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239 related items for PubMed ID: 26668058

  • 1. Proteomic analysis of physiological versus pathological cardiac remodeling in animal models expressing mutations in myosin essential light chains.
    Gomes AV, Kazmierczak K, Cheah JX, Gilda JE, Yuan CC, Zhou Z, Szczesna-Cordary D.
    J Muscle Res Cell Motil; 2015 Dec; 36(6):447-61. PubMed ID: 26668058
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  • 4. Discrete effects of A57G-myosin essential light chain mutation associated with familial hypertrophic cardiomyopathy.
    Kazmierczak K, Paulino EC, Huang W, Muthu P, Liang J, Yuan CC, Rojas AI, Hare JM, Szczesna-Cordary D.
    Am J Physiol Heart Circ Physiol; 2013 Aug 15; 305(4):H575-89. PubMed ID: 23748425
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  • 5. Remodeling of the heart in hypertrophy in animal models with myosin essential light chain mutations.
    Kazmierczak K, Yuan CC, Liang J, Huang W, Rojas AI, Szczesna-Cordary D.
    Front Physiol; 2014 Aug 15; 5():353. PubMed ID: 25295008
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  • 7. The role of the N-terminus of the myosin essential light chain in cardiac muscle contraction.
    Kazmierczak K, Xu Y, Jones M, Guzman G, Hernandez OM, Kerrick WG, Szczesna-Cordary D.
    J Mol Biol; 2009 Apr 03; 387(3):706-25. PubMed ID: 19361417
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  • 8. The E22K mutation of myosin RLC that causes familial hypertrophic cardiomyopathy increases calcium sensitivity of force and ATPase in transgenic mice.
    Szczesna-Cordary D, Guzman G, Zhao J, Hernandez O, Wei J, Diaz-Perez Z.
    J Cell Sci; 2005 Aug 15; 118(Pt 16):3675-83. PubMed ID: 16076902
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  • 9. Hypercontractile mutant of ventricular myosin essential light chain leads to disruption of sarcomeric structure and function and results in restrictive cardiomyopathy in mice.
    Yuan CC, Kazmierczak K, Liang J, Kanashiro-Takeuchi R, Irving TC, Gomes AV, Wang Y, Burghardt TP, Szczesna-Cordary D.
    Cardiovasc Res; 2017 Aug 01; 113(10):1124-1136. PubMed ID: 28371863
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  • 10. Myosin regulatory light chain E22K mutation results in decreased cardiac intracellular calcium and force transients.
    Szczesna-Cordary D, Jones M, Moore JR, Watt J, Kerrick WG, Xu Y, Wang Y, Wagg C, Lopaschuk GD.
    FASEB J; 2007 Dec 01; 21(14):3974-85. PubMed ID: 17606808
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  • 11. New Noonan syndrome model mice with RIT1 mutation exhibit cardiac hypertrophy and susceptibility to β-adrenergic stimulation-induced cardiac fibrosis.
    Takahara S, Inoue SI, Miyagawa-Tomita S, Matsuura K, Nakashima Y, Niihori T, Matsubara Y, Saiki Y, Aoki Y.
    EBioMedicine; 2019 Apr 01; 42():43-53. PubMed ID: 30898653
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  • 12. Prolonged Ca2+ and force transients in myosin RLC transgenic mouse fibers expressing malignant and benign FHC mutations.
    Wang Y, Xu Y, Kerrick WG, Wang Y, Guzman G, Diaz-Perez Z, Szczesna-Cordary D.
    J Mol Biol; 2006 Aug 11; 361(2):286-99. PubMed ID: 16837010
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  • 13. Distinct lipidomic profiles in models of physiological and pathological cardiac remodeling, and potential therapeutic strategies.
    Tham YK, Huynh K, Mellett NA, Henstridge DC, Kiriazis H, Ooi JYY, Matsumoto A, Patterson NL, Sadoshima J, Meikle PJ, McMullen JR.
    Biochim Biophys Acta Mol Cell Biol Lipids; 2018 Mar 11; 1863(3):219-234. PubMed ID: 29217479
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  • 14. Deletion of 1-43 amino acids in cardiac myosin essential light chain blunts length dependency of Ca(2+) sensitivity and cross-bridge detachment kinetics.
    Michael JJ, Gollapudi SK, Ford SJ, Kazmierczak K, Szczesna-Cordary D, Chandra M.
    Am J Physiol Heart Circ Physiol; 2013 Jan 15; 304(2):H253-9. PubMed ID: 23144314
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  • 15. Targeted disruption of Hspa4 gene leads to cardiac hypertrophy and fibrosis.
    Mohamed BA, Barakat AZ, Zimmermann WH, Bittner RE, Mühlfeld C, Hünlich M, Engel W, Maier LS, Adham IM.
    J Mol Cell Cardiol; 2012 Oct 15; 53(4):459-68. PubMed ID: 22884543
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  • 16. Characterizations of myosin essential light chain's N-terminal truncation mutant Δ43 in transgenic mouse papillary muscles by using tension transients in response to sinusoidal length alterations.
    Wang L, Muthu P, Szczesna-Cordary D, Kawai M.
    J Muscle Res Cell Motil; 2013 May 15; 34(2):93-105. PubMed ID: 23397074
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  • 17. Myosin regulatory light chain phosphorylation attenuates cardiac hypertrophy.
    Huang J, Shelton JM, Richardson JA, Kamm KE, Stull JT.
    J Biol Chem; 2008 Jul 11; 283(28):19748-56. PubMed ID: 18474588
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  • 18. Molecular mechanisms of cardiomyopathy phenotypes associated with myosin light chain mutations.
    Huang W, Szczesna-Cordary D.
    J Muscle Res Cell Motil; 2015 Dec 11; 36(6):433-45. PubMed ID: 26385864
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  • 19. Proteomic analysis of short-term preload-induced eccentric cardiac hypertrophy.
    Mohamed BA, Asif AR, Schnelle M, Qasim M, Khadjeh S, Lbik D, Schott P, Hasenfuss G, Toischer K.
    J Transl Med; 2016 May 27; 14(1):149. PubMed ID: 27234427
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  • 20. Constitutive phosphorylation of cardiac myosin regulatory light chain prevents development of hypertrophic cardiomyopathy in mice.
    Yuan CC, Muthu P, Kazmierczak K, Liang J, Huang W, Irving TC, Kanashiro-Takeuchi RM, Hare JM, Szczesna-Cordary D.
    Proc Natl Acad Sci U S A; 2015 Jul 28; 112(30):E4138-46. PubMed ID: 26124132
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