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Journal Abstract Search


265 related items for PubMed ID: 25590316

  • 1. Factors driving the aspirin exacerbated respiratory disease phenotype.
    Steinke JW, Borish L.
    Am J Rhinol Allergy; 2015; 29(1):35-40. PubMed ID: 25590316
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  • 2. Interleukin-4 in the Generation of the AERD Phenotype: Implications for Molecular Mechanisms Driving Therapeutic Benefit of Aspirin Desensitization.
    Steinke JW, Payne SC, Borish L.
    J Allergy (Cairo); 2012; 2012():182090. PubMed ID: 22262978
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  • 3. Eosinophils and Mast Cells in Aspirin-Exacerbated Respiratory Disease.
    Steinke JW, Payne SC, Borish L.
    Immunol Allergy Clin North Am; 2016 Nov; 36(4):719-734. PubMed ID: 27712766
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  • 4. Prominent role of IFN-γ in patients with aspirin-exacerbated respiratory disease.
    Steinke JW, Liu L, Huyett P, Negri J, Payne SC, Borish L.
    J Allergy Clin Immunol; 2013 Oct; 132(4):856-65.e1-3. PubMed ID: 23806637
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  • 5. Aspirin-Exacerbated Respiratory Disease Involves a Cysteinyl Leukotriene-Driven IL-33-Mediated Mast Cell Activation Pathway.
    Liu T, Kanaoka Y, Barrett NA, Feng C, Garofalo D, Lai J, Buchheit K, Bhattacharya N, Laidlaw TM, Katz HR, Boyce JA.
    J Immunol; 2015 Oct 15; 195(8):3537-45. PubMed ID: 26342029
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  • 6. Type 2 Cysteinyl Leukotriene Receptors Drive IL-33-Dependent Type 2 Immunopathology and Aspirin Sensitivity.
    Liu T, Barrett NA, Kanaoka Y, Yoshimoto E, Garofalo D, Cirka H, Feng C, Boyce JA.
    J Immunol; 2018 Feb 01; 200(3):915-927. PubMed ID: 29282304
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  • 9. Aspirin activation of eosinophils and mast cells: implications in the pathogenesis of aspirin-exacerbated respiratory disease.
    Steinke JW, Negri J, Liu L, Payne SC, Borish L.
    J Immunol; 2014 Jul 01; 193(1):41-7. PubMed ID: 24890720
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  • 10. Effect of TGF-β1 on eosinophils to induce cysteinyl leukotriene E4 production in aspirin-exacerbated respiratory disease.
    Choi Y, Sim S, Lee DH, Lee HR, Ban GY, Shin YS, Kim YK, Park HS.
    PLoS One; 2021 Jul 01; 16(8):e0256237. PubMed ID: 34437574
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  • 11. Prostaglandin E2 deficiency causes a phenotype of aspirin sensitivity that depends on platelets and cysteinyl leukotrienes.
    Liu T, Laidlaw TM, Katz HR, Boyce JA.
    Proc Natl Acad Sci U S A; 2013 Oct 15; 110(42):16987-92. PubMed ID: 24085850
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  • 12. Modulation by aspirin of nuclear phospho-signal transducer and activator of transcription 6 expression: Possible role in therapeutic benefit associated with aspirin desensitization.
    Steinke JW, Culp JA, Kropf E, Borish L.
    J Allergy Clin Immunol; 2009 Oct 15; 124(4):724-30.e4. PubMed ID: 19767084
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  • 18. Prostaglandin E2 resistance in granulocytes from patients with aspirin-exacerbated respiratory disease.
    Laidlaw TM, Cutler AJ, Kidder MS, Liu T, Cardet JC, Chhay H, Feng C, Boyce JA.
    J Allergy Clin Immunol; 2014 Jun 15; 133(6):1692-701.e3. PubMed ID: 24486071
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  • 19. Prostaglandin D₂: a dominant mediator of aspirin-exacerbated respiratory disease.
    Cahill KN, Bensko JC, Boyce JA, Laidlaw TM.
    J Allergy Clin Immunol; 2015 Jan 15; 135(1):245-52. PubMed ID: 25218285
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  • 20. Trial of thromboxane receptor inhibition with ifetroban: TP receptors regulate eicosanoid homeostasis in aspirin-exacerbated respiratory disease.
    Laidlaw TM, Buchheit KM, Cahill KN, Hacker J, Cho L, Cui J, Feng C, Chen CC, Le M, Israel E, Boyce JA.
    J Allergy Clin Immunol; 2023 Sep 15; 152(3):700-710.e3. PubMed ID: 37068712
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