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Journal Abstract Search

247 related items for PubMed ID: 26092998

  • 1. Mouse lung development and NOX1 induction during hyperoxia are developmentally regulated and mitochondrial ROS dependent.
    Datta A, Kim GA, Taylor JM, Gugino SF, Farrow KN, Schumacker PT, Berkelhamer SK.
    Am J Physiol Lung Cell Mol Physiol; 2015 Aug 15; 309(4):L369-77. PubMed ID: 26092998
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  • 2. Sphingosine kinase 1 deficiency confers protection against hyperoxia-induced bronchopulmonary dysplasia in a murine model: role of S1P signaling and Nox proteins.
    Harijith A, Pendyala S, Reddy NM, Bai T, Usatyuk PV, Berdyshev E, Gorshkova I, Huang LS, Mohan V, Garzon S, Kanteti P, Reddy SP, Raj JU, Natarajan V.
    Am J Pathol; 2013 Oct 15; 183(4):1169-1182. PubMed ID: 23933064
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  • 6. Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury.
    Poonyagariyagorn HK, Metzger S, Dikeman D, Mercado AL, Malinina A, Calvi C, McGrath-Morrow S, Neptune ER.
    Am J Respir Cell Mol Biol; 2014 Sep 15; 51(3):380-90. PubMed ID: 24673633
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  • 12. Lack of EC-SOD worsens alveolar and vascular development in a neonatal mouse model of bleomycin-induced bronchopulmonary dysplasia and pulmonary hypertension.
    Delaney C, Wright RH, Tang JR, Woods C, Villegas L, Sherlock L, Savani RC, Abman SH, Nozik-Grayck E.
    Pediatr Res; 2015 Dec 15; 78(6):634-40. PubMed ID: 26322414
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  • 16. Targeting transglutaminase 2 partially restores extracellular matrix structure but not alveolar architecture in experimental bronchopulmonary dysplasia.
    Mižíková I, Pfeffer T, Nardiello C, Surate Solaligue DE, Steenbock H, Tatsukawa H, Silva DM, Vadász I, Herold S, Pease RJ, Iismaa SE, Hitomi K, Seeger W, Brinckmann J, Morty RE.
    FEBS J; 2018 Aug 15; 285(16):3056-3076. PubMed ID: 29935061
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  • 17. Overexpression of manganese superoxide dismutase by N-acetylcysteine in hyperoxic lung injury.
    Nagata K, Iwasaki Y, Yamada T, Yuba T, Kono K, Hosogi S, Ohsugi S, Kuwahara H, Marunaka Y.
    Respir Med; 2007 Apr 15; 101(4):800-7. PubMed ID: 17010595
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  • 20. Neonatal therapy with PF543, a sphingosine kinase 1 inhibitor, ameliorates hyperoxia-induced airway remodeling in a murine model of bronchopulmonary dysplasia.
    Ha AW, Sudhadevi T, Ebenezer DL, Fu P, Berdyshev EV, Ackerman SJ, Natarajan V, Harijith A.
    Am J Physiol Lung Cell Mol Physiol; 2020 Sep 01; 319(3):L497-L512. PubMed ID: 32697651
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