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PUBMED FOR HANDHELDS

Journal Abstract Search


109 related items for PubMed ID: 2580483

  • 1. Role of interferon in the resistance of C3H/HeJ mice to infection with herpes simplex virus.
    Chmielarczyk W, Domke I, Kirchner H.
    Antiviral Res; 1985 Feb; 5(1):55-9. PubMed ID: 2580483
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  • 2. The role of natural killer cells and interferon in resistance to acute infection of mice with herpes simplex virus type 1.
    Bukowski JF, Welsh RM.
    J Immunol; 1986 May 01; 136(9):3481-5. PubMed ID: 2420892
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  • 5. Differences in susceptibility to herpes simplex virus infection of inbred strains of mice.
    Kirchner H, Kochen M, Munk K, Hirt HM, Mergenhagen SE, Rosenstreich DL.
    IARC Sci Publ (1971); 1978 May 01; (24 Pt 2):783-8. PubMed ID: 221403
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  • 8. Injection of anti-thy-1.2 serum breaks genetic resistance of mice against herpes simplex virus.
    Chmielarczyk W, Engler H, Ernst R, Opitz U, Kirchner H.
    J Gen Virol; 1985 May 01; 66 ( Pt 5)():1087-94. PubMed ID: 2582081
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  • 10. Experimental infection of inbred mice with herpes simplex virus. II. Interferon production and activation of natural killer cells in the peritoneal exudate.
    Engler H, Zawatzky R, Goldbach A, Schröder CH, Weyand C, Hämmerling GJ, Kirchner H.
    J Gen Virol; 1981 Jul 01; 55(Pt 1):25-30. PubMed ID: 6170726
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  • 15. Role of NK cells in protection of mice against herpes simplex virus-1 infection.
    Rager-Zisman B, Quan PC, Rosner M, Moller JR, Bloom BR.
    J Immunol; 1987 Feb 01; 138(3):884-8. PubMed ID: 3805719
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  • 16. Re-evaluating the role of natural killer cells in innate resistance to herpes simplex virus type 1.
    Halford WP, Maender JL, Gebhardt BM.
    Virol J; 2005 Jul 17; 2():56. PubMed ID: 16022737
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  • 17. NK cells and interferon as mediators of biological response modifier-mediated protection against MCMV and HSV-2 infection in normal and immunocompromised mice.
    Kunder SC, Morahan PS.
    Ann N Y Acad Sci; 1993 Jun 23; 685():618-9. PubMed ID: 7689813
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  • 20. Macrophages from endotoxin-hyporesponsive (Lpsd) C3H/HeJ mice are permissive for vesicular stomatitis virus because of reduced levels of endogenous interferon: possible mechanism for natural resistance to virus infection.
    Vogel SN, Fertsch D.
    J Virol; 1987 Mar 23; 61(3):812-8. PubMed ID: 2433468
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