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PUBMED FOR HANDHELDS

Journal Abstract Search


277 related items for PubMed ID: 25477432

  • 1. Activation of Foxo1 by insulin resistance promotes cardiac dysfunction and β-myosin heavy chain gene expression.
    Qi Y, Zhu Q, Zhang K, Thomas C, Wu Y, Kumar R, Baker KM, Xu Z, Chen S, Guo S.
    Circ Heart Fail; 2015 Jan; 8(1):198-208. PubMed ID: 25477432
    [Abstract] [Full Text] [Related]

  • 2. Kir6.1 improves cardiac dysfunction in diabetic cardiomyopathy via the AKT-FoxO1 signalling pathway.
    Wang J, Bai J, Duan P, Wang H, Li Y, Zhu Q.
    J Cell Mol Med; 2021 Apr; 25(8):3935-3949. PubMed ID: 33547878
    [Abstract] [Full Text] [Related]

  • 3. Early dysregulation of cardiac-specific microRNA-208a is linked to maladaptive cardiac remodelling in diabetic myocardium.
    Rawal S, Nagesh PT, Coffey S, Van Hout I, Galvin IF, Bunton RW, Davis P, Williams MJA, Katare R.
    Cardiovasc Diabetol; 2019 Jan 29; 18(1):13. PubMed ID: 30696455
    [Abstract] [Full Text] [Related]

  • 4. Metabolic stress-induced activation of FoxO1 triggers diabetic cardiomyopathy in mice.
    Battiprolu PK, Hojayev B, Jiang N, Wang ZV, Luo X, Iglewski M, Shelton JM, Gerard RD, Rothermel BA, Gillette TG, Lavandero S, Hill JA.
    J Clin Invest; 2012 Mar 29; 122(3):1109-18. PubMed ID: 22326951
    [Abstract] [Full Text] [Related]

  • 5. Upregulation of MG53 induces diabetic cardiomyopathy through transcriptional activation of peroxisome proliferation-activated receptor α.
    Liu F, Song R, Feng Y, Guo J, Chen Y, Zhang Y, Chen T, Wang Y, Huang Y, Li CY, Cao C, Zhang Y, Hu X, Xiao RP.
    Circulation; 2015 Mar 03; 131(9):795-804. PubMed ID: 25637627
    [Abstract] [Full Text] [Related]

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  • 7. FOXO1 contributes to diabetic cardiomyopathy via inducing imbalanced oxidative metabolism in type 1 diabetes.
    Yan D, Cai Y, Luo J, Liu J, Li X, Ying F, Xie X, Xu A, Ma X, Xia Z.
    J Cell Mol Med; 2020 Jul 03; 24(14):7850-7861. PubMed ID: 32450616
    [Abstract] [Full Text] [Related]

  • 8. Myocardin reverses insulin resistance and ameliorates cardiomyopathy by increasing IRS-1 expression in a murine model of lipodystrophy caused by adipose deficiency of vacuolar H+-ATPase V0d1 subunit.
    Yuan W, Lin H, Sun Y, Liu L, Yan M, Song Y, Zhang X, Lu X, Xu Y, He Q, Ouyang K, Zhang C, Pan Y, Huang Y, Li Y, Lu X, Liu J.
    Theranostics; 2024 Jul 03; 14(5):2246-2264. PubMed ID: 38505620
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  • 10. FoxO1 Plays an Important Role in Regulating β-Cell Compensation for Insulin Resistance in Male Mice.
    Zhang T, Kim DH, Xiao X, Lee S, Gong Z, Muzumdar R, Calabuig-Navarro V, Yamauchi J, Harashima H, Wang R, Bottino R, Alvarez-Perez JC, Garcia-Ocaña A, Gittes G, Dong HH.
    Endocrinology; 2016 Mar 03; 157(3):1055-70. PubMed ID: 26727107
    [Abstract] [Full Text] [Related]

  • 11. Increased insulin receptor substrate 2 expression is associated with steatohepatitis and altered lipid metabolism in obese subjects.
    Rametta R, Mozzi E, Dongiovanni P, Motta BM, Milano M, Roviaro G, Fargion S, Valenti L.
    Int J Obes (Lond); 2013 Jul 03; 37(7):986-92. PubMed ID: 23147115
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  • 13. Angiotensin IV attenuates diabetic cardiomyopathy via suppressing FoxO1-induced excessive autophagy, apoptosis and fibrosis.
    Zhang M, Sui W, Xing Y, Cheng J, Cheng C, Xue F, Zhang J, Wang X, Zhang C, Hao P, Zhang Y.
    Theranostics; 2021 Jul 03; 11(18):8624-8639. PubMed ID: 34522203
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  • 15. Nuclear miR-320 Mediates Diabetes-Induced Cardiac Dysfunction by Activating Transcription of Fatty Acid Metabolic Genes to Cause Lipotoxicity in the Heart.
    Li H, Fan J, Zhao Y, Zhang X, Dai B, Zhan J, Yin Z, Nie X, Fu XD, Chen C, Wang DW.
    Circ Res; 2019 Dec 06; 125(12):1106-1120. PubMed ID: 31638474
    [Abstract] [Full Text] [Related]

  • 16. Myocardial loss of IRS1 and IRS2 causes heart failure and is controlled by p38α MAPK during insulin resistance.
    Qi Y, Xu Z, Zhu Q, Thomas C, Kumar R, Feng H, Dostal DE, White MF, Baker KM, Guo S.
    Diabetes; 2013 Nov 06; 62(11):3887-900. PubMed ID: 24159000
    [Abstract] [Full Text] [Related]

  • 17. Deletion of FoxO1 leads to shortening of QRS by increasing Na(+) channel activity through enhanced expression of both cardiac NaV1.5 and β3 subunit.
    Cai B, Wang N, Mao W, You T, Lu Y, Li X, Ye B, Li F, Xu H.
    J Mol Cell Cardiol; 2014 Sep 06; 74():297-306. PubMed ID: 24956219
    [Abstract] [Full Text] [Related]

  • 18. QKI deficiency promotes FoxO1 mediated nitrosative stress and endoplasmic reticulum stress contributing to increased vulnerability to ischemic injury in diabetic heart.
    Guo W, Jiang T, Lian C, Wang H, Zheng Q, Ma H.
    J Mol Cell Cardiol; 2014 Oct 06; 75():131-40. PubMed ID: 25068621
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  • 20. Reactive oxygen species suppress cardiac NaV1.5 expression through Foxo1.
    Mao W, You T, Ye B, Li X, Dong HH, Hill JA, Li F, Xu H.
    PLoS One; 2012 Oct 06; 7(2):e32738. PubMed ID: 22400069
    [Abstract] [Full Text] [Related]


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