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Journal Abstract Search

313 related items for PubMed ID: 25670011

  • 1. IgG4 inhibits peanut-induced basophil and mast cell activation in peanut-tolerant children sensitized to peanut major allergens.
    Santos AF, James LK, Bahnson HT, Shamji MH, Couto-Francisco NC, Islam S, Houghton S, Clark AT, Stephens A, Turcanu V, Durham SR, Gould HJ, Lack G.
    J Allergy Clin Immunol; 2015 May; 135(5):1249-56. PubMed ID: 25670011
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  • 2. Peanut diversity and specific activity are the dominant IgE characteristics for effector cell activation in children.
    Hemmings O, Niazi U, Kwok M, James LK, Lack G, Santos AF.
    J Allergy Clin Immunol; 2021 Aug; 148(2):495-505.e14. PubMed ID: 33675817
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  • 3. Ara h 2-Specific IgE Presence Rather Than Its Function Is the Best Predictor of Mast Cell Activation in Children.
    Ji C, Huang Y, Yeung LH, Hemmings O, Jama Z, Kwok M, Lack G, Santos AF.
    J Allergy Clin Immunol Pract; 2023 Apr; 11(4):1154-1161.e3. PubMed ID: 36581066
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  • 5. IgE, but not IgG4, antibodies to Ara h 2 distinguish peanut allergy from asymptomatic peanut sensitization.
    Hong X, Caruso D, Kumar R, Liu R, Liu X, Wang G, Pongracic JA, Wang X.
    Allergy; 2012 Dec; 67(12):1538-46. PubMed ID: 23094689
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  • 6. IgE to epitopes of Ara h 2 enhance the diagnostic accuracy of Ara h 2-specific IgE.
    Santos AF, Barbosa-Morais NL, Hurlburt BK, Ramaswamy S, Hemmings O, Kwok M, O'Rourke C, Bahnson HT, Cheng H, James L, Gould HJ, Sutton BJ, Maleki SJ, Lack G.
    Allergy; 2020 Sep; 75(9):2309-2318. PubMed ID: 32248566
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  • 7. Ara h 2 is the dominant peanut allergen despite similarities with Ara h 6.
    Hemmings O, Du Toit G, Radulovic S, Lack G, Santos AF.
    J Allergy Clin Immunol; 2020 Sep; 146(3):621-630.e5. PubMed ID: 32298698
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  • 8. The clinical and immunological effects of Pru p 3 sublingual immunotherapy on peach and peanut allergy in patients with systemic reactions.
    Gomez F, Bogas G, Gonzalez M, Campo P, Salas M, Diaz-Perales A, Rodriguez MJ, Prieto A, Barber D, Blanca M, Torres MJ, Mayorga C.
    Clin Exp Allergy; 2017 Mar; 47(3):339-350. PubMed ID: 28160513
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  • 10. Correlations between basophil activation, allergen-specific IgE with outcome and severity of oral food challenges.
    Song Y, Wang J, Leung N, Wang LX, Lisann L, Sicherer SH, Scurlock AM, Pesek R, Perry TT, Jones SM, Li XM.
    Ann Allergy Asthma Immunol; 2015 Apr; 114(4):319-26. PubMed ID: 25841330
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  • 11. Contribution of Ara h 2 to peanut-specific, immunoglobulin E-mediated, cell activation.
    McDermott RA, Porterfield HS, El Mezayen R, Burks AW, Pons L, Schlichting DG, Solomon B, Redzic JS, Harbeck RJ, Duncan MW, Hansen KC, Dreskin SC.
    Clin Exp Allergy; 2007 May; 37(5):752-63. PubMed ID: 17456223
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  • 15. Analysis of cytokine production by peanut-reactive T cells identifies residual Th2 effectors in highly allergic children who received peanut oral immunotherapy.
    Wisniewski JA, Commins SP, Agrawal R, Hulse KE, Yu MD, Cronin J, Heymann PW, Pomes A, Platts-Mills TA, Workman L, Woodfolk JA.
    Clin Exp Allergy; 2015 Jul; 45(7):1201-13. PubMed ID: 25823600
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  • 17. Basophil allergen threshold sensitivity, CD-sens, IgE-sensitization and DBPCFC in peanut-sensitized children.
    Glaumann S, Nopp A, Johansson SG, Rudengren M, Borres MP, Nilsson C.
    Allergy; 2012 Feb; 67(2):242-7. PubMed ID: 22126416
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