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Journal Abstract Search
406 related items for PubMed ID: 26160521
1. Cytokine responses induced by diesel exhaust particles are suppressed by PAR-2 silencing and antioxidant treatment, and driven by polar and non-polar soluble constituents. Bach N, Bølling AK, Brinchmann BC, Totlandsdal AI, Skuland T, Holme JA, Låg M, Schwarze PE, Øvrevik J. Toxicol Lett; 2015 Oct 14; 238(2):72-82. PubMed ID: 26160521 [Abstract] [Full Text] [Related]
5. Lipophilic components of diesel exhaust particles induce pro-inflammatory responses in human endothelial cells through AhR dependent pathway(s). Brinchmann BC, Skuland T, Rambøl MH, Szoke K, Brinchmann JE, Gutleb AC, Moschini E, Kubátová A, Kukowski K, Le Ferrec E, Lagadic-Gossmann D, Schwarze PE, Låg M, Refsnes M, Øvrevik J, Holme JA. Part Fibre Toxicol; 2018 May 11; 15(1):21. PubMed ID: 29751765 [Abstract] [Full Text] [Related]
9. Differential proinflammatory responses induced by diesel exhaust particles with contrasting PAH and metal content. Totlandsdal AI, Låg M, Lilleaas E, Cassee F, Schwarze P. Environ Toxicol; 2015 Feb 11; 30(2):188-96. PubMed ID: 23900936 [Abstract] [Full Text] [Related]
12. Diesel exhaust particles induce CYP1A1 and pro-inflammatory responses via differential pathways in human bronchial epithelial cells. Totlandsdal AI, Cassee FR, Schwarze P, Refsnes M, Låg M. Part Fibre Toxicol; 2010 Dec 16; 7():41. PubMed ID: 21162728 [Abstract] [Full Text] [Related]
13. Particulate matter containing environmentally persistent free radicals induces AhR-dependent cytokine and reactive oxygen species production in human bronchial epithelial cells. Harmon AC, Hebert VY, Cormier SA, Subramanian B, Reed JR, Backes WL, Dugas TR. PLoS One; 2018 Dec 16; 13(10):e0205412. PubMed ID: 30308017 [Abstract] [Full Text] [Related]