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Journal Abstract Search

561 related items for PubMed ID: 26718724

  • 21. Determination of the critical residues responsible for cardiac myosin binding protein C's interactions.
    Bhuiyan MS, Gulick J, Osinska H, Gupta M, Robbins J.
    J Mol Cell Cardiol; 2012 Dec; 53(6):838-47. PubMed ID: 22982234
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  • 22. N-Terminal Domains of Cardiac Myosin Binding Protein C Cooperatively Activate the Thin Filament.
    Risi C, Belknap B, Forgacs-Lonart E, Harris SP, Schröder GF, White HD, Galkin VE.
    Structure; 2018 Dec 04; 26(12):1604-1611.e4. PubMed ID: 30270174
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  • 23. ADP-stimulated contraction: A predictor of thin-filament activation in cardiac disease.
    Sequeira V, Najafi A, Wijnker PJ, Dos Remedios CG, Michels M, Kuster DW, van der Velden J.
    Proc Natl Acad Sci U S A; 2015 Dec 15; 112(50):E7003-12. PubMed ID: 26621701
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  • 26. Investigation of a truncated cardiac troponin T that causes familial hypertrophic cardiomyopathy: Ca(2+) regulatory properties of reconstituted thin filaments depend on the ratio of mutant to wild-type protein.
    Redwood C, Lohmann K, Bing W, Esposito GM, Elliott K, Abdulrazzak H, Knott A, Purcell I, Marston S, Watkins H.
    Circ Res; 2000 Jun 09; 86(11):1146-52. PubMed ID: 10850966
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  • 28. [Abnormal tropomyosin function in ATPase cycle in hypertrophic and dilated cardiomyopathies].
    Borovikov IuS, Karpicheva OE, Rysev NA, Redwood CS.
    Ross Fiziol Zh Im I M Sechenova; 2013 Jan 09; 99(1):73-80. PubMed ID: 23659058
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  • 31. The HCM-linked W792R mutation in cardiac myosin-binding protein C reduces C6 FnIII domain stability.
    Smelter DF, de Lange WJ, Cai W, Ge Y, Ralphe JC.
    Am J Physiol Heart Circ Physiol; 2018 Jun 01; 314(6):H1179-H1191. PubMed ID: 29451820
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  • 32. Infantile restrictive cardiomyopathy: cTnI-R170G/W impair the interplay of sarcomeric proteins and the integrity of thin filaments.
    Cimiotti D, Fujita-Becker S, Möhner D, Smolina N, Budde H, Wies A, Morgenstern L, Gudkova A, Sejersen T, Sjöberg G, Mügge A, Nowaczyk MM, Reusch P, Pfitzer G, Stehle R, Schröder RR, Mannherz HG, Kostareva A, Jaquet K.
    PLoS One; 2020 Jun 01; 15(3):e0229227. PubMed ID: 32182250
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  • 33. Multiple forms of cardiac myosin-binding protein C exist and can regulate thick filament stability.
    Kulikovskaya I, McClellan GB, Levine R, Winegrad S.
    J Gen Physiol; 2007 May 01; 129(5):419-28. PubMed ID: 17470661
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  • 37. The flexibility of two tropomyosin mutants, D175N and E180G, that cause hypertrophic cardiomyopathy.
    Li XE, Suphamungmee W, Janco M, Geeves MA, Marston SB, Fischer S, Lehman W.
    Biochem Biophys Res Commun; 2012 Aug 03; 424(3):493-6. PubMed ID: 22789852
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  • 38. Cryo-EM structures of the actin:tropomyosin filament reveal the mechanism for the transition from C- to M-state.
    Sousa DR, Stagg SM, Stroupe ME.
    J Mol Biol; 2013 Nov 15; 425(22):4544-55. PubMed ID: 24021812
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  • 39. Structural basis for Ca2+-regulated muscle relaxation at interaction sites of troponin with actin and tropomyosin.
    Murakami K, Yumoto F, Ohki SY, Yasunaga T, Tanokura M, Wakabayashi T.
    J Mol Biol; 2005 Sep 09; 352(1):178-201. PubMed ID: 16061251
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