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Journal Abstract Search

371 related items for PubMed ID: 26035843

  • 1. Finding the most appropriate mouse model of juvenile CLN3 (Batten) disease for therapeutic studies: the importance of genetic background and gender.
    Kovács AD, Pearce DA.
    Dis Model Mech; 2015 Apr; 8(4):351-61. PubMed ID: 26035843
    [Abstract] [Full Text] [Related]

  • 2. Altered sensitivity of cerebellar granule cells to glutamate receptor overactivation in the Cln3(Δex7/8)-knock-in mouse model of juvenile neuronal ceroid lipofuscinosis.
    Finn R, Kovács AD, Pearce DA.
    Neurochem Int; 2011 May; 58(6):648-55. PubMed ID: 21315126
    [Abstract] [Full Text] [Related]

  • 3. Neurodevelopmental delay in the Cln3Deltaex7/8 mouse model for Batten disease.
    Osório NS, Sampaio-Marques B, Chan CH, Oliveira P, Pearce DA, Sousa N, Rodrigues F.
    Genes Brain Behav; 2009 Apr; 8(3):337-45. PubMed ID: 19243453
    [Abstract] [Full Text] [Related]

  • 4. Efficacy of phosphodiesterase-4 inhibitors in juvenile Batten disease (CLN3).
    Aldrich A, Bosch ME, Fallet R, Odvody J, Burkovetskaya M, Rama Rao KV, Cooper JD, Drack AV, Kielian T.
    Ann Neurol; 2016 Dec; 80(6):909-923. PubMed ID: 27804148
    [Abstract] [Full Text] [Related]

  • 5. Self-Complementary AAV9 Gene Delivery Partially Corrects Pathology Associated with Juvenile Neuronal Ceroid Lipofuscinosis (CLN3).
    Bosch ME, Aldrich A, Fallet R, Odvody J, Burkovetskaya M, Schuberth K, Fitzgerald JA, Foust KD, Kielian T.
    J Neurosci; 2016 Sep 14; 36(37):9669-82. PubMed ID: 27629717
    [Abstract] [Full Text] [Related]

  • 6. Large-scale phenotyping of an accurate genetic mouse model of JNCL identifies novel early pathology outside the central nervous system.
    Staropoli JF, Haliw L, Biswas S, Garrett L, Hölter SM, Becker L, Skosyrski S, Da Silva-Buttkus P, Calzada-Wack J, Neff F, Rathkolb B, Rozman J, Schrewe A, Adler T, Puk O, Sun M, Favor J, Racz I, Bekeredjian R, Busch DH, Graw J, Klingenspor M, Klopstock T, Wolf E, Wurst W, Zimmer A, Lopez E, Harati H, Hill E, Krause DS, Guide J, Dragileva E, Gale E, Wheeler VC, Boustany RM, Brown DE, Breton S, Ruether K, Gailus-Durner V, Fuchs H, de Angelis MH, Cotman SL.
    PLoS One; 2012 Sep 14; 7(6):e38310. PubMed ID: 22701626
    [Abstract] [Full Text] [Related]

  • 7. Retinal function in aging homozygous Cln3 (Δex7/8) knock-in mice.
    Volz C, Mirza M, Langmann T, Jägle H.
    Adv Exp Med Biol; 2014 Sep 14; 801():495-501. PubMed ID: 24664736
    [Abstract] [Full Text] [Related]

  • 8. Age-dependent alterations in neuronal activity in the hippocampus and visual cortex in a mouse model of Juvenile Neuronal Ceroid Lipofuscinosis (CLN3).
    Burkovetskaya M, Karpuk N, Kielian T.
    Neurobiol Dis; 2017 Apr 14; 100():19-29. PubMed ID: 28042098
    [Abstract] [Full Text] [Related]

  • 9. Partial correction of the CNS lysosomal storage defect in a mouse model of juvenile neuronal ceroid lipofuscinosis by neonatal CNS administration of an adeno-associated virus serotype rh.10 vector expressing the human CLN3 gene.
    Sondhi D, Scott EC, Chen A, Hackett NR, Wong AM, Kubiak A, Nelvagal HR, Pearse Y, Cotman SL, Cooper JD, Crystal RG.
    Hum Gene Ther; 2014 Mar 14; 25(3):223-39. PubMed ID: 24372003
    [Abstract] [Full Text] [Related]

  • 10. Developmental impairments of select neurotransmitter systems in brains of Cln3(Deltaex7/8) knock-in mice, an animal model of juvenile neuronal ceroid lipofuscinosis.
    Herrmann P, Druckrey-Fiskaaen C, Kouznetsova E, Heinitz K, Bigl M, Cotman SL, Schliebs R.
    J Neurosci Res; 2008 Jun 14; 86(8):1857-70. PubMed ID: 18265413
    [Abstract] [Full Text] [Related]

  • 11. Microglia in juvenile neuronal ceroid lipofuscinosis are primed toward a pro-inflammatory phenotype.
    Xiong J, Kielian T.
    J Neurochem; 2013 Oct 14; 127(2):245-58. PubMed ID: 23919525
    [Abstract] [Full Text] [Related]

  • 12. Immunomodulation with minocycline rescues retinal degeneration in juvenile neuronal ceroid lipofuscinosis mice highly susceptible to light damage.
    Dannhausen K, Möhle C, Langmann T.
    Dis Model Mech; 2018 Sep 05; 11(9):. PubMed ID: 30042155
    [Abstract] [Full Text] [Related]

  • 13. Retinal pathology and function in a Cln3 knockout mouse model of juvenile Neuronal Ceroid Lipofuscinosis (batten disease).
    Seigel GM, Lotery A, Kummer A, Bernard DJ, Greene ND, Turmaine M, Derksen T, Nussbaum RL, Davidson B, Wagner J, Mitchison HM.
    Mol Cell Neurosci; 2002 Apr 05; 19(4):515-27. PubMed ID: 11988019
    [Abstract] [Full Text] [Related]

  • 14. Caspase 1 activity influences juvenile Batten disease (CLN3) pathogenesis.
    Burkovetskaya M, Bosch ME, Karpuk N, Fallet R, Kielian T.
    J Neurochem; 2019 Mar 05; 148(5):652-668. PubMed ID: 29873075
    [Abstract] [Full Text] [Related]

  • 15. Further Characterization of the Predominant Inner Retinal Degeneration of Aging Cln3 Δex7/8 Knock-In Mice.
    Volz C, Mirza M, Langmann T, Jägle H.
    Adv Exp Med Biol; 2018 Mar 05; 1074():403-411. PubMed ID: 29721970
    [Abstract] [Full Text] [Related]

  • 16. A knock-in reporter mouse model for Batten disease reveals predominant expression of Cln3 in visual, limbic and subcortical motor structures.
    Ding SL, Tecedor L, Stein CS, Davidson BL.
    Neurobiol Dis; 2011 Feb 05; 41(2):237-48. PubMed ID: 20875858
    [Abstract] [Full Text] [Related]

  • 17. Cln3(Deltaex7/8) knock-in mice with the common JNCL mutation exhibit progressive neurologic disease that begins before birth.
    Cotman SL, Vrbanac V, Lebel LA, Lee RL, Johnson KA, Donahue LR, Teed AM, Antonellis K, Bronson RT, Lerner TJ, MacDonald ME.
    Hum Mol Genet; 2002 Oct 15; 11(22):2709-21. PubMed ID: 12374761
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