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Journal Abstract Search

163 related items for PubMed ID: 26573464

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  • 2. Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibiting reactive oxygen species-activated extracellular signal-regulated kinase 1/2 in H9c2 cardiac myocytes.
    Liu MH, Lin XL, Zhang Y, He J, Tan TP, Wu SJ, Liu J, Tian W, Chen L, Yu S, Li J, Yuan C.
    Mol Med Rep; 2015 Nov; 12(5):6841-8. PubMed ID: 26299281
    [Abstract] [Full Text] [Related]

  • 3. Hydrogen sulfide attenuates doxorubicin‑induced cardiotoxicity by inhibiting calreticulin expression in H9c2 cells.
    Liu MH, Zhang Y, Lin XL, He J, Tan TP, Wu SJ, Yu S, Chen L, Chen YD, Fu HY, Yuan C, Li J.
    Mol Med Rep; 2015 Oct; 12(4):5197-202. PubMed ID: 26134131
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  • 4. Hydrogen sulfide protects H9c2 cardiac cells against doxorubicin-induced cytotoxicity through the PI3K/Akt/FoxO3a pathway.
    Liu MH, Zhang Y, He J, Tan TP, Wu SJ, Guo DM, He H, Peng J, Tang ZH, Jiang ZS.
    Int J Mol Med; 2016 Jun; 37(6):1661-8. PubMed ID: 27081862
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  • 5. Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibition of the p38 MAPK pathway in H9c2 cells.
    Guo R, Lin J, Xu W, Shen N, Mo L, Zhang C, Feng J.
    Int J Mol Med; 2013 Mar; 31(3):644-50. PubMed ID: 23338126
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  • 6. Hydrogen sulfide protects H9c2 cells against doxorubicin-induced cardiotoxicity through inhibition of endoplasmic reticulum stress.
    Wang XY, Yang CT, Zheng DD, Mo LQ, Lan AP, Yang ZL, Hu F, Chen PX, Liao XX, Feng JQ.
    Mol Cell Biochem; 2012 Apr; 363(1-2):419-26. PubMed ID: 22203419
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  • 7. Exogenous hydrogen sulfide alleviates high glucose-induced cardiotoxicity via inhibition of leptin signaling in H9c2 cells.
    Zhuang XD, Hu X, Long M, Dong XB, Liu DH, Liao XX.
    Mol Cell Biochem; 2014 Jun; 391(1-2):147-55. PubMed ID: 24687304
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  • 8. The cystathionine γ-lyase/hydrogen sulfide pathway mediates the trimetazidine-induced protection of H9c2 cells against hypoxia/reoxygenation-induced apoptosis and oxidative stress.
    Zheng W, Liu C.
    Anatol J Cardiol; 2019 Sep; 22(3):102-111. PubMed ID: 31475956
    [Abstract] [Full Text] [Related]

  • 9. Diallyl trisulfide protects against high glucose-induced cardiac apoptosis by stimulating the production of cystathionine gamma-lyase-derived hydrogen sulfide.
    Tsai CY, Wen SY, Shibu MA, Yang YC, Peng H, Wang B, Wei YM, Chang HY, Lee CY, Huang CY, Kuo WW.
    Int J Cardiol; 2015 Sep 15; 195():300-10. PubMed ID: 26056963
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  • 11. Endogenous Hydrogen Sulfide Persulfidates Caspase-3 at Cysteine 163 to Inhibit Doxorubicin-Induced Cardiomyocyte Apoptosis.
    Ye X, Li Y, Lv B, Qiu B, Zhang S, Peng H, Kong W, Tang C, Huang Y, Du J, Jin H.
    Oxid Med Cell Longev; 2022 Sep 15; 2022():6153772. PubMed ID: 35571249
    [Abstract] [Full Text] [Related]

  • 12. Exogenous hydrogen sulfide protects H9c2 cardiac cells against high glucose-induced injury by inhibiting the activities of the p38 MAPK and ERK1/2 pathways.
    Xu W, Wu W, Chen J, Guo R, Lin J, Liao X, Feng J.
    Int J Mol Med; 2013 Oct 15; 32(4):917-25. PubMed ID: 23912965
    [Abstract] [Full Text] [Related]

  • 13. Resveratrol prevents doxorubicin-induced cardiotoxicity in H9c2 cells through the inhibition of endoplasmic reticulum stress and the activation of the Sirt1 pathway.
    Lou Y, Wang Z, Xu Y, Zhou P, Cao J, Li Y, Chen Y, Sun J, Fu L.
    Int J Mol Med; 2015 Sep 15; 36(3):873-80. PubMed ID: 26202177
    [Abstract] [Full Text] [Related]

  • 14. Exogenous H2S protects H9c2 cardiac cells against high glucose-induced injury and inflammation by inhibiting the activation of the NF-κB and IL-1β pathways.
    Xu W, Chen J, Lin J, Liu D, Mo L, Pan W, Feng J, Wu W, Zheng D.
    Int J Mol Med; 2015 Jan 15; 35(1):177-86. PubMed ID: 25412187
    [Abstract] [Full Text] [Related]

  • 15. Enhanced-autophagy by exenatide mitigates doxorubicin-induced cardiotoxicity.
    Lee KH, Cho H, Lee S, Woo JS, Cho BH, Kang JH, Jeong YM, Cheng XW, Kim W.
    Int J Cardiol; 2017 Apr 01; 232():40-47. PubMed ID: 28159361
    [Abstract] [Full Text] [Related]

  • 16. Hydrogen Sulfide Preserves Endothelial Nitric Oxide Synthase Function by Inhibiting Proline-Rich Kinase 2: Implications for Cardiomyocyte Survival and Cardioprotection.
    Bibli SI, Szabo C, Chatzianastasiou A, Luck B, Zukunft S, Fleming I, Papapetropoulos A.
    Mol Pharmacol; 2017 Dec 01; 92(6):718-730. PubMed ID: 29030392
    [Abstract] [Full Text] [Related]

  • 17. Novel insights into the role of HSP90 in cytoprotection of H2S against chemical hypoxia-induced injury in H9c2 cardiac myocytes.
    Yang Z, Yang C, Xiao L, Liao X, Lan A, Wang X, Guo R, Chen P, Hu C, Feng J.
    Int J Mol Med; 2011 Sep 01; 28(3):397-403. PubMed ID: 21519787
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