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PUBMED FOR HANDHELDS

Journal Abstract Search


296 related items for PubMed ID: 26184620

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  • 4. NADPH oxidase-2 inhibition restores contractility and intracellular calcium handling and reduces arrhythmogenicity in dystrophic cardiomyopathy.
    Gonzalez DR, Treuer AV, Lamirault G, Mayo V, Cao Y, Dulce RA, Hare JM.
    Am J Physiol Heart Circ Physiol; 2014 Sep 01; 307(5):H710-21. PubMed ID: 25015966
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  • 5. Local angiotensin II aggravates cardiac remodeling in hypertension.
    Xu J, Carretero OA, Liao TD, Peng H, Shesely EG, Xu J, Liu TS, Yang JJ, Reudelhuber TL, Yang XP.
    Am J Physiol Heart Circ Physiol; 2010 Nov 01; 299(5):H1328-38. PubMed ID: 20833959
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  • 9. Calcium/Calmodulin Protein Kinase II-Dependent Ryanodine Receptor Phosphorylation Mediates Cardiac Contractile Dysfunction Associated With Sepsis.
    Sepúlveda M, Gonano LA, Viotti M, Morell M, Blanco P, López Alarcón M, Peroba Ramos I, Bastos Carvalho A, Medei E, Vila Petroff M.
    Crit Care Med; 2017 Apr 01; 45(4):e399-e408. PubMed ID: 27648519
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  • 10. Endothelial Nox2 overexpression potentiates vascular oxidative stress and hemodynamic response to angiotensin II: studies in endothelial-targeted Nox2 transgenic mice.
    Bendall JK, Rinze R, Adlam D, Tatham AL, de Bono J, Wilson N, Volpi E, Channon KM.
    Circ Res; 2007 Apr 13; 100(7):1016-25. PubMed ID: 17363703
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  • 12. Endothelial cell-specific reactive oxygen species production increases susceptibility to aortic dissection.
    Fan LM, Douglas G, Bendall JK, McNeill E, Crabtree MJ, Hale AB, Mai A, Li JM, McAteer MA, Schneider JE, Choudhury RP, Channon KM.
    Circulation; 2014 Jun 24; 129(25):2661-72. PubMed ID: 24807872
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  • 15. Angiotensin II-mediated phenotypic cardiomyocyte remodeling leads to age-dependent cardiac dysfunction and failure.
    Domenighetti AA, Wang Q, Egger M, Richards SM, Pedrazzini T, Delbridge LM.
    Hypertension; 2005 Aug 24; 46(2):426-32. PubMed ID: 15998712
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  • 16. Cardiac-specific overexpression of phospholamban alters calcium kinetics and resultant cardiomyocyte mechanics in transgenic mice.
    Kadambi VJ, Ponniah S, Harrer JM, Hoit BD, Dorn GW, Walsh RA, Kranias EG.
    J Clin Invest; 1996 Jan 15; 97(2):533-9. PubMed ID: 8567978
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  • 18. Nox2-containing NADPH oxidase and Akt activation play a key role in angiotensin II-induced cardiomyocyte hypertrophy.
    Hingtgen SD, Tian X, Yang J, Dunlay SM, Peek AS, Wu Y, Sharma RV, Engelhardt JF, Davisson RL.
    Physiol Genomics; 2006 Aug 16; 26(3):180-91. PubMed ID: 16670255
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  • 19. Genetic silencing of Nox2 and Nox4 reveals differential roles of these NADPH oxidase homologues in the vasopressor and dipsogenic effects of brain angiotensin II.
    Peterson JR, Burmeister MA, Tian X, Zhou Y, Guruju MR, Stupinski JA, Sharma RV, Davisson RL.
    Hypertension; 2009 Nov 16; 54(5):1106-14. PubMed ID: 19805637
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  • 20. TMEM16A Contributes to Endothelial Dysfunction by Facilitating Nox2 NADPH Oxidase-Derived Reactive Oxygen Species Generation in Hypertension.
    Ma MM, Gao M, Guo KM, Wang M, Li XY, Zeng XL, Sun L, Lv XF, Du YH, Wang GL, Zhou JG, Guan YY.
    Hypertension; 2017 May 16; 69(5):892-901. PubMed ID: 28320851
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