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Journal Abstract Search

202 related items for PubMed ID: 25530915

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  • 44. Early-Life Cognitive Activity Is Related to Reduced Neurodegeneration in Alzheimer Signature Regions in Late Life.
    Ko K, Byun MS, Yi D, Lee JH, Kim CH, Lee DY.
    Front Aging Neurosci; 2018; 10():70. PubMed ID: 29623037
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  • 45. Spatial Patterns of Hypometabolism and Amyloid Deposition in Variants of Alzheimer's Disease Corresponding to Brain Networks: a Prospective Cohort Study.
    Wang Y, Shi Z, Zhang N, Cai L, Li Y, Yang H, Yao S, Xing X, Ji Y, Gao S.
    Mol Imaging Biol; 2019 Feb; 21(1):140-148. PubMed ID: 29869063
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  • 48. Amyloid-β and glucose metabolism in Alzheimer's disease.
    Furst AJ, Lal RA.
    J Alzheimers Dis; 2011 Feb; 26 Suppl 3():105-16. PubMed ID: 21971455
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  • 50. Differential patterns of regional cerebral hypometabolism according to the level of cerebral amyloid deposition in patients with amnestic mild cognitive impairment.
    Jeon SY, Yi D, Byun MS, Choi HJ, Kim HJ, Lee JH, Baek H, Choe YM, Lee Y, Woo JI, Lee DY.
    Neurosci Lett; 2016 Oct 06; 632():104-8. PubMed ID: 27574728
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  • 51. Longitudinal Assessment of Amyloid-β Deposition by [18F]-Flutemetamol PET Imaging Compared With [11C]-PIB Across the Spectrum of Alzheimer's Disease.
    Hatashita S, Wakebe D, Kikuchi Y, Ichijo A.
    Front Aging Neurosci; 2019 Oct 06; 11():251. PubMed ID: 31572167
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  • 52. Diagnostic accuracy of 18 F-FDG and 11 C-PIB-PET for prediction of short-term conversion to Alzheimer's disease in subjects with mild cognitive impairment.
    Zhang S, Han D, Tan X, Feng J, Guo Y, Ding Y.
    Int J Clin Pract; 2012 Feb 06; 66(2):185-98. PubMed ID: 22257044
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  • 53. Patterns of glucose hypometabolism in Down syndrome resemble sporadic Alzheimer's disease except for the putamen.
    Zammit MD, Laymon CM, Tudorascu DL, Hartley SL, Piro-Gambetti B, Johnson SC, Stone CK, Mathis CA, Zaman SH, Klunk WE, Handen BL, Cohen AD, Christian BT.
    Alzheimers Dement (Amst); 2020 Feb 06; 12(1):e12138. PubMed ID: 33490360
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  • 55. Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography.
    Sha SJ, Ghosh PM, Lee SE, Corbetta-Rastelli C, Jagust WJ, Kornak J, Rankin KP, Grinberg LT, Vinters HV, Mendez MF, Dickson DW, Seeley WW, Gorno-Tempini M, Kramer J, Miller BL, Boxer AL, Rabinovici GD.
    Alzheimers Res Ther; 2015 Feb 06; 7(1):8. PubMed ID: 25733984
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  • 56. Cortical thickness, brain metabolic activity, and in vivo amyloid deposition in asymptomatic, middle-aged offspring of patients with late-onset Alzheimer's disease.
    Duarte-Abritta B, Villarreal MF, Abulafia C, Loewenstein D, Curiel Cid RE, Castro MN, Surace E, Sánchez SM, Vigo DE, Vázquez S, Nemeroff CB, Sevlever G, Guinjoan SM.
    J Psychiatr Res; 2018 Dec 06; 107():11-18. PubMed ID: 30308328
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  • 57. Dynamic relationships between age, amyloid-β deposition, and glucose metabolism link to the regional vulnerability to Alzheimer's disease.
    Oh H, Madison C, Baker S, Rabinovici G, Jagust W.
    Brain; 2016 Aug 06; 139(Pt 8):2275-89. PubMed ID: 27190008
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  • 58. The Precuneus - A Witness for Excessive Aβ Gathering in Alzheimer's Disease Pathology.
    Aghakhanyan G, Vergallo A, Gennaro M, Mazzarri S, Guidoccio F, Radicchi C, Ceravolo R, Tognoni G, Bonuccelli U, Volterrani D.
    Neurodegener Dis; 2018 Aug 06; 18(5-6):302-309. PubMed ID: 30814473
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  • 59. 18F-FPYBF-2, a new F-18-labelled amyloid imaging PET tracer: first experience in 61 volunteers and 55 patients with dementia.
    Higashi T, Nishii R, Kagawa S, Kishibe Y, Takahashi M, Okina T, Suzuki N, Hasegawa H, Nagahama Y, Ishizu K, Oishi N, Kimura H, Watanabe H, Ono M, Saji H, Yamauchi H.
    Ann Nucl Med; 2018 Apr 06; 32(3):206-216. PubMed ID: 29388083
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  • 60. Chemically treated plasma Aβ is a potential blood-based biomarker for screening cerebral amyloid deposition.
    Park JC, Han SH, Cho HJ, Byun MS, Yi D, Choe YM, Kang S, Jung ES, Won SJ, Kim EH, Kim YK, Lee DY, Mook-Jung I.
    Alzheimers Res Ther; 2017 Mar 22; 9(1):20. PubMed ID: 28330509
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