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Journal Abstract Search

545 related items for PubMed ID: 26116692

  • 21. Clinical aspects of left ventricular diastolic function assessed by Doppler echocardiography following acute myocardial infarction.
    Poulsen SH.
    Dan Med Bull; 2001 Nov; 48(4):199-210. PubMed ID: 11767125
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  • 22. Dose-Dependent Cardioprotection of Moderate (32°C) Versus Mild (35°C) Therapeutic Hypothermia in Porcine Acute Myocardial Infarction.
    Dash R, Mitsutake Y, Pyun WB, Dawoud F, Lyons J, Tachibana A, Yahagi K, Matsuura Y, Kolodgie FD, Virmani R, McConnell MV, Illindala U, Ikeno F, Yeung A.
    JACC Cardiovasc Interv; 2018 Jan 22; 11(2):195-205. PubMed ID: 29348013
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  • 23. Chitosan hydrogels significantly limit left ventricular infarction and remodeling and preserve myocardial contractility.
    Henning RJ, Khan A, Jimenez E.
    J Surg Res; 2016 Apr 22; 201(2):490-7. PubMed ID: 27020836
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  • 24. Static cardiomyoplasty with synthetic elastic net suppresses ventricular dilatation and dysfunction after myocardial infarction in the rat: a chronic study.
    Kato N, Kawaguchi AT, Kishida A, Yamaoka T.
    Artif Organs; 2013 Jul 22; 37(7):593-9. PubMed ID: 23461677
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  • 25. Biodegradable elastic patch plasty ameliorates left ventricular adverse remodeling after ischemia-reperfusion injury: a preclinical study of a porous polyurethane material in a porcine model.
    Hashizume R, Fujimoto KL, Hong Y, Guan J, Toma C, Tobita K, Wagner WR.
    J Thorac Cardiovasc Surg; 2013 Aug 22; 146(2):391-9.e1. PubMed ID: 23219497
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  • 26. Effect of Intracoronary Metformin on Myocardial Infarct Size in Swine.
    Techiryan G, Weil BR, Palka BA, Canty JM.
    Circ Res; 2018 Sep 28; 123(8):986-995. PubMed ID: 30355033
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  • 27. Intravenous miR-144 reduces left ventricular remodeling after myocardial infarction.
    Li J, Cai SX, He Q, Zhang H, Friedberg D, Wang F, Redington AN.
    Basic Res Cardiol; 2018 Aug 06; 113(5):36. PubMed ID: 30084039
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  • 29. Reduced scar maturation and contractility lead to exaggerated left ventricular dilation after myocardial infarction in mice lacking AMPKα1.
    Noppe G, Dufeys C, Buchlin P, Marquet N, Castanares-Zapatero D, Balteau M, Hermida N, Bouzin C, Esfahani H, Viollet B, Bertrand L, Balligand JL, Vanoverschelde JL, Beauloye C, Horman S.
    J Mol Cell Cardiol; 2014 Sep 06; 74():32-43. PubMed ID: 24805196
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  • 33. Cardiac overexpression of monocyte chemoattractant protein-1 in transgenic mice prevents cardiac dysfunction and remodeling after myocardial infarction.
    Morimoto H, Takahashi M, Izawa A, Ise H, Hongo M, Kolattukudy PE, Ikeda U.
    Circ Res; 2006 Oct 13; 99(8):891-9. PubMed ID: 16990567
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  • 36. Endothelial cells overexpressing IL-8 receptor reduce cardiac remodeling and dysfunction following myocardial infarction.
    Zhao X, Zhang W, Xing D, Li P, Fu J, Gong K, Hage FG, Oparil S, Chen YF.
    Am J Physiol Heart Circ Physiol; 2013 Aug 15; 305(4):H590-8. PubMed ID: 23771691
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