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Journal Abstract Search

271 related items for PubMed ID: 25631932

  • 1. Anatomical evidence that the uninjured adjacent L4 nerve plays a significant role in the development of peripheral neuropathic pain after L5 spinal nerve ligation in rats.
    Shehab S, Anwer M, Galani D, Abdulkarim A, Al-Nuaimi K, Al-Baloushi A, Tariq S, Nagelkerke N, Ljubisavljevic M.
    J Comp Neurol; 2015 Aug 15; 523(12):1731-47. PubMed ID: 25631932
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  • 2. Fifth lumbar spinal nerve injury causes neurochemical changes in corresponding as well as adjacent spinal segments: a possible mechanism underlying neuropathic pain.
    Shehab SA.
    J Chem Neuroanat; 2014 Jan 15; 55():38-50. PubMed ID: 24394408
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  • 3. Unmyelinated primary afferents from adjacent spinal nerves intermingle in the spinal dorsal horn: a possible mechanism contributing to neuropathic pain.
    Shehab SA, Al-Marashda K, Al-Zahmi A, Abdul-Kareem A, Al-Sultan MA.
    Brain Res; 2008 May 07; 1208():111-9. PubMed ID: 18395190
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  • 4. The role of uninjured nerve in spinal nerve ligated rats points to an improved animal model of neuropathic pain.
    Lee DH, Iyengar S, Lodge D.
    Eur J Pain; 2003 May 07; 7(5):473-9. PubMed ID: 12935800
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  • 5. Perineural application of resiniferatoxin on uninjured L3 and L4 nerves completely alleviates thermal and mechanical hypersensitivity following L5 nerve injury in rats.
    Javed H, Rehmathulla S, Tariq S, Emerald BS, Ljubisavljevic M, Shehab S.
    J Comp Neurol; 2020 Sep 01; 528(13):2195-2217. PubMed ID: 32064609
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  • 6. A single GABA neuron receives contacts from myelinated primary afferents of two adjacent peripheral nerves. A possible role in neuropathic pain.
    Shehab S, Rehmathulla S, Javed H.
    J Comp Neurol; 2018 Dec 15; 526(18):2984-2999. PubMed ID: 30069880
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  • 8. L6 spinal nerve ligation produces prolonged development of mechanical allodynia and gradual increase of GFAP on ipsilateral dorsal horn.
    Wang J, Zeng L, Zhou Q, Xu Y, Pu S, Jiang W, Zhang X, Du D.
    Acta Neurochir (Wien); 2013 May 15; 155(5):935-40. PubMed ID: 23355062
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  • 9. Role of mitogen-activated protein kinase activation in injured and intact primary afferent neurons for mechanical and heat hypersensitivity after spinal nerve ligation.
    Obata K, Yamanaka H, Kobayashi K, Dai Y, Mizushima T, Katsura H, Fukuoka T, Tokunaga A, Noguchi K.
    J Neurosci; 2004 Nov 10; 24(45):10211-22. PubMed ID: 15537893
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  • 10. The role of uninjured C-afferents and injured afferents in the generation of mechanical hypersensitivity after partial peripheral nerve injury in the rat.
    Jang JH, Kim KH, Nam TS, Lee WT, Park KA, Kim DW, Leem JW.
    Exp Neurol; 2007 Mar 10; 204(1):288-98. PubMed ID: 17184773
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  • 11. Acute and chronic sectioning of fifth lumbar spinal nerve has equivalent effects on the primary afferents of sciatic nerve in rat spinal cord.
    Shehab SA.
    J Comp Neurol; 2009 Dec 01; 517(4):481-92. PubMed ID: 19790268
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  • 12. Brain-derived neurotrophic factor increases in the uninjured dorsal root ganglion neurons in selective spinal nerve ligation model.
    Fukuoka T, Kondo E, Dai Y, Hashimoto N, Noguchi K.
    J Neurosci; 2001 Jul 01; 21(13):4891-900. PubMed ID: 11425916
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  • 16. Spinal nerve ligation decreases γ-aminobutyric acidB receptors on specific populations of immunohistochemically identified neurons in L5 dorsal root ganglion of the rat.
    Engle MP, Merrill MA, Marquez De Prado B, Hammond DL.
    J Comp Neurol; 2012 Jun 01; 520(8):1663-77. PubMed ID: 22120979
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  • 17. Do central terminals of intact myelinated primary afferents sprout into the superficial dorsal horn of rat spinal cord after injury to a neighboring peripheral nerve?
    Shehab SA, Spike RC, Todd AJ.
    J Comp Neurol; 2004 Jun 28; 474(3):427-37. PubMed ID: 15174085
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  • 18. Contribution of the spinal cord BDNF to the development of neuropathic pain by activation of the NR2B-containing NMDA receptors in rats with spinal nerve ligation.
    Geng SJ, Liao FF, Dang WH, Ding X, Liu XD, Cai J, Han JS, Wan Y, Xing GG.
    Exp Neurol; 2010 Apr 28; 222(2):256-66. PubMed ID: 20079352
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