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Journal Abstract Search


240 related items for PubMed ID: 26503399

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  • 2. Possible mechanisms underlying slow component of V̇O2 on-kinetics in skeletal muscle.
    Korzeniewski B, Zoladz JA.
    J Appl Physiol (1985); 2015 May 15; 118(10):1240-9. PubMed ID: 25767031
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  • 3. Regulation of oxidative phosphorylation is different in electrically- and cortically-stimulated skeletal muscle.
    Korzeniewski B.
    PLoS One; 2018 May 15; 13(4):e0195620. PubMed ID: 29698403
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  • 4. Mechanisms of the effect of oxidative phosphorylation deficiencies on the skeletal muscle bioenergetic system in patients with mitochondrial myopathies.
    Korzeniewski B.
    J Appl Physiol (1985); 2021 Aug 01; 131(2):768-777. PubMed ID: 34197225
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  • 7. Regulation of oxidative phosphorylation during work transitions results from its kinetic properties.
    Korzeniewski B.
    J Appl Physiol (1985); 2014 Jan 01; 116(1):83-94. PubMed ID: 24157529
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  • 9. Influence of rapid changes in cytosolic pH on oxidative phosphorylation in skeletal muscle: theoretical studies.
    Korzeniewski B, Zoladz JA.
    Biochem J; 2002 Jul 01; 365(Pt 1):249-58. PubMed ID: 12132435
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  • 17. Slow VO2 off-kinetics in skeletal muscle is associated with fast PCr off-kinetics--and inversely.
    Korzeniewski B, Zoladz JA.
    J Appl Physiol (1985); 2013 Sep 01; 115(5):605-12. PubMed ID: 23788573
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  • 19. Effects of OXPHOS complex deficiencies and ESA dysfunction in working intact skeletal muscle: implications for mitochondrial myopathies.
    Korzeniewski B.
    Biochim Biophys Acta; 2015 Oct 01; 1847(10):1310-9. PubMed ID: 26188374
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