These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

396 related items for PubMed ID: 25772296

  • 1. Contribution of sodium channel neuronal isoform Nav1.1 to late sodium current in ventricular myocytes from failing hearts.
    Mishra S, Reznikov V, Maltsev VA, Undrovinas NA, Sabbah HN, Undrovinas A.
    J Physiol; 2015 Mar 15; 593(6):1409-27. PubMed ID: 25772296
    [Abstract] [Full Text] [Related]

  • 2. Chronic heart failure slows late sodium current in human and canine ventricular myocytes: implications for repolarization variability.
    Maltsev VA, Silverman N, Sabbah HN, Undrovinas AI.
    Eur J Heart Fail; 2007 Mar 15; 9(3):219-27. PubMed ID: 17067855
    [Abstract] [Full Text] [Related]

  • 3. A multi-modal composition of the late Na+ current in human ventricular cardiomyocytes.
    Maltsev VA, Undrovinas AI.
    Cardiovasc Res; 2006 Jan 15; 69(1):116-27. PubMed ID: 16223473
    [Abstract] [Full Text] [Related]

  • 4. Post-transcriptional silencing of SCN1B and SCN2B genes modulates late sodium current in cardiac myocytes from normal dogs and dogs with chronic heart failure.
    Mishra S, Undrovinas NA, Maltsev VA, Reznikov V, Sabbah HN, Undrovinas A.
    Am J Physiol Heart Circ Physiol; 2011 Oct 15; 301(4):H1596-605. PubMed ID: 21705762
    [Abstract] [Full Text] [Related]

  • 5. Calpain inhibition reduces amplitude and accelerates decay of the late sodium current in ventricular myocytes from dogs with chronic heart failure.
    Undrovinas A, Maltsev VA, Sabbah HN.
    PLoS One; 2013 Oct 15; 8(4):e54436. PubMed ID: 23596505
    [Abstract] [Full Text] [Related]

  • 6. Contribution of the neuronal sodium channel NaV1.8 to sodium- and calcium-dependent cellular proarrhythmia.
    Bengel P, Ahmad S, Tirilomis P, Trum M, Dybkova N, Wagner S, Maier LS, Hasenfuß G, Sossalla S.
    J Mol Cell Cardiol; 2020 Jul 15; 144():35-46. PubMed ID: 32418916
    [Abstract] [Full Text] [Related]

  • 7. Differential regulation of sodium channels as a novel proarrhythmic mechanism in the human failing heart.
    Dybkova N, Ahmad S, Pabel S, Tirilomis P, Hartmann N, Fischer TH, Bengel P, Tirilomis T, Ljubojevic S, Renner A, Gummert J, Ellenberger D, Wagner S, Frey N, Maier LS, Streckfuss-Bömeke K, Hasenfuss G, Sossalla S.
    Cardiovasc Res; 2018 Nov 01; 114(13):1728-1737. PubMed ID: 29931291
    [Abstract] [Full Text] [Related]

  • 8. Basal late sodium current is a significant contributor to the duration of action potential of guinea pig ventricular myocytes.
    Song Y, Belardinelli L.
    Physiol Rep; 2017 May 01; 5(10):e13295. PubMed ID: 28554967
    [Abstract] [Full Text] [Related]

  • 9. Enhanced Depolarization Drive in Failing Rabbit Ventricular Myocytes: Calcium-Dependent and β-Adrenergic Effects on Late Sodium, L-Type Calcium, and Sodium-Calcium Exchange Currents.
    Hegyi B, Morotti S, Liu C, Ginsburg KS, Bossuyt J, Belardinelli L, Izu LT, Chen-Izu Y, Bányász T, Grandi E, Bers DM.
    Circ Arrhythm Electrophysiol; 2019 Mar 01; 12(3):e007061. PubMed ID: 30879336
    [Abstract] [Full Text] [Related]

  • 10. Role of late sodium current as a potential arrhythmogenic mechanism in the progression of pressure-induced heart disease.
    Toischer K, Hartmann N, Wagner S, Fischer TH, Herting J, Danner BC, Sag CM, Hund TJ, Mohler PJ, Belardinelli L, Hasenfuss G, Maier LS, Sossalla S.
    J Mol Cell Cardiol; 2013 Aug 01; 61():111-22. PubMed ID: 23570977
    [Abstract] [Full Text] [Related]

  • 11. About half of the late sodium current in cardiac myocytes from dog ventricle is due to non-cardiac-type Na(+) channels.
    Biet M, Barajas-Martínez H, Ton AT, Delabre JF, Morin N, Dumaine R.
    J Mol Cell Cardiol; 2012 Nov 01; 53(5):593-8. PubMed ID: 22759452
    [Abstract] [Full Text] [Related]

  • 12. Activation of reverse Na+-Ca2+ exchanger by skeletal Na+ channel isoform increases excitation-contraction coupling efficiency in rabbit cardiomyocytes.
    Torres NS.
    Am J Physiol Heart Circ Physiol; 2021 Feb 01; 320(2):H593-H603. PubMed ID: 33275521
    [Abstract] [Full Text] [Related]

  • 13. Late sodium current contributes to diastolic cell Ca2+ accumulation in chronic heart failure.
    Undrovinas NA, Maltsev VA, Belardinelli L, Sabbah HN, Undrovinas A.
    J Physiol Sci; 2010 Jul 01; 60(4):245-57. PubMed ID: 20490740
    [Abstract] [Full Text] [Related]

  • 14. Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts.
    Lin X, O'Malley H, Chen C, Auerbach D, Foster M, Shekhar A, Zhang M, Coetzee W, Jalife J, Fishman GI, Isom L, Delmar M.
    J Physiol; 2015 Mar 15; 593(6):1389-407. PubMed ID: 25772295
    [Abstract] [Full Text] [Related]

  • 15. A computational modelling approach combined with cellular electrophysiology data provides insights into the therapeutic benefit of targeting the late Na+ current.
    Yang PC, Song Y, Giles WR, Horvath B, Chen-Izu Y, Belardinelli L, Rajamani S, Clancy CE.
    J Physiol; 2015 Mar 15; 593(6):1429-42. PubMed ID: 25545172
    [Abstract] [Full Text] [Related]

  • 16. Reduced availability of voltage-gated sodium channels by depolarization or blockade by tetrodotoxin boosts burst firing and catecholamine release in mouse chromaffin cells.
    Vandael DH, Ottaviani MM, Legros C, Lefort C, Guérineau NC, Allio A, Carabelli V, Carbone E.
    J Physiol; 2015 Feb 15; 593(4):905-27. PubMed ID: 25620605
    [Abstract] [Full Text] [Related]

  • 17. Novel, ultraslow inactivating sodium current in human ventricular cardiomyocytes.
    Maltsev VA, Sabbah HN, Higgins RS, Silverman N, Lesch M, Undrovinas AI.
    Circulation; 1998 Dec 08; 98(23):2545-52. PubMed ID: 9843461
    [Abstract] [Full Text] [Related]

  • 18. Intracellular calcium attenuates late current conducted by mutant human cardiac sodium channels.
    Potet F, Beckermann TM, Kunic JD, George AL.
    Circ Arrhythm Electrophysiol; 2015 Aug 08; 8(4):933-41. PubMed ID: 26022185
    [Abstract] [Full Text] [Related]

  • 19. Balance Between Rapid Delayed Rectifier K+ Current and Late Na+ Current on Ventricular Repolarization: An Effective Antiarrhythmic Target?
    Hegyi B, Chen-Izu Y, Izu LT, Rajamani S, Belardinelli L, Bers DM, Bányász T.
    Circ Arrhythm Electrophysiol; 2020 Apr 08; 13(4):e008130. PubMed ID: 32202931
    [Abstract] [Full Text] [Related]

  • 20. Ranolazine improves abnormal repolarization and contraction in left ventricular myocytes of dogs with heart failure by inhibiting late sodium current.
    Undrovinas AI, Belardinelli L, Undrovinas NA, Sabbah HN.
    J Cardiovasc Electrophysiol; 2006 May 08; 17 Suppl 1(Suppl 1):S169-S177. PubMed ID: 16686675
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 20.