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Journal Abstract Search

254 related items for PubMed ID: 26253170

  • 1.
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  • 2. Hierarchical involvement of Bak, VDAC1 and Bax in cisplatin-induced cell death.
    Tajeddine N, Galluzzi L, Kepp O, Hangen E, Morselli E, Senovilla L, Araujo N, Pinna G, Larochette N, Zamzami N, Modjtahedi N, Harel-Bellan A, Kroemer G.
    Oncogene; 2008 Jul 10; 27(30):4221-32. PubMed ID: 18362892
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  • 5. VDAC2 inhibits BAK activation and mitochondrial apoptosis.
    Cheng EH, Sheiko TV, Fisher JK, Craigen WJ, Korsmeyer SJ.
    Science; 2003 Jul 25; 301(5632):513-7. PubMed ID: 12881569
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  • 7. Involvement of proapoptotic molecules Bax and Bak in tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced mitochondrial disruption and apoptosis: differential regulation of cytochrome c and Smac/DIABLO release.
    Kandasamy K, Srinivasula SM, Alnemri ES, Thompson CB, Korsmeyer SJ, Bryant JL, Srivastava RK.
    Cancer Res; 2003 Apr 01; 63(7):1712-21. PubMed ID: 12670926
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  • 8. Bnip3 mediates mitochondrial dysfunction and cell death through Bax and Bak.
    Kubli DA, Ycaza JE, Gustafsson AB.
    Biochem J; 2007 Aug 01; 405(3):407-15. PubMed ID: 17447897
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  • 9. Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocator.
    Shimizu S, Shinohara Y, Tsujimoto Y.
    Oncogene; 2000 Sep 07; 19(38):4309-18. PubMed ID: 10980606
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  • 10. Bax and Bak genes are essential for maximum apoptotic response by curcumin, a polyphenolic compound and cancer chemopreventive agent derived from turmeric, Curcuma longa.
    Shankar S, Srivastava RK.
    Carcinogenesis; 2007 Jun 07; 28(6):1277-86. PubMed ID: 17277231
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  • 11. Bax targets mitochondria by distinct mechanisms before or during apoptotic cell death: a requirement for VDAC2 or Bak for efficient Bax apoptotic function.
    Ma SB, Nguyen TN, Tan I, Ninnis R, Iyer S, Stroud DA, Menard M, Kluck RM, Ryan MT, Dewson G.
    Cell Death Differ; 2014 Dec 07; 21(12):1925-35. PubMed ID: 25146925
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  • 12. Assembly of the mitochondrial apoptosis-induced channel, MAC.
    Martinez-Caballero S, Dejean LM, Kinnally MS, Oh KJ, Mannella CA, Kinnally KW.
    J Biol Chem; 2009 May 01; 284(18):12235-45. PubMed ID: 19261612
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  • 13. Indirect effects of Bax and Bak initiate the mitochondrial alterations that lead to cytochrome c release during arsenic trioxide-induced apoptosis.
    Nutt LK, Gogvadze V, Uthaisang W, Mirnikjoo B, McConkey DJ, Orrenius S.
    Cancer Biol Ther; 2005 Apr 01; 4(4):459-67. PubMed ID: 15846091
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  • 14. VDAC1-interacting anion transport inhibitors inhibit VDAC1 oligomerization and apoptosis.
    Ben-Hail D, Shoshan-Barmatz V.
    Biochim Biophys Acta; 2016 Jul 01; 1863(7 Pt A):1612-23. PubMed ID: 27064145
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  • 15. Regulation of stress-induced nuclear protein redistribution: a new function of Bax and Bak uncoupled from Bcl-x(L).
    Lindenboim L, Blacher E, Borner C, Stein R.
    Cell Death Differ; 2010 Feb 01; 17(2):346-59. PubMed ID: 19816507
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  • 16. Requirement of voltage-dependent anion channel 2 for pro-apoptotic activity of Bax.
    Yamagata H, Shimizu S, Nishida Y, Watanabe Y, Craigen WJ, Tsujimoto Y.
    Oncogene; 2009 Oct 08; 28(40):3563-72. PubMed ID: 19617898
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  • 17. Increased expression of VDAC1 sensitizes carcinoma cells to apoptosis induced by DNA cross-linking agents.
    Sharaf el dein O, Gallerne C, Brenner C, Lemaire C.
    Biochem Pharmacol; 2012 May 01; 83(9):1172-82. PubMed ID: 22285227
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  • 18. Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells.
    Shimizu S, Matsuoka Y, Shinohara Y, Yoneda Y, Tsujimoto Y.
    J Cell Biol; 2001 Jan 22; 152(2):237-50. PubMed ID: 11266442
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  • 19. Inactivation of prosurvival Bcl-2 proteins activates Bax/Bak through the outer mitochondrial membrane.
    O'Neill KL, Huang K, Zhang J, Chen Y, Luo X.
    Genes Dev; 2016 Apr 15; 30(8):973-88. PubMed ID: 27056669
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  • 20. Bax-dependent regulation of Bak by voltage-dependent anion channel 2.
    Chandra D, Choy G, Daniel PT, Tang DG.
    J Biol Chem; 2005 May 13; 280(19):19051-61. PubMed ID: 15757910
    [Abstract] [Full Text] [Related]

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