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Journal Abstract Search

595 related items for PubMed ID: 26456374

  • 1. Higher and lower active circulating VWF levels: different facets of von Willebrand disease.
    Casonato A, Pontara E, Morpurgo M, Sartorello F, De Groot PG, Cattini MG, Daidone V, De Marco L.
    Br J Haematol; 2015 Dec; 171(5):845-53. PubMed ID: 26456374
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  • 2. Homozygous C2362F von Willebrand factor induces intracellular retention of mutant von Willebrand factor resulting in autosomal recessive severe von Willebrand disease.
    Tjernberg P, Castaman G, Vos HL, Bertina RM, Eikenboom JC.
    Br J Haematol; 2006 May; 133(4):409-18. PubMed ID: 16643449
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  • 3. The p.R1819_C1948delinsS mutation makes von Willebrand factor ADAMTS13-resistant and reduces its collagen-binding capacity.
    Daidone V, Saga G, Barbon G, Pontara E, Cattini MG, Morpurgo M, Zanotti G, Casonato A.
    Br J Haematol; 2015 Aug; 170(4):564-73. PubMed ID: 25904363
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  • 4. C2362F mutation gives rise to an ADAMTS13-resistant von Willebrand factor.
    Casonato A, Pontara E, Battiston M, Morpurgo M, Cattini MG, Casarin E, Saga G, Daidone V, De Marco L.
    Thromb Haemost; 2013 Jun; 109(6):999-1006. PubMed ID: 23446343
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  • 5. Post-DDAVP thrombocytopenia in type 2B von Willebrand disease is not associated with platelet consumption: failure to demonstrate glycocalicin increase or platelet activation.
    Casonato A, Steffan A, Pontara E, Zucchetto A, Rossi C, De Marco L, Girolami A.
    Thromb Haemost; 1999 Feb; 81(2):224-8. PubMed ID: 10063996
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  • 11. A new L1446P mutation is responsible for impaired von Willebrand factor synthesis, structure, and function.
    Casonato A, Cattini MG, Soldera C, Marcato S, Sartorello F, Pontara E, Pagnan A.
    J Lab Clin Med; 2004 Nov; 144(5):254-9. PubMed ID: 15570243
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  • 14. Type 2M von Willebrand disease variant characterized by abnormal von willebrand factor multimerization.
    Casonato A, Pontara E, Sartorello F, Bertomoro A, Durante C, Girolami A.
    J Lab Clin Med; 2001 Jan; 137(1):70-6. PubMed ID: 11150026
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  • 15. Laboratory diagnosis of von Willebrand disease type 1/2E (2A subtype IIE), type 1 Vicenza and mild type 1 caused by mutations in the D3, D4, B1-B3 and C1-C2 domains of the von Willebrand factor gene. Role of von Willebrand factor multimers and the von Willebrand factor propeptide/antigen ratio.
    Gadisseur A, Berneman Z, Schroyens W, Michiels JJ.
    Acta Haematol; 2009 Jan; 121(2-3):128-38. PubMed ID: 19506359
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  • 18. Enhanced Local Disorder in a Clinically Elusive von Willebrand Factor Provokes High-Affinity Platelet Clumping.
    Tischer A, Machha VR, Frontroth JP, Brehm MA, Obser T, Schneppenheim R, Mayne L, Walter Englander S, Auton M.
    J Mol Biol; 2017 Jul 07; 429(14):2161-2177. PubMed ID: 28533135
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