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248 related items for PubMed ID: 25595581

  • 1. Advances in revealing the molecular targets downstream of oxidative stress-induced proapoptotic kinase signaling in diabetic embryopathy.
    Wang F, Reece EA, Yang P.
    Am J Obstet Gynecol; 2015 Aug; 213(2):125-34. PubMed ID: 25595581
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  • 2. Oxidative stress-induced JNK1/2 activation triggers proapoptotic signaling and apoptosis that leads to diabetic embryopathy.
    Li X, Weng H, Xu C, Reece EA, Yang P.
    Diabetes; 2012 Aug; 61(8):2084-92. PubMed ID: 22688338
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  • 5. c-Jun NH2-terminal kinase 1/2 and endoplasmic reticulum stress as interdependent and reciprocal causation in diabetic embryopathy.
    Li X, Xu C, Yang P.
    Diabetes; 2013 Feb; 62(2):599-608. PubMed ID: 22961085
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  • 6. Maternal hyperglycemia activates an ASK1-FoxO3a-caspase 8 pathway that leads to embryonic neural tube defects.
    Yang P, Li X, Xu C, Eckert RL, Reece EA, Zielke HR, Wang F.
    Sci Signal; 2013 Aug 27; 6(290):ra74. PubMed ID: 23982205
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  • 7. Superoxide dismutase 2 overexpression alleviates maternal diabetes-induced neural tube defects, restores mitochondrial function and suppresses cellular stress in diabetic embryopathy.
    Zhong J, Xu C, Gabbay-Benziv R, Lin X, Yang P.
    Free Radic Biol Med; 2016 Jul 27; 96():234-44. PubMed ID: 27130031
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  • 8. Deficiency of the oxidative stress-responsive kinase p70S6K1 restores autophagy and ameliorates neural tube defects in diabetic embryopathy.
    Cao S, Shen WB, Reece EA, Yang P.
    Am J Obstet Gynecol; 2020 Nov 27; 223(5):753.e1-753.e14. PubMed ID: 32416155
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  • 9. The increased activity of a transcription factor inhibits autophagy in diabetic embryopathy.
    Xu C, Chen X, Reece EA, Lu W, Yang P.
    Am J Obstet Gynecol; 2019 Jan 27; 220(1):108.e1-108.e12. PubMed ID: 30312583
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  • 10. Activation of oxidative stress signaling that is implicated in apoptosis with a mouse model of diabetic embryopathy.
    Yang P, Zhao Z, Reece EA.
    Am J Obstet Gynecol; 2008 Jan 27; 198(1):130.e1-7. PubMed ID: 18166327
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  • 11. Oxidative stress is responsible for maternal diabetes-impaired transforming growth factor beta signaling in the developing mouse heart.
    Wang F, Reece EA, Yang P.
    Am J Obstet Gynecol; 2015 May 27; 212(5):650.e1-11. PubMed ID: 25595579
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  • 12. Dominant negative FADD dissipates the proapoptotic signalosome of the unfolded protein response in diabetic embryopathy.
    Wang F, Weng H, Quon MJ, Yu J, Wang JY, Hueber AO, Yang P.
    Am J Physiol Endocrinol Metab; 2015 Nov 15; 309(10):E861-73. PubMed ID: 26419589
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  • 13. Modulation of nuclear factor-κB signaling and reduction of neural tube defects by quercetin-3-glucoside in embryos of diabetic mice.
    Tan C, Meng F, Reece EA, Zhao Z.
    Am J Obstet Gynecol; 2018 Aug 15; 219(2):197.e1-197.e8. PubMed ID: 29733843
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  • 14. Superoxide Dismutase 1 In Vivo Ameliorates Maternal Diabetes Mellitus-Induced Apoptosis and Heart Defects Through Restoration of Impaired Wnt Signaling.
    Wang F, Fisher SA, Zhong J, Wu Y, Yang P.
    Circ Cardiovasc Genet; 2015 Oct 15; 8(5):665-76. PubMed ID: 26232087
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  • 15. Oxidation-triggered c-Jun N-terminal kinase (JNK) and p38 mitogen-activated protein (MAP) kinase pathways for apoptosis in human leukaemic cells stimulated by epigallocatechin-3-gallate (EGCG): a distinct pathway from those of chemically induced and receptor-mediated apoptosis.
    Saeki K, Kobayashi N, Inazawa Y, Zhang H, Nishitoh H, Ichijo H, Saeki K, Isemura M, Yuo A.
    Biochem J; 2002 Dec 15; 368(Pt 3):705-20. PubMed ID: 12206715
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  • 16. Oxidative stress-induced miR-27a targets the redox gene nuclear factor erythroid 2-related factor 2 in diabetic embryopathy.
    Zhao Y, Dong D, Reece EA, Wang AR, Yang P.
    Am J Obstet Gynecol; 2018 Jan 15; 218(1):136.e1-136.e10. PubMed ID: 29100869
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  • 17. Type 2 diabetes mellitus induces congenital heart defects in murine embryos by increasing oxidative stress, endoplasmic reticulum stress, and apoptosis.
    Wu Y, Reece EA, Zhong J, Dong D, Shen WB, Harman CR, Yang P.
    Am J Obstet Gynecol; 2016 Sep 15; 215(3):366.e1-366.e10. PubMed ID: 27038779
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  • 20. Dissociation of Akt1 from its negative regulator JIP1 is mediated through the ASK1-MEK-JNK signal transduction pathway during metabolic oxidative stress: a negative feedback loop.
    Song JJ, Lee YJ.
    J Cell Biol; 2005 Jul 04; 170(1):61-72. PubMed ID: 15998799
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