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Journal Abstract Search

1256 related items for PubMed ID: 26108573

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  • 2. Sense-encoded poly-GR dipeptide repeat proteins correlate to neurodegeneration and uniquely co-localize with TDP-43 in dendrites of repeat-expanded C9orf72 amyotrophic lateral sclerosis.
    Saberi S, Stauffer JE, Jiang J, Garcia SD, Taylor AE, Schulte D, Ohkubo T, Schloffman CL, Maldonado M, Baughn M, Rodriguez MJ, Pizzo D, Cleveland D, Ravits J.
    Acta Neuropathol; 2018 Mar; 135(3):459-474. PubMed ID: 29196813
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  • 3. Circadian sleep/wake-associated cells show dipeptide repeat protein aggregates in C9orf72-related ALS and FTLD cases.
    Dedeene L, Van Schoor E, Vandenberghe R, Van Damme P, Poesen K, Thal DR.
    Acta Neuropathol Commun; 2019 Dec 02; 7(1):189. PubMed ID: 31791419
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  • 6. Drosha inclusions are new components of dipeptide-repeat protein aggregates in FTLD-TDP and ALS C9orf72 expansion cases.
    Porta S, Kwong LK, Trojanowski JQ, Lee VM.
    J Neuropathol Exp Neurol; 2015 Apr 02; 74(4):380-7. PubMed ID: 25756586
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  • 7. p62 positive, TDP-43 negative, neuronal cytoplasmic and intranuclear inclusions in the cerebellum and hippocampus define the pathology of C9orf72-linked FTLD and MND/ALS.
    Al-Sarraj S, King A, Troakes C, Smith B, Maekawa S, Bodi I, Rogelj B, Al-Chalabi A, Hortobágyi T, Shaw CE.
    Acta Neuropathol; 2011 Dec 02; 122(6):691-702. PubMed ID: 22101323
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  • 9. Cytoplasmic poly-GA aggregates impair nuclear import of TDP-43 in C9orf72 ALS/FTLD.
    Khosravi B, Hartmann H, May S, Möhl C, Ederle H, Michaelsen M, Schludi MH, Dormann D, Edbauer D.
    Hum Mol Genet; 2017 Feb 15; 26(4):790-800. PubMed ID: 28040728
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  • 10. C9orf72 poly GA RAN-translated protein plays a key role in amyotrophic lateral sclerosis via aggregation and toxicity.
    Lee YB, Baskaran P, Gomez-Deza J, Chen HJ, Nishimura AL, Smith BN, Troakes C, Adachi Y, Stepto A, Petrucelli L, Gallo JM, Hirth F, Rogelj B, Guthrie S, Shaw CE.
    Hum Mol Genet; 2017 Dec 15; 26(24):4765-4777. PubMed ID: 28973350
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  • 11. Clinical and neuropathologic heterogeneity of c9FTD/ALS associated with hexanucleotide repeat expansion in C9ORF72.
    Murray ME, DeJesus-Hernandez M, Rutherford NJ, Baker M, Duara R, Graff-Radford NR, Wszolek ZK, Ferman TJ, Josephs KA, Boylan KB, Rademakers R, Dickson DW.
    Acta Neuropathol; 2011 Dec 15; 122(6):673-90. PubMed ID: 22083254
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  • 12. Increased prevalence of granulovacuolar degeneration in C9orf72 mutation.
    Riku Y, Duyckaerts C, Boluda S, Plu I, Le Ber I, Millecamps S, Salachas F, Brainbank NeuroCEB Neuropathology Network, Yoshida M, Ando T, Katsuno M, Sobue G, Seilhean D.
    Acta Neuropathol; 2019 Nov 15; 138(5):783-793. PubMed ID: 31144027
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  • 13. Neurodegeneration in frontotemporal lobar degeneration and motor neurone disease associated with expansions in C9orf72 is linked to TDP-43 pathology and not associated with aggregated forms of dipeptide repeat proteins.
    Davidson Y, Robinson AC, Liu X, Wu D, Troakes C, Rollinson S, Masuda-Suzukake M, Suzuki G, Nonaka T, Shi J, Tian J, Hamdalla H, Ealing J, Richardson A, Jones M, Pickering-Brown S, Snowden JS, Hasegawa M, Mann DM.
    Neuropathol Appl Neurobiol; 2016 Apr 15; 42(3):242-54. PubMed ID: 26538301
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  • 14. Prevalence of brain and spinal cord inclusions, including dipeptide repeat proteins, in patients with the C9ORF72 hexanucleotide repeat expansion: a systematic neuropathological review.
    Schipper LJ, Raaphorst J, Aronica E, Baas F, de Haan R, de Visser M, Troost D.
    Neuropathol Appl Neurobiol; 2016 Oct 15; 42(6):547-60. PubMed ID: 26373655
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  • 15. C9orf72 FTLD/ALS-associated Gly-Ala dipeptide repeat proteins cause neuronal toxicity and Unc119 sequestration.
    May S, Hornburg D, Schludi MH, Arzberger T, Rentzsch K, Schwenk BM, Grässer FA, Mori K, Kremmer E, Banzhaf-Strathmann J, Mann M, Meissner F, Edbauer D.
    Acta Neuropathol; 2014 Oct 15; 128(4):485-503. PubMed ID: 25120191
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  • 16. An MND/ALS phenotype associated with C9orf72 repeat expansion: abundant p62-positive, TDP-43-negative inclusions in cerebral cortex, hippocampus and cerebellum but without associated cognitive decline.
    Troakes C, Maekawa S, Wijesekera L, Rogelj B, Siklós L, Bell C, Smith B, Newhouse S, Vance C, Johnson L, Hortobágyi T, Shatunov A, Al-Chalabi A, Leigh N, Shaw CE, King A, Al-Sarraj S.
    Neuropathology; 2012 Oct 15; 32(5):505-14. PubMed ID: 22181065
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  • 17. NUP62 localizes to ALS/FTLD pathological assemblies and contributes to TDP-43 insolubility.
    Gleixner AM, Verdone BM, Otte CG, Anderson EN, Ramesh N, Shapiro OR, Gale JR, Mauna JC, Mann JR, Copley KE, Daley EL, Ortega JA, Cicardi ME, Kiskinis E, Kofler J, Pandey UB, Trotti D, Donnelly CJ.
    Nat Commun; 2022 Jun 13; 13(1):3380. PubMed ID: 35697676
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  • 18. Nucleolar stress in C9orf72 and sporadic ALS spinal motor neurons precedes TDP-43 mislocalization.
    Aladesuyi Arogundade O, Nguyen S, Leung R, Wainio D, Rodriguez M, Ravits J.
    Acta Neuropathol Commun; 2021 Feb 15; 9(1):26. PubMed ID: 33588953
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  • 19. Accumulation of dipeptide repeat proteins predates that of TDP-43 in frontotemporal lobar degeneration associated with hexanucleotide repeat expansions in C9ORF72 gene.
    Baborie A, Griffiths TD, Jaros E, Perry R, McKeith IG, Burn DJ, Masuda-Suzukake M, Hasegawa M, Rollinson S, Pickering-Brown S, Robinson AC, Davidson YS, Mann DM.
    Neuropathol Appl Neurobiol; 2015 Aug 15; 41(5):601-12. PubMed ID: 25185840
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  • 20. Poly-A binding protein-1 localization to a subset of TDP-43 inclusions in amyotrophic lateral sclerosis occurs more frequently in patients harboring an expansion in C9orf72.
    McGurk L, Lee VM, Trojanowksi JQ, Van Deerlin VM, Lee EB, Bonini NM.
    J Neuropathol Exp Neurol; 2014 Sep 15; 73(9):837-45. PubMed ID: 25111021
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