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Journal Abstract Search

300 related items for PubMed ID: 26323581

  • 41. [Role of LIGHT signal pathway in Chlamydia muridarum urogenital infection in mice].
    Chen L, Sun Y, Xu S, Fu X, Zhou Z, Lu C, Yang F, Xu G, Wu Y.
    Wei Sheng Wu Xue Bao; 2015 Apr 04; 55(4):492-500. PubMed ID: 26211324
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  • 42. IL-6-mediated signaling pathways limit Chlamydia muridarum infection and exacerbate its pathogenicity in the mouse genital tract.
    Sun X, Tian Q, Wang L, Xue M, Zhong G.
    Microbes Infect; 2017 Nov 04; 19(11):536-545. PubMed ID: 28864426
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  • 44. Interleukin-27 (IL-27) Promotes Chlamydial Infection in the Female Genital Tract.
    Zhao Y, Huo Z, Zhou Z, Cervantes C, Chen J, Xu Z, Zhong G.
    Infect Immun; 2022 Apr 21; 90(4):e0065121. PubMed ID: 35258318
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  • 45. Characterization of Pathogenic CD8+ T cells in Chlamydia-Infected OT1 Mice.
    Zhou Z, Tian Q, Wang L, Sun X, Zhang N, Xue M, Xu D, Zhong G.
    Infect Immun; 2022 Jan 25; 90(1):e0045321. PubMed ID: 34724387
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  • 46. Type I interferon signaling exacerbates Chlamydia muridarum genital infection in a murine model.
    Nagarajan UM, Prantner D, Sikes JD, Andrews CW, Goodwin AM, Nagarajan S, Darville T.
    Infect Immun; 2008 Oct 25; 76(10):4642-8. PubMed ID: 18663004
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  • 47. Chlamydia muridarum plasmid induces mouse oviduct pathology by promoting chlamydial survival and ascending infection and triggering host inflammation.
    Hou S, Yue L, Xu R, Zhu C, Shan S, Wang H, Liu Q.
    Eur J Dermatol; 2018 Oct 01; 28(5):628-636. PubMed ID: 30442635
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  • 49. Chlamydia Spreading from the Genital Tract to the Gastrointestinal Tract - A Two-Hit Hypothesis.
    Zhong G.
    Trends Microbiol; 2018 Jul 01; 26(7):611-623. PubMed ID: 29289422
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  • 50. IL-1α Is Essential for Oviduct Pathology during Genital Chlamydial Infection in Mice.
    Gyorke CE, Kollipara A, Allen J, Zhang Y, Ezzell JA, Darville T, Montgomery SA, Nagarajan UM.
    J Immunol; 2020 Dec 01; 205(11):3037-3049. PubMed ID: 33087404
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  • 54. Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract.
    Mercado MAB, Du W, Malaviarachchi PA, Gann JI, Li LX.
    Infect Immun; 2021 Feb 16; 89(3):. PubMed ID: 33257535
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  • 56. MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resolution.
    Nagarajan UM, Sikes J, Prantner D, Andrews CW, Frazer L, Goodwin A, Snowden JN, Darville T.
    Infect Immun; 2011 Jan 16; 79(1):486-98. PubMed ID: 21078858
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  • 58. Vaccination with the polymorphic membrane protein A reduces Chlamydia muridarum induced genital tract pathology.
    Müller T, Becker E, Stallmann S, Waldhuber A, Römmler-Dreher F, Albrecht S, Mohr F, Hegemann JH, Miethke T.
    Vaccine; 2017 May 15; 35(21):2801-2810. PubMed ID: 28413133
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  • 59. Interleukin-17 contributes to generation of Th1 immunity and neutrophil recruitment during Chlamydia muridarum genital tract infection but is not required for macrophage influx or normal resolution of infection.
    Scurlock AM, Frazer LC, Andrews CW, O'Connell CM, Foote IP, Bailey SL, Chandra-Kuntal K, Kolls JK, Darville T.
    Infect Immun; 2011 Mar 15; 79(3):1349-62. PubMed ID: 21149587
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