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Journal Abstract Search

197 related items for PubMed ID: 26025071

  • 1. Shigella flexneri cell-to-cell spread, and growth and inflammation in mice, is limited by the outer membrane protease IcsP.
    Tran EN, Attridge SR, Teh MY, Morona R.
    FEMS Microbiol Lett; 2015 Jun; 362(12):fnv088. PubMed ID: 26025071
    [Abstract] [Full Text] [Related]

  • 2. Outer membrane protein A (OmpA): a new player in shigella flexneri protrusion formation and inter-cellular spreading.
    Ambrosi C, Pompili M, Scribano D, Zagaglia C, Ripa S, Nicoletti M.
    PLoS One; 2012 Jun; 7(11):e49625. PubMed ID: 23166731
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  • 3. Disruption of IcsP, the major Shigella protease that cleaves IcsA, accelerates actin-based motility.
    Shere KD, Sallustio S, Manessis A, D'Aversa TG, Goldberg MB.
    Mol Microbiol; 1997 Aug; 25(3):451-62. PubMed ID: 9302008
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  • 4. Genetic modulation of Shigella flexneri 2a lipopolysaccharide O antigen modal chain length reveals that it has been optimized for virulence.
    Morona R, Daniels C, Van Den Bosch L.
    Microbiology (Reading); 2003 Apr; 149(Pt 4):925-939. PubMed ID: 12686635
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  • 5. Cardiolipin Synthesis and Outer Membrane Localization Are Required for Shigella flexneri Virulence.
    Rossi RM, Yum L, Agaisse H, Payne SM.
    mBio; 2017 Aug 29; 8(4):. PubMed ID: 28851846
    [Abstract] [Full Text] [Related]

  • 6. Modulation of an outer membrane protease contributes to the virulence defect of Shigella flexneri strains carrying a mutation in the virK locus.
    Wing HJ, Goldman SR, Ally S, Goldberg MB.
    Infect Immun; 2005 Feb 29; 73(2):1217-20. PubMed ID: 15664968
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  • 7. IcsA surface presentation in Shigella flexneri requires the periplasmic chaperones DegP, Skp, and SurA.
    Purdy GE, Fisher CR, Payne SM.
    J Bacteriol; 2007 Aug 29; 189(15):5566-73. PubMed ID: 17526712
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  • 8. The unipolar Shigella surface protein IcsA is targeted directly to the bacterial old pole: IcsP cleavage of IcsA occurs over the entire bacterial surface.
    Steinhauer J, Agha R, Pham T, Varga AW, Goldberg MB.
    Mol Microbiol; 1999 Apr 29; 32(2):367-77. PubMed ID: 10231492
    [Abstract] [Full Text] [Related]

  • 9. The actin-based motility defect of a Shigella flexneri rmlD rough LPS mutant is not due to loss of IcsA polarity.
    Van den Bosch L, Morona R.
    Microb Pathog; 2003 Jul 29; 35(1):11-8. PubMed ID: 12860454
    [Abstract] [Full Text] [Related]

  • 10. Actin-based motility is sufficient for bacterial membrane protrusion formation and host cell uptake.
    Monack DM, Theriot JA.
    Cell Microbiol; 2001 Sep 29; 3(9):633-47. PubMed ID: 11553015
    [Abstract] [Full Text] [Related]

  • 11. LPS unmasking of Shigella flexneri reveals preferential localisation of tagged outer membrane protease IcsP to septa and new poles.
    Tran EN, Doyle MT, Morona R.
    PLoS One; 2013 Sep 29; 8(7):e70508. PubMed ID: 23936222
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  • 13. Avirulence of rough mutants of Shigella flexneri: requirement of O antigen for correct unipolar localization of IcsA in the bacterial outer membrane.
    Sandlin RC, Lampel KA, Keasler SP, Goldberg MB, Stolzer AL, Maurelli AT.
    Infect Immun; 1995 Jan 29; 63(1):229-37. PubMed ID: 7528731
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  • 18. Contribution of the periplasmic chaperone Skp to efficient presentation of the autotransporter IcsA on the surface of Shigella flexneri.
    Wagner JK, Heindl JE, Gray AN, Jain S, Goldberg MB.
    J Bacteriol; 2009 Feb 29; 191(3):815-21. PubMed ID: 19047350
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