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Journal Abstract Search

676 related items for PubMed ID: 26502195

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  • 2. Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies.
    Kariya S, Re DB, Jacquier A, Nelson K, Przedborski S, Monani UR.
    Hum Mol Genet; 2012 Aug 01; 21(15):3421-34. PubMed ID: 22581780
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  • 5. TDP-43 expression in mouse models of amyotrophic lateral sclerosis and spinal muscular atrophy.
    Turner BJ, Bäumer D, Parkinson NJ, Scaber J, Ansorge O, Talbot K.
    BMC Neurosci; 2008 Oct 28; 9():104. PubMed ID: 18957104
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  • 6. Transcriptional profiling of differentially vulnerable motor neurons at pre-symptomatic stage in the Smn (2b/-) mouse model of spinal muscular atrophy.
    Murray LM, Beauvais A, Gibeault S, Courtney NL, Kothary R.
    Acta Neuropathol Commun; 2015 Sep 15; 3():55. PubMed ID: 26374403
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  • 7. Defective neuromuscular junction organization and postnatal myogenesis in mice with severe spinal muscular atrophy.
    Dachs E, Hereu M, Piedrafita L, Casanovas A, Calderó J, Esquerda JE.
    J Neuropathol Exp Neurol; 2011 Jun 15; 70(6):444-61. PubMed ID: 21572339
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  • 8. Reduced survival of motor neuron (SMN) protein in motor neuronal progenitors functions cell autonomously to cause spinal muscular atrophy in model mice expressing the human centromeric (SMN2) gene.
    Park GH, Maeno-Hikichi Y, Awano T, Landmesser LT, Monani UR.
    J Neurosci; 2010 Sep 08; 30(36):12005-19. PubMed ID: 20826664
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  • 9. Macrophage-mediated inflammation and glial response in the skeletal muscle of a rat model of familial amyotrophic lateral sclerosis (ALS).
    Van Dyke JM, Smit-Oistad IM, Macrander C, Krakora D, Meyer MG, Suzuki M.
    Exp Neurol; 2016 Mar 08; 277():275-282. PubMed ID: 26775178
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  • 11. Synaptotagmin-2, and -1, linked to neurotransmission impairment and vulnerability in Spinal Muscular Atrophy.
    Tejero R, Lopez-Manzaneda M, Arumugam S, Tabares L.
    Hum Mol Genet; 2016 Nov 01; 25(21):4703-4716. PubMed ID: 28173138
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  • 13. Selective Neuromuscular Denervation in Taiwanese Severe SMA Mouse Can Be Reversed by Morpholino Antisense Oligonucleotides.
    Lin TL, Chen TH, Hsu YY, Cheng YH, Juang BT, Jong YJ.
    PLoS One; 2016 Nov 01; 11(4):e0154723. PubMed ID: 27124114
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  • 14. A critical smn threshold in mice dictates onset of an intermediate spinal muscular atrophy phenotype associated with a distinct neuromuscular junction pathology.
    Bowerman M, Murray LM, Beauvais A, Pinheiro B, Kothary R.
    Neuromuscul Disord; 2012 Mar 01; 22(3):263-76. PubMed ID: 22071333
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  • 16. Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Clark JA, Southam KA, Blizzard CA, King AE, Dickson TC.
    J Chem Neuroanat; 2016 Oct 01; 76(Pt A):35-47. PubMed ID: 27038603
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  • 17. Survival motor neuron deficiency enhances progression in an amyotrophic lateral sclerosis mouse model.
    Turner BJ, Parkinson NJ, Davies KE, Talbot K.
    Neurobiol Dis; 2009 Jun 01; 34(3):511-7. PubMed ID: 19332122
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  • 18. Pathogenic commonalities between spinal muscular atrophy and amyotrophic lateral sclerosis: Converging roads to therapeutic development.
    Bowerman M, Murray LM, Scamps F, Schneider BL, Kothary R, Raoul C.
    Eur J Med Genet; 2018 Nov 01; 61(11):685-698. PubMed ID: 29313812
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  • 20. SAHA ameliorates the SMA phenotype in two mouse models for spinal muscular atrophy.
    Riessland M, Ackermann B, Förster A, Jakubik M, Hauke J, Garbes L, Fritzsche I, Mende Y, Blumcke I, Hahnen E, Wirth B.
    Hum Mol Genet; 2010 Apr 15; 19(8):1492-506. PubMed ID: 20097677
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