated rat islets by prolonged exposure to the sulfonylurea tolbutamide.
    Zawalich WS.
    Endocrinology; 1989 Jul 15; 125(1):281-6. PubMed ID: 2544404
    [Abstract] [Full Text] [Related]

  • 6. Effect of glucose on polyphosphoinositide metabolism in isolated rat islets of Langerhans.
    Montague W, Morgan NG, Rumford GM, Prince CA.
    Biochem J; 1985 Apr 15; 227(2):483-9. PubMed ID: 2988501
    [Abstract] [Full Text] [Related]

  • 7. Interleukin-1 alpha exerts glucose-dependent stimulatory and inhibitory effects on islet cell phosphoinositide hydrolysis and insulin secretion.
    Zawalich WS, Zawalich KC, Rasmussen H.
    Endocrinology; 1989 May 15; 124(5):2350-7. PubMed ID: 2539976
    [Abstract] [Full Text] [Related]

  • 8. Forskolin-induced desensitization of pancreatic beta-cell insulin secretory responsiveness: possible involvement of impaired information flow in the inositol-lipid cycle.
    Zawalich WS, Zawalich KC.
    Endocrinology; 1990 May 15; 126(5):2307-12. PubMed ID: 1691696
    [Abstract] [Full Text] [Related]

  • 9. Biochemical mechanisms involved in monomethyl succinate-induced insulin secretion.
    Zawalich WS, Zawalich KC.
    Endocrinology; 1992 Aug 15; 131(2):649-54. PubMed ID: 1322278
    [Abstract] [Full Text] [Related]

  • 10. Interactions of cholecystokinin and glucose in rat pancreatic islets.
    Zawalich W, Takuwa N, Takuwa Y, Diaz VA, Rasmussen H.
    Diabetes; 1987 Apr 15; 36(4):426-33. PubMed ID: 3028890
    [Abstract] [Full Text] [Related]

  • 11. Chronic in vivo hyperglycemia impairs phosphoinositide hydrolysis and insulin release in isolated perifused rat islets.
    Zawalich WS, Zawalich KC, Shulman GI, Rossetti L.
    Endocrinology; 1990 Jan 15; 126(1):253-60. PubMed ID: 2152864
    [Abstract] [Full Text] [Related]

  • 12. Phosphoinositide metabolism and insulin secretion from isolated rat pancreatic islets.
    Clements RS, Rhoten WB.
    J Clin Invest; 1976 Mar 15; 57(3):684-91. PubMed ID: 175090
    [Abstract] [Full Text] [Related]

  • 13. Influence of pyruvic acid methyl ester on rat pancreatic islets. Effects on insulin secretion, phosphoinositide hydrolysis, and sensitization of the beta cell.
    Zawalich WS, Zawalich KC.
    J Biol Chem; 1997 Feb 07; 272(6):3527-31. PubMed ID: 9013600
    [Abstract] [Full Text] [Related]

  • 14. Gonadotropin-releasing hormone (GnRH) rapidly stimulates the formation of inositol phosphates and diacyglycerol in rat granulosa cells: further evidence for the involvement of Ca2+ and protein kinase C in the action of GnRH.
    Davis JS, West LA, Farese RV.
    Endocrinology; 1986 Jun 07; 118(6):2561-71. PubMed ID: 3009164
    [Abstract] [Full Text] [Related]

  • 15. Glucosamine-induced desensitization of beta-cell responses: possible involvement of impaired information flow in the phosphoinositide cycle.
    Zawalich WS, Zawalich KC.
    Endocrinology; 1992 Jun 07; 130(6):3135-42. PubMed ID: 1317776
    [Abstract] [Full Text] [Related]

  • 16. Phosphoinositide hydrolysis and insulin release from isolated perifused rat islets. Studies with glucose.
    Zawalich WS, Zawalich KC.
    Diabetes; 1988 Sep 07; 37(9):1294-300. PubMed ID: 2842212
    [Abstract] [Full Text] [Related]

  • 17. The digitonin-permeabilized pancreatic islet model. Effect of myo-inositol 1,4,5-trisphosphate on Ca2+ mobilization.
    Wolf BA, Comens PG, Ackermann KE, Sherman WR, McDaniel ML.
    Biochem J; 1985 May 01; 227(3):965-9. PubMed ID: 3890834
    [Abstract] [Full Text] [Related]

  • 18. The conditions under which rat islets are labelled with [3H]inositol alter the subsequent responses of these islets to a high glucose concentration.
    Zawalich WS, Zawalich KC, Rasmussen H.
    Biochem J; 1989 May 01; 259(3):743-9. PubMed ID: 2658979
    [Abstract] [Full Text] [Related]

  • 19. Effect of cholecystokinin on the accumulation of inositol phosphates in isolated pancreatic islets.
    Florholmen J, Malm D, Vonen B, Burhol PG.
    Am J Physiol; 1989 Dec 01; 257(6 Pt 1):G865-70. PubMed ID: 2692460
    [Abstract] [Full Text] [Related]

  • 20. Influence of cAMP and calcium on [3H]inositol efflux, inositol phosphate accumulation, and insulin release from isolated rat islets.
    Zawalich WS, Diaz VA, Zawalich KC.
    Diabetes; 1988 Nov 01; 37(11):1478-83. PubMed ID: 2846390
    [Abstract] [Full Text] [Related]


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