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Journal Abstract Search

295 related items for PubMed ID: 26850598

  • 1. First Report of a Dominantly Inherited β-Thalassemia Caused by a Novel Elongated β-Globin Chain.
    Farashi S, Rad F, Shahmohammadi B, Imanian H, Azarkeivan A, Najmabadi H.
    Hemoglobin; 2016; 40(2):102-7. PubMed ID: 26850598
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  • 3. Dominantly Inherited beta-Thalassemia.
    Efremov GD.
    Hemoglobin; 2007; 31(2):193-207. PubMed ID: 17486503
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  • 4. Hb Filottrano [codon 120 (-A)]: a novel frameshift mutation in exon 3 of the β-globin gene causing dominantly inherited β-thalassemia intermedia.
    Amato A, Cappabianca MP, Perri M, Zaghis I, Mastropietro F, Ponzini D, Di Biagio P, Piscitelli R.
    Hemoglobin; 2012; 36(5):480-4. PubMed ID: 22992010
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  • 5. Hb Knossos (HBB: c.82G > T), β-globin CD 5 (-CT) (HBB: c.17_18delCT) and δ-globin CD 59 (-a) (HBD: c.179delA) mutations in a Syrian patient with β-thalassemia intermedia.
    Moassas F, Nweder MS, Murad H.
    BMC Pediatr; 2019 Feb 18; 19(1):61. PubMed ID: 30777047
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  • 9. Dominantly Inherited β-Thalassemia Caused by a Single Nucleotide Deletion in Exon 3 of the β-Globin Gene: Hb Xiangyang (HBB: c.393delT).
    Lin XM, Jiang F, Li J, Li DZ.
    Hemoglobin; 2022 Jul 18; 46(4):253-255. PubMed ID: 35686459
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  • 10. Krüppel-Like Factor 1 Gene Mutations in Thalassemia Patients from North Iran: Report of a New Mutation Associated with β-Thalassemia Intermedia.
    Tamaddoni A, Khabaz Astaneh S, Tabaripour R, Akhavan-Niaki H.
    Hemoglobin; 2019 Jan 18; 43(1):12-17. PubMed ID: 30747024
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  • 12. Elevated Hb A₂ Levels in a Patient with a Compound Heterozygosity for the (β⁺) -31 (A > G) and (β⁰) Codon 17 (A > T) Mutations Together with a Single α-Globin Gene.
    Panyasai S, Jaiping K, Pornprasert S.
    Hemoglobin; 2015 Jan 18; 39(4):292-5. PubMed ID: 26029792
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  • 14. Severe Thalassemia Caused by Hb Zunyi [β147(HC3)Stop→Gln; HBB: c.442T>C)] on the β-Globin Gene.
    Su Q, Chen S, Wu L, Tian R, Yang X, Huang X, Chen Y, Peng Z, Chen J.
    Hemoglobin; 2019 Jan 18; 43(1):7-11. PubMed ID: 31084366
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  • 16. A Novel Frameshift Mutation at Codons 138/139 (HBB: c.417_418insT) on the β-Globin Gene Leads to β-Thalassemia.
    Jiang F, Huang LY, Chen GL, Zhou JY, Xie XM, Li DZ.
    Hemoglobin; 2017 Jan 18; 41(1):59-60. PubMed ID: 28460555
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  • 17. IVS-II-648/649 (-T) (HBB: c.316-202del) Triggers a Novel β-Thalassemia Phenotype.
    Azimi A, Alibakhshi R, Hayati H, Tahmasebi S, Alimoradi S.
    Hemoglobin; 2017 Jan 18; 41(1):44-46. PubMed ID: 28475449
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  • 18. Co-heredity of silent CAP + 1570 T>C (HBB:c*96T>C) defect and severe β-thal mutation: a cause of mild β-thalassemia intermedia.
    Vinciguerra M, Passarello C, Cassarà F, Leto F, Cannata M, Calvaruso G, Di Maggio R, Renda D, Maggio A, Giambona A.
    Int J Lab Hematol; 2016 Feb 18; 38(1):17-26. PubMed ID: 26418075
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  • 19. Molecular Basis of β-Thalassemia Intermedia in Erbil Province of Iraqi Kurdistan.
    Shamoon RP, Al-Allawi NA, Cappellini MD, Di Pierro E, Brancaleoni V, Granata F.
    Hemoglobin; 2015 Feb 18; 39(3):178-83. PubMed ID: 25902180
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  • 20. A novel promoter mutation (HBB: c.-75G>T) was identified as a cause of β(+)-thalassemia.
    Li Z, Li L, Yao Y, Li N, Li Y, Zhang Z, Yan F, Qiu H, Wu C, Zhang Z.
    Hemoglobin; 2015 Feb 18; 39(2):115-20. PubMed ID: 25657036
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