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Journal Abstract Search


206 related items for PubMed ID: 26116906

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  • 10. In vivo formation of gamma-H2AX and 53BP1 DNA repair foci in blood cells after radioiodine therapy of differentiated thyroid cancer.
    Lassmann M, Hänscheid H, Gassen D, Biko J, Meineke V, Reiners C, Scherthan H.
    J Nucl Med; 2010 Aug; 51(8):1318-25. PubMed ID: 20660387
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  • 13. The opportunistic pathogen Pseudomonas aeruginosa activates the DNA double-strand break signaling and repair pathway in infected cells.
    Elsen S, Collin-Faure V, Gidrol X, Lemercier C.
    Cell Mol Life Sci; 2013 Nov; 70(22):4385-97. PubMed ID: 23760206
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  • 18. γ-H2AX/53BP1/pKAP-1 foci and their linear tracks induced by in vitro exposure to radon and its progeny in human peripheral blood lymphocytes.
    Ding D, Zhang Y, Wang J, Wang X, Fan D, He L, Zhang X, Gao Y, Li Q, Chen H.
    Sci Rep; 2016 Dec 06; 6():38295. PubMed ID: 27922110
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  • 19. Relationship between DNA double-strand break rejoining and cell survival after exposure to ionizing radiation in human fibroblast strains with differing ATM/p53 status: implications for evaluation of clinical radiosensitivity.
    Mirzayans R, Severin D, Murray D.
    Int J Radiat Oncol Biol Phys; 2006 Dec 01; 66(5):1498-505. PubMed ID: 17126209
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  • 20. PML nuclear body disruption impairs DNA double-strand break sensing and repair in APL.
    di Masi A, Cilli D, Berardinelli F, Talarico A, Pallavicini I, Pennisi R, Leone S, Antoccia A, Noguera NI, Lo-Coco F, Ascenzi P, Minucci S, Nervi C.
    Cell Death Dis; 2016 Jul 28; 7(7):e2308. PubMed ID: 27468685
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