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156 related items for PubMed ID: 25655931

  • 1.
    ; . PubMed ID:
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  • 2. Transitions of protein traffic from cardiac ER to junctional SR.
    Sleiman NH, McFarland TP, Jones LR, Cala SE.
    J Mol Cell Cardiol; 2015 Apr; 81():34-45. PubMed ID: 25640161
    [Abstract] [Full Text] [Related]

  • 3. The role of calsequestrin, triadin, and junctin in conferring cardiac ryanodine receptor responsiveness to luminal calcium.
    Györke I, Hester N, Jones LR, Györke S.
    Biophys J; 2004 Apr; 86(4):2121-8. PubMed ID: 15041652
    [Abstract] [Full Text] [Related]

  • 4. Junctional sarcoplasmic reticulum transmembrane proteins in the heart.
    Müller FU, Kirchhefer U, Begrow F, Reinke U, Neumann J, Schmitz W.
    Basic Res Cardiol; 2002 Apr; 97 Suppl 1():I52-5. PubMed ID: 12479235
    [Abstract] [Full Text] [Related]

  • 5. Phosphorylation of skeletal muscle calsequestrin enhances its Ca2+ binding capacity and promotes its association with junctin.
    Beard NA, Wei L, Cheung SN, Kimura T, Varsányi M, Dulhunty AF.
    Cell Calcium; 2008 Oct; 44(4):363-73. PubMed ID: 19230141
    [Abstract] [Full Text] [Related]

  • 6. Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane.
    Zhang L, Kelley J, Schmeisser G, Kobayashi YM, Jones LR.
    J Biol Chem; 1997 Sep 12; 272(37):23389-97. PubMed ID: 9287354
    [Abstract] [Full Text] [Related]

  • 7. Overexpression of junctin causes adaptive changes in cardiac myocyte Ca(2+) signaling.
    Kirchhefer U, Hanske G, Jones LR, Justus I, Kaestner L, Lipp P, Schmitz W, Neumann J.
    Cell Calcium; 2006 Feb 12; 39(2):131-42. PubMed ID: 16289269
    [Abstract] [Full Text] [Related]

  • 8. Altered stored calcium release in skeletal myotubes deficient of triadin and junctin.
    Wang Y, Li X, Duan H, Fulton TR, Eu JP, Meissner G.
    Cell Calcium; 2009 Jan 12; 45(1):29-37. PubMed ID: 18620751
    [Abstract] [Full Text] [Related]

  • 9. Sarcoplasmic reticulum calcium overloading in junctin deficiency enhances cardiac contractility but increases ventricular automaticity.
    Yuan Q, Fan GC, Dong M, Altschafl B, Diwan A, Ren X, Hahn HH, Zhao W, Waggoner JR, Jones LR, Jones WK, Bers DM, Dorn GW, Wang HS, Valdivia HH, Chu G, Kranias EG.
    Circulation; 2007 Jan 23; 115(3):300-9. PubMed ID: 17224479
    [Abstract] [Full Text] [Related]

  • 10. Triadin/Junctin double null mouse reveals a differential role for Triadin and Junctin in anchoring CASQ to the jSR and regulating Ca(2+) homeostasis.
    Boncompagni S, Thomas M, Lopez JR, Allen PD, Yuan Q, Kranias EG, Franzini-Armstrong C, Perez CF.
    PLoS One; 2012 Jan 23; 7(7):e39962. PubMed ID: 22768324
    [Abstract] [Full Text] [Related]

  • 11. On the role of junctin in cardiac Ca2+ handling, contractility, and heart failure.
    Gergs U, Berndt T, Buskase J, Jones LR, Kirchhefer U, Müller FU, Schlüter KD, Schmitz W, Neumann J.
    Am J Physiol Heart Circ Physiol; 2007 Jul 23; 293(1):H728-34. PubMed ID: 17400717
    [Abstract] [Full Text] [Related]

  • 12. Triadin binding to the C-terminal luminal loop of the ryanodine receptor is important for skeletal muscle excitation contraction coupling.
    Goonasekera SA, Beard NA, Groom L, Kimura T, Lyfenko AD, Rosenfeld A, Marty I, Dulhunty AF, Dirksen RT.
    J Gen Physiol; 2007 Oct 23; 130(4):365-78. PubMed ID: 17846166
    [Abstract] [Full Text] [Related]

  • 13. Structural alterations in cardiac calcium release units resulting from overexpression of junctin.
    Zhang L, Franzini-Armstrong C, Ramesh V, Jones LR.
    J Mol Cell Cardiol; 2001 Feb 23; 33(2):233-47. PubMed ID: 11162129
    [Abstract] [Full Text] [Related]

  • 14. Junctin and triadin each activate skeletal ryanodine receptors but junctin alone mediates functional interactions with calsequestrin.
    Wei L, Gallant EM, Dulhunty AF, Beard NA.
    Int J Biochem Cell Biol; 2009 Nov 23; 41(11):2214-24. PubMed ID: 19398037
    [Abstract] [Full Text] [Related]

  • 15. Age-dependent biochemical and contractile properties in atrium of transgenic mice overexpressing junctin.
    Kirchhefer U, Baba HA, Hanske G, Jones LR, Kirchhof P, Schmitz W, Neumann J.
    Am J Physiol Heart Circ Physiol; 2004 Nov 23; 287(5):H2216-25. PubMed ID: 15205169
    [Abstract] [Full Text] [Related]

  • 16. Ablation of junctin or triadin is associated with increased cardiac injury following ischaemia/reperfusion.
    Cai WF, Pritchard T, Florea S, Lam CK, Han P, Zhou X, Yuan Q, Lehnart SE, Allen PD, Kranias EG.
    Cardiovasc Res; 2012 May 01; 94(2):333-41. PubMed ID: 22411973
    [Abstract] [Full Text] [Related]

  • 17. Sarcoplasmic reticulum Ca2+ release in neonatal rat cardiac myocytes.
    Gergs U, Kirchhefer U, Buskase J, Kiele-Dunsche K, Buchwalow IB, Jones LR, Schmitz W, Traub O, Neumann J.
    J Mol Cell Cardiol; 2011 Nov 01; 51(5):682-8. PubMed ID: 21871897
    [Abstract] [Full Text] [Related]

  • 18. Assembly and dynamics of proteins of the longitudinal and junctional sarcoplasmic reticulum in skeletal muscle cells.
    Cusimano V, Pampinella F, Giacomello E, Sorrentino V.
    Proc Natl Acad Sci U S A; 2009 Mar 24; 106(12):4695-700. PubMed ID: 19261851
    [Abstract] [Full Text] [Related]

  • 19. Control of muscle ryanodine receptor calcium release channels by proteins in the sarcoplasmic reticulum lumen.
    Beard NA, Wei L, Dulhunty AF.
    Clin Exp Pharmacol Physiol; 2009 Mar 24; 36(3):340-5. PubMed ID: 19278523
    [Abstract] [Full Text] [Related]

  • 20. Impaired relaxation in transgenic mice overexpressing junctin.
    Kirchhefer U, Neumann J, Bers DM, Buchwalow IB, Fabritz L, Hanske G, Justus I, Riemann B, Schmitz W, Jones LR.
    Cardiovasc Res; 2003 Aug 01; 59(2):369-79. PubMed ID: 12909320
    [Abstract] [Full Text] [Related]

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