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Journal Abstract Search

427 related items for PubMed ID: 25485900

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  • 7. Beta-adrenergic enhancement of sarcoplasmic reticulum calcium leak in cardiac myocytes is mediated by calcium/calmodulin-dependent protein kinase.
    Curran J, Hinton MJ, Ríos E, Bers DM, Shannon TR.
    Circ Res; 2007 Feb 16; 100(3):391-8. PubMed ID: 17234966
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  • 8. Calcium-calmodulin-dependent protein kinase mediates the intracellular signalling pathways of cardiac apoptosis in mice with impaired glucose tolerance.
    Federico M, Portiansky EL, Sommese L, Alvarado FJ, Blanco PG, Zanuzzi CN, Dedman J, Kaetzel M, Wehrens XHT, Mattiazzi A, Palomeque J.
    J Physiol; 2017 Jun 15; 595(12):4089-4108. PubMed ID: 28105734
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  • 9. Reactive oxygen species contribute to the development of arrhythmogenic Ca²⁺ waves during β-adrenergic receptor stimulation in rabbit cardiomyocytes.
    Bovo E, Lipsius SL, Zima AV.
    J Physiol; 2012 Jul 15; 590(14):3291-304. PubMed ID: 22586224
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  • 10. Autonomous activation of CaMKII exacerbates diastolic calcium leak during beta-adrenergic stimulation in cardiomyocytes of metabolic syndrome rats.
    Romero-García T, Landa-Galvan HV, Pavón N, Mercado-Morales M, Valdivia HH, Rueda A.
    Cell Calcium; 2020 Nov 15; 91():102267. PubMed ID: 32920522
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  • 11. Calcium/calmodulin-dependent kinase II and nitric oxide synthase 1-dependent modulation of ryanodine receptors during β-adrenergic stimulation is restricted to the dyadic cleft.
    Dries E, Santiago DJ, Johnson DM, Gilbert G, Holemans P, Korte SM, Roderick HL, Sipido KR.
    J Physiol; 2016 Oct 15; 594(20):5923-5939. PubMed ID: 27121757
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  • 12. Ablation of phospholamban rescues reperfusion arrhythmias but exacerbates myocardium infarction in hearts with Ca2+/calmodulin kinase II constitutive phosphorylation of ryanodine receptors.
    Valverde CA, Mazzocchi G, Di Carlo MN, Ciocci Pardo A, Salas N, Ragone MI, Felice JI, Cely-Ortiz A, Consolini AE, Portiansky E, Mosca S, Kranias EG, Wehrens XHT, Mattiazzi A.
    Cardiovasc Res; 2019 Mar 01; 115(3):556-569. PubMed ID: 30169578
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  • 13. CaMKIIδC slows [Ca]i decline in cardiac myocytes by promoting Ca sparks.
    Guo T, Zhang T, Ginsburg KS, Mishra S, Brown JH, Bers DM.
    Biophys J; 2012 Jun 06; 102(11):2461-70. PubMed ID: 22713561
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  • 14. Maximal acceleration of Ca2+ release refractoriness by β-adrenergic stimulation requires dual activation of kinases PKA and CaMKII in mouse ventricular myocytes.
    Poláková E, Illaste A, Niggli E, Sobie EA.
    J Physiol; 2015 Mar 15; 593(6):1495-507. PubMed ID: 25772298
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  • 15. JNK2, a Newly-Identified SERCA2 Enhancer, Augments an Arrhythmic [Ca2+]SR Leak-Load Relationship.
    Yan J, Bare DJ, DeSantiago J, Zhao W, Mei Y, Chen Z, Ginsburg K, Solaro RJ, Wolska BM, Bers DM, Chen SRW, Ai X.
    Circ Res; 2021 Feb 19; 128(4):455-470. PubMed ID: 33334123
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  • 16. High-mobility group box 1 (HMGB1) impaired cardiac excitation-contraction coupling by enhancing the sarcoplasmic reticulum (SR) Ca(2+) leak through TLR4-ROS signaling in cardiomyocytes.
    Zhang C, Mo M, Ding W, Liu W, Yan D, Deng J, Luo X, Liu J.
    J Mol Cell Cardiol; 2014 Sep 19; 74():260-73. PubMed ID: 24937603
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  • 20. Transgenic CaMKIIdeltaC overexpression uniquely alters cardiac myocyte Ca2+ handling: reduced SR Ca2+ load and activated SR Ca2+ release.
    Maier LS, Zhang T, Chen L, DeSantiago J, Brown JH, Bers DM.
    Circ Res; 2003 May 02; 92(8):904-11. PubMed ID: 12676813
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