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Journal Abstract Search
912 related items for PubMed ID: 2580009
1. The target minor H antigen for F1 cytotoxic T lymphocytes induced by Igh-congenic parental spleen cells is coded for by gene linked to H-2. Ishikawa H, Kubota E, Suzuki H, Saito K. J Immunol; 1985 May; 134(5):2953-9. PubMed ID: 2580009 [Abstract] [Full Text] [Related]
2. Cytotoxic T lymphocyte response to minor H-42a alloantigen in H-42b mice: clonal inactivation of the precursor cytotoxic T lymphocytes by veto-like spleen cells that express the H-42a antigen. Ishikawa H, Hino T, Kato H, Suzuki H, Saito K. J Immunol; 1986 Oct 01; 137(7):2080-8. PubMed ID: 3489752 [Abstract] [Full Text] [Related]
3. Modulation of F1 cytotoxic potentials by GvHR: role and mode of action of non-MHC genes that determine the hybrid resistance to GvHR-associated suppression of F1 cytotoxic potential. Ishikawa H, Kubota E, Saito K. J Immunol; 1984 May 01; 132(5):2218-25. PubMed ID: 6232314 [Abstract] [Full Text] [Related]
5. Modulation of F1 cytotoxic potentials by GvHR. Host- and donor-derived cytotoxic lymphocytes arise in the unirradiated F1 host spleens under the condition of GvHR-associated immunosuppression. Kubota E, Ishikawa H, Saito K. J Immunol; 1983 Sep 01; 131(3):1142-8. PubMed ID: 6604089 [Abstract] [Full Text] [Related]
6. Cytotoxic T lymphocyte response to minor H-43a alloantigen in H-43b mice. Privileged H-2Kb restriction to the response is not due to immunodominance or epistatic effect but due to Ir gene function of H-2Kb itself. Hino T, Ishikawa H, Saito K. J Immunol; 1988 Jan 01; 140(1):44-51. PubMed ID: 3121741 [Abstract] [Full Text] [Related]
8. Induction and characterization of minor histocompatibility antigens. Specific primary cytotoxic T lymphocyte responses in vitro. Ando K, Nakashima I, Nagase F, Isobe K, Kawashima K, Hasegawa Y, Yoshida T, Iwamoto T, Hasegawa T, Muro Y. J Immunol; 1988 Feb 01; 140(3):723-9. PubMed ID: 2448374 [Abstract] [Full Text] [Related]
12. Genetic control of the induction of cytolytic T lymphocyte responses to AKR/Gross viral leukemias. II. Negative control by the Fv-1 locus in AKR mice of responder H-2b haplotype. Green WR. J Immunol; 1984 May 01; 132(5):2665-71. PubMed ID: 6425410 [Abstract] [Full Text] [Related]
13. Abrogation of hybrid resistance to bone marrow engraftment by graft-vs-host-induced immune deficiency. Hakim FT, Shearer GM. J Immunol; 1986 Nov 15; 137(10):3109-16. PubMed ID: 3095424 [Abstract] [Full Text] [Related]
15. Investigations into the nature of Igh-V region-restricted T cell interactions by using antibodies to antigens on methylcholanthrene-induced sarcomas. I. Analysis of an Igh-V-restricted suppressor-inducer factor. Flood PM. J Immunol; 1985 Mar 15; 134(3):1665-72. PubMed ID: 2578515 [Abstract] [Full Text] [Related]
17. Effect of graft-versus-host disease on anti-tumor immunity. Schreiber KL, Forman J. J Immunol; 1990 Mar 01; 144(5):2018-26. PubMed ID: 1968493 [Abstract] [Full Text] [Related]
18. Genetic control of the induction of cytolytic T lymphocyte responses to AKR/Gross viral leukemias. I. H-2-encoded dominant gene control. Green WR. J Immunol; 1984 May 01; 132(5):2658-64. PubMed ID: 6425409 [Abstract] [Full Text] [Related]
19. Cytotoxic T lymphocyte responses to minor H-43 alloantigens in H-43a and H-43b mice. Both anti-H-43b and anti-H-43a CTL activities are generated exclusively in the context of the same H-2Kb restriction element. Ishikawa H, Kusakabe A, Hayakawa J, Hino T. J Immunol; 1988 Nov 01; 141(9):2918-23. PubMed ID: 3139769 [Abstract] [Full Text] [Related]