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Journal Abstract Search

321 related items for PubMed ID: 2551525

  • 1. Role of intracellular Na+ in Ca2+ overload and depressed recovery of ventricular function of reperfused ischemic rat hearts. Possible involvement of H+-Na+ and Na+-Ca2+ exchange.
    Tani M, Neely JR.
    Circ Res; 1989 Oct; 65(4):1045-56. PubMed ID: 2551525
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  • 2. Intermittent perfusion of ischemic myocardium. Possible mechanisms of protective effects on mechanical function in isolated rat heart.
    Tani M, Neely JR.
    Circulation; 1990 Aug; 82(2):536-48. PubMed ID: 2372900
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  • 3. Protective effects of dimethyl amiloride against postischemic myocardial dysfunction in rabbit hearts: phosphorus 31-nuclear magnetic resonance measurements of intracellular pH and cellular energy.
    Koike A, Akita T, Hotta Y, Takeya K, Kodama I, Murase M, Abe T, Toyama J.
    J Thorac Cardiovasc Surg; 1996 Sep; 112(3):765-75. PubMed ID: 8800166
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  • 5. Role of Na(+)/H(+) exchanger during ischemia and preconditioning in the isolated rat heart.
    Xiao XH, Allen DG.
    Circ Res; 1999 Oct 15; 85(8):723-30. PubMed ID: 10521246
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  • 10. Protective effect of SM-20550, a selective Na+ - H+ exchange inhibitor, on ischemia-reperfusion-injured hearts.
    Hotta Y, Nakagawa J, Ishikawa N, Wakida Y, Ando H, Takeya K, Ohashi N, Matsui K.
    J Cardiovasc Pharmacol; 2001 Feb 15; 37(2):143-54. PubMed ID: 11209997
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  • 13. Effects of SM-20550, a selective Na+-H+ exchange inhibitor, on the ion transport of myocardial mitochondria.
    Hotta Y, Ishikawa N, Ohashi N, Matsui K.
    Mol Cell Biochem; 2001 Mar 15; 219(1-2):83-90. PubMed ID: 11354258
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  • 14. [Effect of amiloride on the early and late stage of postischemic recovery in isolated perfused rat hearts--possible involvement of Na+/H+ and Na+/Ca2+ exchange].
    Yoshizumi M, Kitagawa T, Masuda Y, Hori T, Kawahito T, Chikugo F, Fukuta Y, Katoh I.
    Nihon Kyobu Geka Gakkai Zasshi; 1996 Sep 15; 44(9):1735-41. PubMed ID: 8911047
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  • 16. Inhibition by KB-r7943 of the reverse mode of the Na+/Ca2+ exchanger reduces Ca2+ overload in ischemic-reperfused rat hearts.
    Seki S, Taniguchi M, Takeda H, Nagai M, Taniguchi I, Mochizuki S.
    Circ J; 2002 Apr 15; 66(4):390-6. PubMed ID: 11954956
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  • 17. Overexpression of the cardiac Na+/Ca2+ exchanger increases susceptibility to ischemia/reperfusion injury in male, but not female, transgenic mice.
    Cross HR, Lu L, Steenbergen C, Philipson KD, Murphy E.
    Circ Res; 2002 Apr 15; 83(12):1215-23. PubMed ID: 9851938
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  • 18. Intracellular sodium accumulation during ischemia as the substrate for reperfusion injury.
    Imahashi K, Kusuoka H, Hashimoto K, Yoshioka J, Yamaguchi H, Nishimura T.
    Circ Res; 1999 Jun 25; 84(12):1401-6. PubMed ID: 10381892
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  • 19. Short-term inhibition of the Na-H exchanger limits acidosis and reduces ischemic injury in the rat heart.
    Schaefer S, Ramasamy R.
    Cardiovasc Res; 1997 May 25; 34(2):329-36. PubMed ID: 9205547
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  • 20. Na(+)/H(+) exchange inhibition with HOE642 improves postischemic recovery due to attenuation of Ca(2+) overload and prolonged acidosis on reperfusion.
    Strömer H, de Groot MC, Horn M, Faul C, Leupold A, Morgan JP, Scholz W, Neubauer S.
    Circulation; 2000 Jun 13; 101(23):2749-55. PubMed ID: 10851214
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