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112 related items for PubMed ID: 2542083

  • 1. Stimulation of adenosine receptor enhances alpha 1-adrenergic receptor-mediated activation of phospholipase C and Ca2+ mobilization in a pertussis toxin-sensitive manner in FRTL-5 thyroid cells.
    Okajima F, Sato K, Sho K, Kondo Y.
    FEBS Lett; 1989 May 08; 248(1-2):145-9. PubMed ID: 2542083
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  • 2. A permissive role of pertussis toxin substrate G-protein in P2-purinergic stimulation of phosphoinositide turnover and arachidonate release in FRTL-5 thyroid cells. Cooperative mechanism of signal transduction systems.
    Okajima F, Sato K, Nazarea M, Sho K, Kondo Y.
    J Biol Chem; 1989 Aug 05; 264(22):13029-37. PubMed ID: 2546944
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  • 3. Permissive stimulation of Ca(2+)-induced phospholipase A2 by an adenosine receptor agonist in a pertussis toxin-sensitive manner in FRTL-5 thyroid cells: a new 'cross-talk' mechanism in Ca2+ signalling.
    Shimegi S, Okajima F, Kondo Y.
    Biochem J; 1994 May 01; 299 ( Pt 3)(Pt 3):845-51. PubMed ID: 8192675
    [Abstract] [Full Text] [Related]

  • 4. Inhibition of atrial natriuretic peptide-induced cGMP accumulation by purinergic agonists in FRTL-5 thyroid cells. Involvement of both pertussis toxin-sensitive and insensitive mechanisms.
    Okajima F, Kondo Y.
    J Biol Chem; 1990 Dec 15; 265(35):21741-8. PubMed ID: 2174885
    [Abstract] [Full Text] [Related]

  • 5. Role of pertussis toxin sensitive G proteins in the alpha 1 adrenergic receptor but not in the thyrotropin receptor mediated activation of membrane phospholipases and iodide fluxes in FRTL-5 thyroid cells.
    Corda D, Kohn LD.
    Biochem Biophys Res Commun; 1986 Dec 30; 141(3):1000-6. PubMed ID: 3028381
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  • 6. Thyrotropin-induced hydrogen peroxide production in FRTL-5 thyroid cells is mediated not by adenosine 3',5'-monophosphate, but by Ca2+ signaling followed by phospholipase-A2 activation and potentiated by an adenosine derivative.
    Kimura T, Okajima F, Sho K, Kobayashi I, Kondo Y.
    Endocrinology; 1995 Jan 30; 136(1):116-23. PubMed ID: 7828520
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  • 7. Inhibition by islet-activating protein, pertussis toxin, of P2-purinergic receptor-mediated iodide efflux and phosphoinositide turnover in FRTL-5 cells.
    Okajima F, Sho K, Kondo Y.
    Endocrinology; 1988 Aug 30; 123(2):1035-43. PubMed ID: 2840260
    [Abstract] [Full Text] [Related]

  • 8. Phospholipase A2 and phospholipase C are activated by distinct GTP-binding proteins in response to alpha 1-adrenergic stimulation in FRTL5 thyroid cells.
    Burch RM, Luini A, Axelrod J.
    Proc Natl Acad Sci U S A; 1986 Oct 30; 83(19):7201-5. PubMed ID: 3020540
    [Abstract] [Full Text] [Related]

  • 9. P2-purinergic activation of phosphoinositide turnover is potentiated by A1-receptor stimulation in thyroid cells.
    Nazarea M, Okajima F, Kondo Y.
    Eur J Pharmacol; 1991 Jan 25; 206(1):47-52. PubMed ID: 1648495
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  • 12. Enkephalin activates the phospholipase C/Ca2+ system through cross-talk between opioid receptors and P2-purinergic or bradykinin receptors in NG 108-15 cells. A permissive role for pertussis toxin-sensitive G-proteins.
    Okajima F, Tomura H, Kondo Y.
    Biochem J; 1993 Feb 15; 290 ( Pt 1)(Pt 1):241-7. PubMed ID: 8382479
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  • 14. Possible interaction of alpha 1-adrenergic receptor with pertussis-toxin-sensitive guanine-nucleotide-binding regulatory proteins (G proteins) responsible for phospholipase C activation in rat liver plasma membranes.
    Tohkin M, Yagami T, Katada T, Matsubara T.
    Eur J Biochem; 1990 Nov 26; 194(1):81-7. PubMed ID: 2174782
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  • 15. Betagamma subunits of pertussis toxin-sensitive G proteins mediate A1 adenosine receptor agonist-induced activation of phospholipase C in collaboration with thyrotropin. A novel stimulatory mechanism through the cross-talk of two types of receptors.
    Tomura H, Itoh H, Sho K, Sato K, Nagao M, Ui M, Kondo Y, Okajima F.
    J Biol Chem; 1997 Sep 12; 272(37):23130-7. PubMed ID: 9287315
    [Abstract] [Full Text] [Related]

  • 16. Inhibition of thyrotropin-stimulated adenosine 3',5'-monophosphate formation in rat thyroid cells by an adenosine analog. Evidence that the inhibition is mediated by the putative inhibitory guanine nucleotide regulatory protein.
    Berman MI, Thomas CG, Nayfeh SN.
    J Cyclic Nucleotide Protein Phosphor Res; 1986 Sep 12; 11(2):99-111. PubMed ID: 3016048
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  • 17. Alpha 1-adrenergic regulation of TSH-stimulated cyclic AMP accumulation in rat thyroid cells.
    Berman MI, Jerdack G, Thomas CG, Nayfeh SN.
    Arch Biochem Biophys; 1987 Feb 15; 253(1):249-56. PubMed ID: 2434027
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  • 18. Intracellular cross-talk between receptors coupled to phospholipase C via pertussis toxin-sensitive and insensitive G-proteins in DDT1MF-2 cells.
    Dickenson JM, Hill SJ.
    Br J Pharmacol; 1993 Jul 15; 109(3):719-24. PubMed ID: 8358567
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  • 19. Extracellular ATP stimulates three different receptor-signal transduction systems in FRTL-5 thyroid cells. Activation of phospholipase C, and inhibition and activation of adenylate cyclase.
    Sato K, Okajima F, Kondo Y.
    Biochem J; 1992 Apr 01; 283 ( Pt 1)(Pt 1):281-7. PubMed ID: 1314567
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