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Journal Abstract Search

113 related items for PubMed ID: 26631092

  • 1. GABAergic Agonists Modulate the Glutamate Release from Frontal Cortex Synaptosomes of Rats with Experimental Autoimmune Encephalomyelitis.
    Fernández Hurst N, Chanaday NL, Roth GA.
    Inflamm Allergy Drug Targets; 2015; 14(2):105-10. PubMed ID: 26631092
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  • 2. Participation of the GABAergic system on the glutamate release of frontal cortex synaptosomes from Wistar rats with experimental autoimmune encephalomyelitis.
    Cid MP, Vilcaes AA, Rupil LL, Salvatierra NA, Roth GA.
    Neuroscience; 2011 Aug 25; 189():337-44. PubMed ID: 21635939
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  • 4. Inhibitory role of diazepam on autoimmune inflammation in rats with experimental autoimmune encephalomyelitis.
    Bibolini MJ, Chanaday NL, Báez NS, Degano AL, Monferran CG, Roth GA.
    Neuroscience; 2011 Dec 29; 199():421-8. PubMed ID: 21964471
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  • 6. Presynaptic GABA(B) receptor modulation of glutamate exocytosis from rat cerebrocortical nerve terminals: receptor decoupling by protein kinase C.
    Perkinton MS, Sihra TS.
    J Neurochem; 1998 Apr 29; 70(4):1513-22. PubMed ID: 9523568
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  • 7. Environmental training is beneficial to clinical symptoms and cortical presynaptic defects in mice suffering from experimental autoimmune encephalomyelitis.
    Bonfiglio T, Olivero G, Vergassola M, Di Cesare Mannelli L, Pacini A, Iannuzzi F, Summa M, Bertorelli R, Feligioni M, Ghelardini C, Pittaluga A.
    Neuropharmacology; 2019 Feb 29; 145(Pt A):75-86. PubMed ID: 29402503
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  • 9. Mechanisms underlying the riluzole inhibition of glutamate release from rat cerebral cortex nerve terminals (synaptosomes).
    Wang SJ, Wang KY, Wang WC.
    Neuroscience; 2004 Feb 29; 125(1):191-201. PubMed ID: 15051158
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  • 13. Acute stress is not acute: sustained enhancement of glutamate release after acute stress involves readily releasable pool size and synapsin I activation.
    Musazzi L, Tornese P, Sala N, Popoli M.
    Mol Psychiatry; 2017 Sep 29; 22(9):1226-1227. PubMed ID: 27698433
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  • 14. Complement tunes glutamate release and supports synaptic impairments in an animal model of multiple sclerosis.
    Olivero G, Taddeucci A, Vallarino G, Trebesova H, Roggeri A, Gagliani MC, Cortese K, Grilli M, Pittaluga A.
    Br J Pharmacol; 2024 Jun 29; 181(12):1812-1828. PubMed ID: 38369641
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  • 16. Opposing changes in phosphorylation of specific sites in synapsin I during Ca2+-dependent glutamate release in isolated nerve terminals.
    Jovanovic JN, Sihra TS, Nairn AC, Hemmings HC, Greengard P, Czernik AJ.
    J Neurosci; 2001 Oct 15; 21(20):7944-53. PubMed ID: 11588168
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