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Journal Abstract Search

406 related items for PubMed ID: 26719414

  • 1.
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  • 2. Human superoxide dismutase 1 overexpression in motor neurons of Caenorhabditis elegans causes axon guidance defect and neurodegeneration.
    Li J, Li T, Zhang X, Tang Y, Yang J, Le W.
    Neurobiol Aging; 2014 Apr; 35(4):837-46. PubMed ID: 24126158
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  • 3. MIF inhibits the formation and toxicity of misfolded SOD1 amyloid aggregates: implications for familial ALS.
    Shvil N, Banerjee V, Zoltsman G, Shani T, Kahn J, Abu-Hamad S, Papo N, Engel S, Bernhagen J, Israelson A.
    Cell Death Dis; 2018 Jan 25; 9(2):107. PubMed ID: 29371591
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  • 4. Large SOD1 aggregates, unlike trimeric SOD1, do not impact cell viability in a model of amyotrophic lateral sclerosis.
    Zhu C, Beck MV, Griffith JD, Deshmukh M, Dokholyan NV.
    Proc Natl Acad Sci U S A; 2018 May 01; 115(18):4661-4665. PubMed ID: 29666246
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  • 5. Parkin Protects Against Misfolded SOD1 Toxicity by Promoting Its Aggresome Formation and Autophagic Clearance.
    Yung C, Sha D, Li L, Chin LS.
    Mol Neurobiol; 2016 Nov 01; 53(9):6270-6287. PubMed ID: 26563499
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  • 6. Mutant SOD1 instability: implications for toxicity in amyotrophic lateral sclerosis.
    Tiwari A, Hayward LJ.
    Neurodegener Dis; 2005 Nov 01; 2(3-4):115-27. PubMed ID: 16909016
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  • 7. Many roads lead to Rome? Multiple modes of Cu,Zn superoxide dismutase destabilization, misfolding and aggregation in amyotrophic lateral sclerosis.
    Broom HR, Rumfeldt JA, Meiering EM.
    Essays Biochem; 2014 Nov 01; 56():149-65. PubMed ID: 25131593
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  • 8. The metal cofactor zinc and interacting membranes modulate SOD1 conformation-aggregation landscape in an in vitro ALS model.
    Sannigrahi A, Chowdhury S, Das B, Banerjee A, Halder A, Kumar A, Saleem M, Naganathan AN, Karmakar S, Chattopadhyay K.
    Elife; 2021 Apr 07; 10():. PubMed ID: 33825682
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  • 9.
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  • 10. Does wild-type Cu/Zn-superoxide dismutase have pathogenic roles in amyotrophic lateral sclerosis?
    Furukawa Y, Tokuda E.
    Transl Neurodegener; 2020 Aug 19; 9(1):33. PubMed ID: 32811540
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  • 11. Prion-like propagation of mutant SOD1 misfolding and motor neuron disease spread along neuroanatomical pathways.
    Ayers JI, Fromholt SE, O'Neal VM, Diamond JH, Borchelt DR.
    Acta Neuropathol; 2016 Jan 19; 131(1):103-14. PubMed ID: 26650262
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  • 13. Oxidized/misfolded superoxide dismutase-1: the cause of all amyotrophic lateral sclerosis?
    Kabashi E, Valdmanis PN, Dion P, Rouleau GA.
    Ann Neurol; 2007 Dec 19; 62(6):553-9. PubMed ID: 18074357
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  • 14. A novel monoclonal antibody reveals a conformational alteration shared by amyotrophic lateral sclerosis-linked SOD1 mutants.
    Fujisawa T, Homma K, Yamaguchi N, Kadowaki H, Tsuburaya N, Naguro I, Matsuzawa A, Takeda K, Takahashi Y, Goto J, Tsuji S, Nishitoh H, Ichijo H.
    Ann Neurol; 2012 Nov 19; 72(5):739-49. PubMed ID: 23280792
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  • 15. Curcumin binds to the pre-fibrillar aggregates of Cu/Zn superoxide dismutase (SOD1) and alters its amyloidogenic pathway resulting in reduced cytotoxicity.
    Bhatia NK, Srivastava A, Katyal N, Jain N, Khan MA, Kundu B, Deep S.
    Biochim Biophys Acta; 2015 May 19; 1854(5):426-36. PubMed ID: 25666897
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  • 17. [SOD1 gene mutations in patients with amyotrophic lateral sclerosis: potential for the method of molecular].
    Lysogorskaia EV, Rossokhin AV, Abramycheva NIu, Zakharova MN, Illarioshkin SN.
    Mol Biol (Mosk); 2013 May 19; 47(5):861-7. PubMed ID: 25509359
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